90 research outputs found
Evidence-based Kernels: Fundamental Units of Behavioral Influence
This paper describes evidence-based kernels, fundamental units of behavioral influence that appear to underlie effective prevention and treatment for children, adults, and families. A kernel is a behavior–influence procedure shown through experimental analysis to affect a specific behavior and that is indivisible in the sense that removing any of its components would render it inert. Existing evidence shows that a variety of kernels can influence behavior in context, and some evidence suggests that frequent use or sufficient use of some kernels may produce longer lasting behavioral shifts. The analysis of kernels could contribute to an empirically based theory of behavioral influence, augment existing prevention or treatment efforts, facilitate the dissemination of effective prevention and treatment practices, clarify the active ingredients in existing interventions, and contribute to efficiently developing interventions that are more effective. Kernels involve one or more of the following mechanisms of behavior influence: reinforcement, altering antecedents, changing verbal relational responding, or changing physiological states directly. The paper describes 52 of these kernels, and details practical, theoretical, and research implications, including calling for a national database of kernels that influence human behavior
Myocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection
Role of the phosphocreatine system on energetic homeostasis in skeletal and cardiac muscles
Metabolische Kompensationen des menschlichen Herzmuskelgewebes bei der koronaren Herzerkrankung
Herabgesetzte Kontraktilität des nicht infarzierten Herzmuskels nach experimentellem Infarkt
Combined effects of age and dietary fat on beta 1-receptors and Ca2+ channels in rat hearts
Suppression of protein degradation in progressive cardiac hypertrophy of chronic aortic regurgitation.
Experimental myocardial infarction in the cat. I. Reversible decline in contractility of noninfarcted muscle.
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