Renewed interest in the kallikrein-kinin system was awakened in the early seventies by the suggestion that it might be involved in the regulation of sodium excretion [1, 2] or in the development of hypertension [3, 4]. However, due to complexities inherent in the measurement of the enzyme in the kidneys, only a few reports on the activity of the renal kallikrein have been published to date [5–8]

Breypohl, Gera