Chiral Transition of SU(4) Gauge Theory with Fermions in Multiple Representations

We report preliminary results on the finite temperature behavior of SU(4) gauge theory with dynamical quarks in both the fundamental and two-index antisymmetric representations. This system is a candidate to present scale separation behavior, where fermions in different representations condense at different temperature or coupling scales. Our simulations, however, reveal a single finite-temperature phase transition at which both representations deconfine and exhibit chiral restoration. It appears to be strongly first order. We compare our results to previous single-representation simulations. We also describe a Pisarski-Wilczek stability analysis, which suggests that the transition should be first order.Comment: 8 pages, 5 figures. Presented at at Lattice 2017, the 35th International Symposium on Lattice Field Theory, Granada, Spain, 18-24 June 201

Automated lattice data generation

The process of generating ensembles of gauge configurations (and measuring various observables over them) can be tedious and error-prone when done "by hand". In practice, most of this procedure can be automated with the use of a workflow manager. We discuss how this automation can be accomplished using Taxi, a minimal Python-based workflow manager built for generating lattice data. We present a case study demonstrating this technology.Comment: 7 pages, 1 figure. Presented at Lattice 2017, the 35th International Symposium on Lattice Field Theory, Granada, Spain, 18-24 June 201

The Operator Product Expansion for Radial Lattice Quantization of 3D ϕ4\phi^4 Theory

At its critical point, the three-dimensional lattice Ising model is described by a conformal field theory (CFT), the 3d Ising CFT. Instead of carrying out simulations on Euclidean lattices, we use the Quantum Finite Elements method to implement radially quantized critical $\phi^4$ theory on simplicial lattices approaching $\mathbb{R} \times S^2$. Computing the four-point function of identical scalars, we demonstrate the power of radial quantization by the accurate determination of the scaling dimensions $\Delta_{\epsilon}$ and $\Delta_{T}$ as well as ratios of the operator product expansion (OPE) coefficients $f_{\sigma \sigma \epsilon}$ and $f_{\sigma \sigma T}$ of the first spin-0 and spin-2 primary operators $\epsilon$ and $T$ of the 3d Ising CFT.Comment: 16 pages, 10 figure

NF-κB/Rel-Mediated Regulation of the Neural Fate in Drosophila

Two distinct roles are described for Dorsal, Dif and Relish, the three NF-κB/Rel proteins of Drosophila, in the development of the peripheral nervous system. First, these factors regulate transcription of scute during the singling out of sensory organ precursors from clusters of cells expressing the proneural genes achaete and scute. This effect is possibly mediated through binding sites for NF-κB/Rel proteins in a regulatory module of the scute gene required for maintenance of scute expression in precursors as well as repression in cells surrounding precursors. Second, genetic evidence suggests that the receptor Toll-8, Relish, Dif and Dorsal, and the caspase Dredd pathway are active over the entire imaginal disc epithelium, but Toll-8 expression is excluded from sensory organ precursors. Relish promotes rapid turnover of transcripts of the target genes scute and asense through an indirect, post-transcriptional mechanism. We propose that this buffering of gene expression levels serves to keep the neuro-epithelium constantly poised for neurogenesis

Toll-8/Tollo Negatively Regulates Antimicrobial Response in the Drosophila Respiratory Epithelium

Barrier epithelia that are persistently exposed to microbes have evolved potent immune tools to eliminate such pathogens. If mechanisms that control Drosophila systemic responses are well-characterized, the epithelial immune responses remain poorly understood. Here, we performed a genetic dissection of the cascades activated during the immune response of the Drosophila airway epithelium i.e. trachea. We present evidence that bacteria induced-antimicrobial peptide (AMP) production in the trachea is controlled by two signalling cascades. AMP gene transcription is activated by the inducible IMD pathway that acts non-cell autonomously in trachea. This IMD-dependent AMP activation is antagonized by a constitutively active signalling module involving the receptor Toll-8/Tollo, the ligand Spätzle2/DNT1 and Ect-4, the Drosophila ortholog of the human Sterile alpha and HEAT/ARMadillo motif (SARM). Our data show that, in addition to Toll-1 whose function is essential during the systemic immune response, Drosophila relies on another Toll family member to control the immune response in the respiratory epithelium