Converting normal insulators into topological insulators via tuning orbital levels

Tuning the spin-orbit coupling strength via foreign element doping and/or modifying bonding strength via strain engineering are the major routes to convert normal insulators to topological insulators. We here propose an alternative strategy to realize topological phase transition by tuning the orbital level. Following this strategy, our first-principles calculations demonstrate that a topological phase transition in some cubic perovskite-type compounds CsGeBr$_3$ and CsSnBr$_3$ could be facilitated by carbon substitutional doping. Such unique topological phase transition predominantly results from the lower orbital energy of the carbon dopant, which can pull down the conduction bands and even induce band inversion. Beyond conventional approaches, our finding of tuning the orbital level may greatly expand the range of topologically nontrivial materials

Improving the security of quantum direct communication with authentication

Two protocols of quantum direct communication with authentication [Phys. Rev. A {\bf 73}, 042305 (2006)] are recently proposed by Lee, Lim and Yang. In this paper we will show that in the two protocols the authenticator Trent should be prevented from knowing the secret message of communication. The first protocol can be eavesdropped by Trent using the the intercept-measure-resend attack, while the second protocol can be eavesdropped by Trent using single-qubit measurement. To fix these leaks, I revise the original versions of the protocols by using the Pauli-Z operation $\sigma_z$ instead of the original bit-flip operation $X$. As a consequence, the protocol securities are improved.Comment: Any suggestion,comment or help is welcome

Two Hox cofactors, the Meis/Hth homolog UNC-62 and the Pbx/Exd homolog CEH-20, function together during C. elegans postembryonic mesodermal development

AbstractThe TALE homeodomain-containing PBC and MEIS proteins play multiple roles during metazoan development. Mutations in these proteins can cause various disorders, including cancer. In this study, we examined the roles of MEIS proteins in mesoderm development in C. elegans using the postembryonic mesodermal M lineage as a model system. We found that the MEIS protein UNC-62 plays essential roles in regulating cell fate specification and differentiation in the M lineage. Furthermore, UNC-62 appears to function together with the PBC protein CEH-20 in regulating these processes. Both unc-62 and ceh-20 have overlapping expression patterns within and outside of the M lineage, and they share physical and regulatory interactions. In particular, we found that ceh-20 is genetically required for the promoter activity of unc-62, providing evidence for another layer of regulatory interactions between MEIS and PBC proteins