Fabrication of Worm-Like Nanorods and Ultrafine Nanospheres of Silver Via Solid-State Photochemical Decomposition

Worm-like nanorods and nanospheres of silver have been synthesized by photochemical decomposition of silver oxalate in water by UV irradiation in the presence of CTAB and PVP, respectively. No external seeds have been employed for the synthesis of Ag nanorods. The synthesized Ag colloids have been characterized by UV-visible spectra, powder XRD, HRTEM, and selected area electron diffraction (SAED). Ag nanospheres of average size around 2 nm have been obtained in the presence of PVP. XRD and TEM analyses revealed that top and basal planes of nanorods are bound with {111} facets. Williamson–Hall plot has revealed the presence of defects in the Ag nanospheres and nanorods. Formation of defective Ag nanocrystals is attributed to the heating effect of UV-visible irradiation

The relationships among leadership, goal orientation, and service quality in high-contact service industries : an empirical study

2012-2013 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptOthersThis research was supported in part by The Hong Kong Polytechnic University under Grant no.1-ZV6DPublishe

Market competitiveness and quality performance in high-contact service industries

2012-2013 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptRGCPolyU 551410Publishe

Pathogenesis of the pandemic swine-origin influenza H1N1 virus (H1N1pdm) in humans: studies of viral tropism and host repsonse in primary differentiated human respiratory epithelial cell cultures in vitro and in ex vivo

Ref. No. RFCID: HSI-Lab-15Resp. diseases Ab0

A safe and efficient lipidic nanoparticle carrier of gold porphryin for the treatment of neuroblastoma

The 3rd European Conference for Clinical Nanomedicine (CLINAM), Basel, Switzerland, 10-12 May 2010

H5N1 influenza virus-induced mediators upregulate RIG-I in uninfected cells by paracrine effects contributing to amplified cytokine cascades

Highly pathogenic avian influenza H5N1 viruses cause severe disease in humans, and dysregulation of cytokine responses is believed to contribute to the pathogenesis of human H5N1 disease. However, mechanisms leading to the increased induction of proinflammatory cytokines by H5N1 viruses are poorly understood. We show that the innate sensing receptor RIG-I is involved in interferon regulatory factor 3 (IRF3), NF-κB nuclear translocation, p38 activation, and the subsequent interferon (IFN) β, IFN-λ1, and tumor necrosis factor α induction during H5N1 infection. Soluble mediators from H5N1-infected human macrophages upregulate RIG-I, MDA5, and TLR3 to much higher levels than those from seasonal H1N1 in uninfected human macrophages and alveolar epithelial cells via paracrine IFNAR1/JAK but not IFN-λ receptor signaling. Compared with H1N1 virus-induced mediators, H5N1 mediators markedly enhance the cytokine response to PolyIC and to both seasonal and H5N1 virus infection in a RIG-I-dependent manner. Thus, sensitizing neighboring cells by upregulation of RIG-I contributes to the amplified cytokine cascades during H5N1 infection. © 2011 The Author.link_to_OA_fulltex