12,128 research outputs found
Bifurcation structures and transient chaos in a four-dimensional Chua model
A four-dimensional four-parameter Chua model with cubic nonlinearity is
studied applying numerical continuation and numerical solutions methods.
Regarding numerical solution methods, its dynamics is characterized on Lyapunov
and isoperiodic diagrams and regarding numerical continuation method, the
bifurcation curves are obtained. Combining both methods the bifurcation
structures of the model were obtained with the possibility to describe the {\it
shrimp}-shaped domains and their endoskeletons. We study the effect of a
parameter that controls the dimension of the system leading the model to
present transient chaos with its corresponding basin of attraction being
riddled.Comment: 9 figures, to appear in PL
Tunnel and thermal c-axis transport in BSCCO in the normal and pseudogap state
We consider the problem of c-axis transport in double-layered cuprates, in
particular with reference to BiSrCaCuO
compounds. We exploit the effect of the two barriers on the thermal and tunnel
transport. The resulting model is able to describe accurately the normal state
c-axis resistivity in BiSrCaCuO, from the
underdoped side up to the strongly overdoped. We extend the model, without
introducing additional parameters, in order to allow for the decrease of the
barrier when an external voltage bias is applied. The extended model is found
to describe properly the c-axis resistivity for small voltage bias above the
pseudogap temperature , the c-axis resistivity for large voltage bias
even below , and the differential curves taken in mesa structures.Comment: 12 pages, 6 figures. Submitted to Superconductor Science and
Technolog
ETosis: A Microbicidal Mechanism beyond Cell Death
Netosis is a recently described type of neutrophil death occurring with the release to the extracellular milieu of a lattice composed of DNA associated with histones and granular and cytoplasmic proteins. These webs, initially named neutrophil extracellular traps (NETs), ensnare and kill microorganisms. Similarly, other cell types, such as eosinophils, mast cells, and macrophages, can also dye by this mechanism; thus, it was renamed as ETosis, meaning death with release of extracellular traps (ETs). Here, we review the mechanism of NETosis/etosis, emphasizing its role in diseases caused by protozoan parasites, fungi, and viruses
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