Decay studies of 288−287115^{288-287}115 alpha-decay chains

The $\alpha$-decay chains of $^{288-287}115$ are studied along with the possible cluster decay modes by using the preformed cluster model (PCM). The calculated $\alpha$-decay half-lives are compared with experimental data and other model calculations. The calculated Q-values, penetration probabilities and preformation probabilities factors for $\alpha$-decay suggest that $^{283}_{170}113$,$^{287}_{172}115$ and $^{272}_{165}107$ parent nuclei are more stable against the $\alpha$-decay. These alpha decay chains are further explored for the possibilities of cluster decay. Decay half lives of different cluster from different nuclei of the decay chains point to the extra stability near or at the deformed shells Z=108, N=162 and Z=100, N=152. The decay half-lives for $^{14}C$ and $^{48}Ca$ clusters are lower than the current experimental limit ($\approx$ $10^{28}$sec)

Adiabatic quantum tunneling in heavy-ion sub-barrier fusion

High precision measurements of the fusion excitation functions for the reactions $^{40}$Ca + $^{194}$Pt, $^{192}$Os clearly demonstrate that projectile excitation significantly modifies the potential barrier distribution. In sharp contrast, fusion of $^{16}$O + $^{144}$Sm appears to show no influence of the projectile excitation on the shape of the barrier distribution. These apparently conflicting conclusions are reconciled in this work, using realistic coupled--channels calculations, which show that high energy states produce an adiabatic potential renormalisation. This result indicates that adiabatic effects restrict, in a natural way, the states which influence the shape of a fusion barrier distribution. The analysis of barrier distributions thus offers a criterion for the relevance of the `counter term' prescription in the Caldeira-Leggett approach

Entropy Corrections for Schwarzschild and Reissner-Nordstr\"om Black Holes

Schwarzschild black hole being thermodynamically unstable, corrections to its entropy due to small thermal fluctuations cannot be computed. However, a thermodynamically stable Schwarzschild solution can be obtained within a cavity of any finite radius by immersing it in an isothermal bath. For these boundary conditions, classically there are either two black hole solutions or no solution. In the former case, the larger mass solution has a positive specific heat and hence is locally thermodynamically stable. We find that the entropy of this black hole, including first order fluctuation corrections is given by: {\cal S} = S_{BH} - \ln[\f{3}{R} (S_{BH}/4\p)^{1/2} -2]^{-1} + (1/2) \ln(4\p), where $S_{BH}=A/4$ is its Bekenstein-Hawking entropy and $R$ is the radius of the cavity. We extend our results to four dimensional Reissner-Nordstr\"om black holes, for which the corresponding expression is: {\cal S} = S_{BH} - \f{1}{2} \ln [ {(S_{BH}/\p R^2) ({3S_{BH}}/{\p R^2} - 2\sqrt{{S_{BH}}/{\p R^2 -\a^2}}) \le(\sqrt{{S_{BH}}/{\p R^2}} - \a^2 \ri)}/ {\le({S_{BH}}/{\p R^2} -\a^2 \ri)^2} ]^{-1} +(1/2)\ln(4\p). Finally, we generalise the stability analysis to Reissner-Nordstr\"om black holes in arbitrary spacetime dimensions, and compute their leading order entropy corrections. In contrast to previously studied examples, we find that the entropy corrections in these cases have a different character.Comment: 6 pages, Revtex. References added, minor changes. Version to appear in Class. Quant. Gra

Black Hole Thermodynamics: Entropy, Information and Beyond

We review some recent advances in black hole thermodynamics, including statistical mechanical origins of black hole entropy and its leading order corrections, from the viewpoints of various quantum gravity theories. We then examine the information loss problem and some possible approaches to its resolution. Finally, we study some proposed experiments which may be able to provide experimental signatures of black holes.Comment: Plenary talk given at the Fifth International Conference on Gravitation and Cosmology, Cochin, 7 January 2004. 13 pages, Revte

Cloning and heterologous expression of a gene encoding lycopene-epsilon-cyclase, a precursor of lutein in tea (Camellia sinensis var assamica)

This report describes the cloning and expression of a gene lycopene epsilon cyclase, (LCYE) from Camellia sinensis var assamica which is a precursor of the carotenoid lutein in tea. The 1982 bp cDNA sequence with 1599 bp open reading frame of LCYE was identified from an SSH library constructed for quality trait in tea. 5’ and 3’ RACE (rapid-amplification of cDNA ends) was done to clone the full length cDNA of LCYE. Homology studies showed that the deduced amino acid sequence of LCYE gene had the highest sequence identity of up to 84% with Vitis vinefera. The cloned gene was successfully expressed in a PET based Escherichia coli expression system. The size of the expressed protein was 59615 Daltons. A suppression subtractive library was constructed using a quality clone H3111 (tester) and a garden series clone T3E3 (driver).Key words: Carotenoid, RACE, heterologous expression, lutein, tea

Anti-de Sitter black holes, perfect fluids, and holography

We consider asymptotically anti-de Sitter black holes in $d$-spacetime dimensions in the thermodynamically stable regime. We show that the Bekenstein-Hawking entropy and its leading order corrections due to thermal fluctuations can be reproduced by a weakly interacting fluid of bosons and fermions (`dual gas') in $\Delta=\alpha(d-2)+1$ spacetime dimensions, where the energy-momentum dispersion relation for the constituents of the fluid is assumed to be $\epsilon = \kappa p^\alpha$. We examine implications of this result for entropy bounds and the holographic hypothesis.Comment: Minor changes to match published version. 9 Pages, Revte

Vortex dynamics and upper critical fields in ultrathin Bi films

Current-voltage (I-V) characteristics of quench condensed, superconducting, ultrathin $Bi$ films in a magnetic field are reported. These I-V's show hysteresis for all films, grown both with and without thin $Ge$ underlayers. Films on Ge underlayers, close to superconductor-insulator transition (SIT), show a peak in the critical current, indicating a structural transformation of the vortex solid (VS). These underlayers, used to make the films more homogeneous, are found to be more effective in pinning the vortices. The upper critical fields (B$_{c2}$) of these films are determined from the resistive transitions in perpendicular magnetic field. The temperature dependence of the upper critical field is found to differ significantly from Ginzburg-Landau theory, after modifications for disorder.Comment: Phys Rev B, to be published Figure 6 replaced with correct figur

Long-term moderate calorie restriction inhibits inflammation without impairing cell-mediated immunity: A randomized controlled trial in non-obese humans

Calorie restriction (CR) inhibits inflammation and slows aging in many animal species, but in rodents housed in pathogen-free facilities, CR impairs immunity against certain pathogens. However, little is known about the effects of long-term moderate CR on immune function in humans. In this multi-center, randomized clinical trial to determine CR's effect on inflammation and cell-mediated immunity, 218 healthy non-obese adults (20-50 y), were assigned 25% CR (n=143) or an ad-libitum (AL) diet (n=75), and outcomes tested at baseline, 12, and 24 months of CR. CR induced a 10.4% weight loss over the 2-y period. Relative to AL group, CR reduced circulating inflammatory markers, including total WBC and lymphocyte counts, ICAM-1 and leptin. Serum CRP and TNF-α concentrations were about 40% and 50% lower in CR group, respectively. CR had no effect on the delayed-type hypersensitivity skin response or antibody response to vaccines, nor did it cause difference in clinically significant infections. In conclusion, long-term moderate CR without malnutrition induces a significant and persistent inhibition of inflammation without impairing key in vivo indicators of cell-mediated immunity. Given the established role of these pro-inflammatory molecules in the pathogenesis of multiple chronic diseases, these CR-induced adaptations suggest a shift toward a healthy phenotype