73 research outputs found
Evolution of language driven by social dynamics
The survival of endangered languages in complex language competition depends
on socio-cultural status and honour endowed (by itself and by the other) among
them. The restriction in the endorsement of this honour leads to language
extinction of one language, and rise of the other. Endorsing proper mutual
honour each other trigger the co-existence of language speakers and can save
both languages from extinction. The lost of respect to each other drives the
death of both languages. We found a minimal or critical mutual honour
(a=0.9635) which protects the two languages from extinction. The increase in
mutual honour from this minimal value allows increase in the populations of the
two languages speakers. The state of co-existence of competing languages
abolishes the concept of minority and majority in language competition which
can be obtained by mutual honour. Further, excess biased honour to a particular
language (minority or majority) force the language to extinct. In mean-field
approximation of language competition, magnetization parameter can be taken as
an indicator of survival of a language.Comment: 5 pages, 4 figure
Bifurcation in cell cycle dynamics regulated by p53
We study the regulating mechanism of p53 on the properties of cell cycle
dynamics in the light of the proposed model of interacting p53 and cell cycle
networks via p53. Irradiation (IR) introduce to p53 compel p53 dynamics to
suffer different phases, namely oscillating and oscillation death (stabilized)
phases. The IR induced p53 dynamics undergo collapse of oscillation with
collapse time \Delta t which depends on IR strength. The stress p53 via IR
drive cell cycle molecular species MPF and cyclin dynamics to different states,
namely, oscillation death, oscillations of periods, chaotic and sustain
oscillation in their bifurcation diagram. We predict that there could be a
critical \Delta t_c induced by p53 via IR_c, where, if \Delta t < \Delta t_c
the cell cycle may come back to normal state, otherwise it will go to cell
cycle arrest (apoptosis)
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