4,116 research outputs found
Top quark pair production at small transverse momentum in hadronic collisions
We investigate the transverse momentum resummation for top quark pair
production at hadron colliders using the soft-collinear effective theory and
the heavy-quark effective theory. We derive the factorization formula for
production at small pair transverse momentum, and show in detail the
procedure for calculating the key ingredient of the factorization formula: the
next-to-leading order soft functions. We compare our numerical results with
experimental data and find that they are consistent within theoretical and
experimental uncertainties. To verify the correctness of our resummation
formula, we expand it to the next-to-leading order and the
next-to-next-to-leading order, and compare those expressions with the exact
fixed-order results numerically. Finally, using the results of transverse
momentum resummation, we discuss the transverse-momentum-dependent
forward-backward asymmetry at the Tevatron.Comment: 39 pages, 7 figures, 1 table; final version in PR
Precise QCD predictions on top quark pair production mediated by massive color octet vector boson at hadron colliders
We present a theoretical framework for systematically calculating
next-to-leading order (NLO) QCD effects to various experimental observables in
models with massive COVB in a model independent way at hadron colliders.
Specifically, we show the numerical results for the NLO QCD corrections to
total cross sections, invariant mass distribution and AFB of top quark pairs
production mediated by a massive COVB in both the fixed scale (top quark mass)
scheme and the dynamical scale (top pair invariant mass) scheme. Our results
show that the NLO QCD calculations in the dynamical scale scheme is more
reasonable than the fixed scheme and the naive estimate of the NLO effects by
simple rescaling of the LO results with the SM NLO K-factor is not appropriate.Comment: 6 pages, 5 figures, 2 tables; version published in EPJ
Parton distribution functions and nuclear EMC effect in a statistical model
A new and simple statistical approach is performed to calculate the parton
distribution functions (PDFs) of the nucleon in terms of light-front kinematic
variables. Analytic expressions of x-dependent PDFs are obtained in the whole x
region. And thereafter, we treat the temperature T as a parameter of the atomic
number A to explain the nuclear EMC effect in the region . We
give the predictions of PDF ratios, and they are very different from those by
other models, thus experiments aiming at measuring PDF ratios are suggested to
provide a discrimination of different models.Comment: 4 pages, no figure; talk given at the 5th International Conference On
Quarks and Nuclear Physics (QNP09), Sep 2009, Beijing Chin
Constraints on flavor-changing neutral-current couplings from the signal of associated production with QCD next-to-leading order accuracy at the LHC
We study a generic Higgs boson and a top quark associated production via
model-independent flavor-changing neutral-current couplings at the LHC,
including complete QCD next-to-leading order (NLO) corrections to the
production and decay of the top quark and the Higgs boson. We find that QCD NLO
corrections can increase the total production cross sections by about 48.9% and
57.9% for the and coupling induced processes at the LHC,
respectively. After kinematic cuts are imposed on the decay products of the top
quark and the Higgs boson, the QCD NLO corrections are reduced to 11% for the
coupling induced process and almost vanish for the coupling induced
process. Moreover, QCD NLO corrections reduce the dependence of the total cross
sections on the renormalization and factorization scales. We also discuss
signals of the associated production with the decay mode t \rightarrow
bl^{+}E \slash_{T}, H \rightarrow b\bar{b} and production with the
decay mode \bar{t} \rightarrow H\bar{q}, t\rightarrow bl^{+}E \slash_{T}. Our
results show that, in some parameter regions, the LHC may observe the above
signals at the level. Otherwise, the upper limits on the FCNC
couplings can be set.Comment: 28 pages, 14 figures, 5 tables; version published in PR
Smoking cessation and risk of esophageal cancer by histological type : systematic review and meta-analysis
Background
Tobacco smoking strongly increases risk of esophageal squamous cell carcinoma and moderately increases risk of esophageal adenocarcinoma. How smoking cessation influences esophageal cancer risk across histological subtypes, time latencies, and geographic regions is not clear.
Methods
Studies were systematically searched on Medline, Embase, Web of Science, Cochrane Library, and ClinicalTrials.gov. Pooled estimates of risk ratios (RRs) were derived using a random effects model. Cochran’s Q test and I2 statistic were used to detect heterogeneity.
Results
Among 15 009 studies, 52 fulfilled the inclusion criteria. Using nonsmokers as a reference, risk of esophageal squamous cell carcinoma was lower among former smokers (RR = 2.05, 95% confidence interval [CI] = 1.71 to 2.45) than among current smokers (RR = 4.18, 95% CI = 3.42 to 5.12). Compared with current smokers, a strong risk reduction was evident after five or more years (RR = 0.59, 95% CI = 0.47 to 0.75), and became stronger after 10 or more years (RR = 0.42, 95% CI = 0.34 to 0.51) and 20 or more years (RR = 0.34, 95% CI = 0.25 to 0.47) following smoking cessation. The risk reduction was strong in Western populations, while weak in Asian populations. Using nonsmokers as reference, the risk of esophageal adenocarcinoma was only slightly lower among former smokers (RR = 1.66, 95% CI = 1.48 to 1.85) than among current smokers (RR = 2.34, 95% CI = 2.04 to 2.69). The risk of esophageal adenocarcinoma did not show any clear reduction over time after smoking cessation, with a risk ratio of 0.72 (95% CI = 0.52 to 1.01) 20 or more years after smoking cessation, compared with current smokers.
Conclusions
Smoking cessation time-dependently decreases risk of esophageal squamous cell carcinoma, particularly in Western populations, while it has limited influence on the risk of esophageal adenocarcinoma.Swedish Cancer SocietySwedish Research CouncilAccepte
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