1,725 research outputs found
Macroscopic Features of Light Heavy-Ion Fission Reactions
Global macroscopic features observed in the fully-damped binary processes in
light di-nuclear systems, such as limiting angular momenta, mean total kinetic
energies and energy thresholds for fusion-fission processes (''fission
thresholds") are presented. Their deduced systematics are consistent with that
obtained for heavier systems and follow a fusion-fission picture which can be
described by a realistic rotating liquid drop model considering diffuse-surface
and finite-nuclear-range effects.Comment: 8 pages(REVTeX), 3 figures available upon request, to appear in Phys.
Rev.
Binary Decay of Light Nuclear Systems
A review of the characteristic features found in fully energy-damped,
binarydecay yields from light heavy-ion reactions with is presented. The different aspects of these yields that
have been used to support models of compound-nucleus (CN) fission and
deep-inelastic dinucleus orbiting are highlighted. Cross section calculations
based on the statistical phase space at different stages of the reaction are
presented and compared to the experimental results. Although the statistical
models are found to reproduce most of the observed experimental behaviors, an
additional reaction component corresponding to a heavy-ion resonance or
orbiting mechanism is also evident in certain systems. The system dependence of
this second component is discussed. The extent to which the binary yields in
very light systems can be viewed as resulting from a
fusion-fission mechanism is explored. A number of unresolved questions, such as
whether the different observed behaviors reflect characteristically different
reaction times, are discussed.Comment: 79 pages REVTeX file, 39 ps Figures included - to be publihed in
Physics Report
Entrance-channel Mass-asymmetry Dependence of Compound-nucleus Formation Time in Light Heavy-ion Reactions
The entrance-channel mass-asymmetry dependence of the compound nucleus
formation time in light heavy-ion reactions has been investigated within the
framework of semiclassical dissipative collision models. the model calculations
have been succesfully applied to the formation of the Ar compound
nucleus as populated via the Be+Si, B+Al,
C+Mg and F+F entrance channels. The shape evolution
of several other light composite systems appears to be consistent with the
so-called "Fusion Inhibition Factor" which has been experimentally observed. As
found previously in more massive systems for the fusion-evaporation process,
the entrance-channel mass-asymmetry degree of freedom appears to determine the
competition between the different mechanisms as well as the time scales
involved.Comment: 12 pages, 3 Figures available upon request, Submitted at Phys. Rev.
Irredundant Triangular Decomposition
Triangular decomposition is a classic, widely used and well-developed way to
represent algebraic varieties with many applications. In particular, there
exist sharp degree bounds for a single triangular set in terms of intrinsic
data of the variety it represents, and powerful randomized algorithms for
computing triangular decompositions using Hensel lifting in the
zero-dimensional case and for irreducible varieties. However, in the general
case, most of the algorithms computing triangular decompositions produce
embedded components, which makes it impossible to directly apply the intrinsic
degree bounds. This, in turn, is an obstacle for efficiently applying Hensel
lifting due to the higher degrees of the output polynomials and the lower
probability of success. In this paper, we give an algorithm to compute an
irredundant triangular decomposition of an arbitrary algebraic set defined
by a set of polynomials in C[x_1, x_2, ..., x_n]. Using this irredundant
triangular decomposition, we were able to give intrinsic degree bounds for the
polynomials appearing in the triangular sets and apply Hensel lifting
techniques. Our decomposition algorithm is randomized, and we analyze the
probability of success
Thromboelastometry detects enhancement of coagulation in blood by emicizumab via intrinsic pathway
Non peer reviewe
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