Does Excellence in Academic Research Attract Foreign R&D?.

We examine the role of host countries’ academic research strengths in global R&D location decisions by multinational firms. While we expect that a firm’s propensity to perform R&D in a host country increases with the strength of local academic research, firms are expected to be heterogeneously positioned to benefit from academic research strengths due to differences in the capacity to absorb and utilize scientific knowledge. We find support for these conjectures in an analysis of foreign R&D activities in 40 host countries and 30 technology fields by 176 leading European, US and Japanese firms during the periods 1995-1998 and 1999-2002. Controlling for a wide range of host country factors, the number of relevant ISI publications by scientists based in the host country has a substantial positive impact on the propensity to conduct foreign R&D. The effect of academic research is significantly larger for firms with a stronger science orientation in R&D - as indicated by citations to scientific literature in prior patents. For host countries with a strong relevant science base, this greater responsiveness of science oriented firms more than offsets a generally greater inclination to concentrate R&D at home. The findings appear robust across a variety of specifications.

Does Excellence in Academic Research Attract Foreign R&D?

We examine the role of host countries’ academic research strengths in global R&D location decisions by multinational firms. While we expect that a firm’s propensity to perform R&D in a host country increases with the strength of local academic research, firms are expected to be heterogeneously positioned to benefit from academic research strengths due to differences in the capacity to absorb and utilize scientific knowledge. We find support for these conjectures in an analysis of foreign R&D activities in 40 host countries and 30 technology fields by 176 leading European, US and Japanese firms during the periods 1995-1998 and 1999-2002. Controlling for a wide range of host country factors, the number of relevant ISI publications by scientists based in the host country has a substantial positive impact on the propensity to conduct foreign R&D. The effect of academic research is significantly larger for firms with a stronger science orientation in R&D - as indicated by citations to scientific literature in prior patents. For host countries with a strong relevant science base, this greater responsiveness of science oriented firms more than offsets a generally greater inclination to concentrate R&D at home. The findings appear robust across a variety of specifications.R&D Internationalization, Knowledge sourcing, Absorptive capacity, Industry-science links

Does Excellence in Academic Research Attract Foreign R&D?

We examine the role of host countries' academic research strengths in global R&D location decisions by multinational firms. While we expect that a firm's propensity to perform R&D in a host country increases with the strength of local academic research, firms are expected to be heterogeneously positioned to benefit from academic research strengths due to differences in the capacity to absorb and utilize scientific knowledge. We find support for these conjectures in an analysis of foreign R&D activities in 40 host countries and 30 technology fields by 176 leading European, US and Japanese firms during the periods 1995-1998 and 1999-2002. Controlling for a wide range of host country factors, the number of relevant ISI publications by scientists based in the host country has a substantial positive impact on the propensity to conduct foreign R&D. The effect of academic research is significantly larger for firms with a stronger science orientation in R&D - as indicated by citations to scientific literature in prior patents. For host countries with a strong relevant science base, this greater responsiveness of science oriented firms more than offsets a generally greater inclination to concentrate R&D at home. The findings appear robust across a variety of specifications.R&D internationalization, Knowledge sourcing, Absorptive capacity, Industry-science links

Causality of Photon Propagation under Dominant Energy Condition in Non-linear Electrodynamics

Recently, various types of regular black hole model are reintroduced as the solution of the Einstein equations coupled with nonlinear electrodynamics (NED). In NED, it is known that photons do not propagate along the null geodesics of the spacetime geometry, but of so-called effective geometry, which suggests the possibility of so-called ``faster/slower than light" photons. We study the relation between the causality of photons and the dominant energy condition (DEC) in some static and spherically symmetric black hole spacetimes in NED. We show that if photon trajectories with a nonzero angular momentum are timelike in the spacetime geometry, DEC is always satisfied in static and spherically symmetric spacetimes in any NED that admits the Maxwell limit, and vice versa, at least, in the weak field limit. Thus, this implies that in such NED, the violation of DEC admits the existence of faster than light photons.Comment: 14 page

Stable bound orbits in microstate geometries

We show the existence of stable bound orbits for the massive and massless particles moving in the simplest microstate geometry spacetime in the bosonic sector of the five-dimensional minimal supergravity. In our analysis, reducing the motion of particles to a two-dimensional potential problem, we numerically investigate whether the potential has a negative local minimum.Comment: 18 pages, 30 figure

Dynamics of Myers-Perry black holes with almost equal angular momenta in odd dimensions

We investigate the nonlinear dynamics of D=2N+3 Myers-Perry black holes with almost equal angular momenta, which have N equal spins out of possible N+1 spins. In particular, we study the ultraspinning instability and the fate of its nonlinear evolution using the large D effective theory approach. We find that every stationary phase can be mapped to the counterpart in the singly rotating phase within the leading order effective theory. From the known results of singly rotating solutions, we obtain the phase diagram of almost equally rotating black holes. We also obtain a certain implication for the possible topology changing transition.Comment: v1: 33 pages, 6 figures; v2: fixed minor typo

A black lens in bubble of nothing

Applying the inverse scattering method to static and bi-axisymmetric Einstein equations, we construct a non-rotating black lens inside bubble of nothing whose horizon is topologically lens space L(n,1)=S^3/Z_n. Using this solution, we discuss whether a static black lens can be in equilibrium by the force balance between the expansion and gravitational attraction.Comment: 14 pages, 2 figures. arXiv admin note: text overlap with arXiv:1902.1054

Reticuloendothelial cell function in autoimmune hemolytic anemia (AIHA): studies on the mechanism of peripheral monocyte activation.

We examined the activity of peripheral blood monocytes in patients with autoimmune hemolytic anemia (AIHA) using an in vitro assay of monocyte-macrophage interaction with erythrocytes and an antibody-dependent cell-mediated cytotoxicity (ADCC) assay. The monocytes of AIHA patients in the hemolyzing period phagocytized autologous sensitized red cells and anti-D coated red cells more avidly than normal control monocytes. There was no significant relationship between phagocytic activity and ADCC activity. The activated monocytes phagocytized autologous sensitized red cells, but had no ADCC activity in a short time 51Cr release assay. Phagocytic activity of the patients' monocytes against autologous erythrocytes rapidly decreased after treatment with prednisolone even though the red cell sensitization with antibody remained almost the same as during the hemolyzing period. We postulated that the activation of monocytes in AIHA was due to the &#34;arming&#34; effect of anti-erythrocyte antibody, but we think that other mechanisms may also be involved in the activation of monocytes.</p