Strain gradient crystal plasticity with evolving length scale: Application to voided irradiated materials

International audienceA micromorphic crystal plasticity model is used to simulate slip band localization in single crystalsunder simple shear at finite deformations. Closed form analytical solutions are derived for singleslip in the case of positive, zero and negative strain hardening. Linear negative strain hardening,i.e. linear softening, leads to a constant localization slip band width, while non linear softening andsaturating behaviour results in an increasing band width. An enhanced model is therefore proposed inorder to maintain a bounded localization slip band width when considering an exponential softeningbehaviour. Analytical solutions are used to validate finite element computation of the same boundaryvalue problems. The enhanced micromorphic crystal plasticity model is then applied to predict theinteraction between localized slip bands and voids encountered in voided irradiated materials. For thatpurpose, periodic porous unit cells are loaded in simple shear with a strain gradient crystal plasticitymatrix material. The finite element simulation results show that, for a given void volume fraction, thelarger the voids, the wider the localization band. However, for a given void size, the larger the voidvolume fraction, the narrower the localization band. In addition a satisfactory qualitative agreementof the rotation and elongation of the voids with the experimental observations made in irradiatedmaterials is observed, where small voids are shown to remain ellipsoidal for larger shear strains thanlarge voids which deform into peanut-like shapes

Negative-emissions technology portfolios to meet the 1.5 °C target

Our carbon-intensive economy has led to an average temperature rise of 1 °C since pre-industrial times. As a consequence, the world has seen increasing droughts, significant shrinking of the polar ice caps, and steady sea-level rise. To stall these issues’ worsening further, we must limit global warming to 1.5 °C. In addition to the economy’s decarbonization, this endeavour requires the use of negative-emissions technologies (NETs) that remove the main greenhouse gas, carbon dioxide, from the atmosphere. While techno-economic feasibility alone has driven the definition of negative-emissions solutions, NETs’ diverse, far-reaching implications demand a more holistic assessment. Here, we present a comprehensive framework, integrating NETs’ critical performance aspects of feasibility, effectiveness, and side impacts, to define the optimal technology mix within realistic outlooks. The resulting technology portfolios provide a useful new benchmark to compare carbon avoidance and removal measures and deliberately choose the best path to solve the climate emergency.Industrial Ecolog

Model Systems to Study the Mechanism of Vascular Aging

Cardiovascular diseases are the leading cause of death globally. Within cardiovascular aging, arterial aging holds significant importance, as it involves structural and functional alterations in arteries that contribute substantially to the overall decline in cardiovascular health during the aging process. As arteries age, their ability to respond to stress and injury diminishes, while their luminal diameter increases. Moreover, they experience intimal and medial thickening, endothelial dysfunction, loss of vascular smooth muscle cells, cellular senescence, extracellular matrix remodeling, and deposition of collagen and calcium. This aging process also leads to overall arterial stiffening and cellular remodeling. The process of genomic instability plays a vital role in accelerating vascular aging. Progeria syndromes, rare genetic disorders causing premature aging, exemplify the impact of genomic instability. Throughout life, our DNA faces constant challenges from environmental radiation, chemicals, and endogenous metabolic products, leading to DNA damage and genome instability as we age. The accumulation of unrepaired damages over time manifests as an aging phenotype. To study vascular aging, various models are available, ranging from in vivo mouse studies to cell culture options, and there are also microfluidic in vitro model systems known as vessels-on-a-chip. Together, these models offer valuable insights into the aging process of blood vessels.</p

A comparison between global nutrient retention models for freshwater systems

Environmental Biolog

Global regionalized characterization factors for phosphorus and nitrogen impacts on freshwater fish biodiversity

Inefficient global nutrient (i.e., phosphorus (P) and nitrogen (N)) management leads to an increase in nutrient delivery to freshwater and coastal ecosystems and induces eutrophication in these aquatic environments. This process threatens the various species inhabiting these ecosystems. In this study, we developed regionalized characterization factors (CFs) for freshwater eutrophication at 0.5 × 0.5-degree resolution, considering different fates for direct emissions to freshwater, diffuse emissions, and increased erosion due to agricultural land use. The CFs were provided for global and regional species loss of freshwater fish. CFs for global species loss were quantified by integrating global extinction probabilities. Results showed that the CFs for P and N impacts on freshwater fish are higher in densely populated regions that encompass either large lakes or the headwaters of large rivers. Focusing on nutrient-limited areas increases country-level CFs in 51.9 % of the countries for P and 49.5 % of the countries for N compared to not considering nutrient limitation. This study highlights the relevance of considering freshwater eutrophication impacts via both P and N emissions and identifying the limiting nutrient when performing life cycle impact assessments.Environmental Biolog

Effects of nitrogen emissions on fish species richness across the world’s freshwater ecoregions

Industrial EcologyEnvironmental Biolog

A calibration method for broad-bandwidth cavity enhanced absorption spectroscopy performed with supercontinuum radiation

An efficient calibration method has been developed for broad-bandwidth cavity enhanced absorption spectroscopy. The calibration is performed using phase shift cavity ring-down spectroscopy, which is conveniently implemented through use of an acousto-optic tunable filter (AOTF). The AOTF permits a narrowband portion of the SC spectrum to be scanned over the full high-reflectivity bandwidth of the cavity mirrors. After calibration the AOTF is switched off and broad-bandwidth CEAS can be performed with the same light source without any loss of alignment to the set-up. We demonstrate the merits of the method by probing transitions of oxygen molecules O-2 and collisional pairs of oxygen molecules (O-2)(2) in the visible spectral range

Towards an Asymptotic-Safety Scenario for Chiral Yukawa Systems

We search for asymptotic safety in a Yukawa system with a chiral $U(N_L)_L\otimes U(1)_R$ symmetry, serving as a toy model for the standard-model Higgs sector. Using the functional RG as a nonperturbative tool, the leading-order derivative expansion exhibits admissible non-Ga\ssian fixed-points for $1 \leq N_L \leq 57$ which arise from a conformal threshold behavior induced by self-balanced boson-fermion fluctuations. If present in the full theory, the fixed-point would solve the triviality problem. Moreover, as one fixed point has only one relevant direction even with a reduced hierarchy problem, the Higgs mass as well as the top mass are a prediction of the theory in terms of the Higgs vacuum expectation value. In our toy model, the fixed point is destabilized at higher order due to massless Goldstone and fermion fluctuations, which are particular to our model and have no analogue in the standard model.Comment: 16 pages, 8 figure

Model Systems to Study the Mechanism of Vascular Aging

Cardiovascular diseases are the leading cause of death globally. Within cardiovascular aging, arterial aging holds significant importance, as it involves structural and functional alterations in arteries that contribute substantially to the overall decline in cardiovascular health during the aging process. As arteries age, their ability to respond to stress and injury diminishes, while their luminal diameter increases. Moreover, they experience intimal and medial thickening, endothelial dysfunction, loss of vascular smooth muscle cells, cellular senescence, extracellular matrix remodeling, and deposition of collagen and calcium. This aging process also leads to overall arterial stiffening and cellular remodeling. The process of genomic instability plays a vital role in accelerating vascular aging. Progeria syndromes, rare genetic disorders causing premature aging, exemplify the impact of genomic instability. Throughout life, our DNA faces constant challenges from environmental radiation, chemicals, and endogenous metabolic products, leading to DNA damage and genome instability as we age. The accumulation of unrepaired damages over time manifests as an aging phenotype. To study vascular aging, various models are available, ranging from in vivo mouse studies to cell culture options, and there are also microfluidic in vitro model systems known as vessels-on-a-chip. Together, these models offer valuable insights into the aging process of blood vessels