Non-syndromic oligodontia in permanent dentition of monozygotic twins: report of a rare case

Aim: To report a case of non-syndromic oligodontia involving 26 permanent teeth in monozygotic twin sisters and to discuss the possible genetic etiology, inheritance pattern and associated dental anomalies of this condition.\ud \ud Background: Hypodontia constitutes one of the most common developmental anomalies in humans and is defined as developmental absence of one or more teeth with reported prevalence of 1.6 to 9.6% in the permanent dentition. Oligodontia is defined as agenesis of six or more teeth excluding third molars.\ud \ud Case Report: This article describes a case of non-syndromic oligodontia involving 26 permanent teeth in monozygotic twin sisters. The twins had positive family history of hypodontia in their paternal grandmother, parental consanguinity (first cousin) and similarity in pattern of oligodontia. Root formation of permanent maxillary first molars and central incisors was delayed in both the twins.\ud \ud Summary: This article reports a case of non-syndromic oligodontia in permanent dentition of monozygotic twins. Possible genetic etiology, inheritance pattern and associated dental anomalies are discussed.\ud \ud Clinical Significance: Strong genetic link associated with oligodontia help the dentist to know the possibility of its occurrence in other family members and in future generations

Oral myiasis associated with drug induced gingival hyperplasia – a rare case report

Introduction: Myiasis (Greek: myi = fly) refers to infestation of living tissues of humans and animals by Dipterous eggs or larvae. Incidence of oral myiasis is comparatively lesser than that of cutaneous myiasis. We report a rare case of oral myiasis of anterior maxilla associated with drug induced gingival enlargement. Observation: We report a rare case of oral myiasis of anterior maxilla with amlodipine induced gingival enlargement in a sixty-two-year-old male with history of hypertension, hemiplegia and diabetes mellitus. Patient was mouth breather and presented with poor oral hygiene. Generalized gingival hyperplasia was observed. Multiple maggots were observed in the ulcerated areas on maxillary anterior alveolus. The maggots were mechanically removed with curettage after application of turpentine oil. Cleaning and debridement of the wound was done. After one month follow-up there was complete healing of the lesion. Maintenance of oral hygiene and gingivectomy as indicated was planned for this patient. Conclusion: Clinical significance of presented case is its rarity of association with drug induced gingival hyperplasia and typical presence of multiple predisposing factors. This case report provides interesting information about predisposing factors, clinical features and management of oral myiasis that could help clinicians in the diagnosis and management of this condition