202 research outputs found
Combining nano-silicon with oxide glass in anodes for Li-ion batteries
Vanadium-tellurite glasses (VT) have emerged as promising anode materials for lithium-ion batteries (LIBs). Despite this, the Li-ion storage capacity of the VT glass anode is still insufficient to meet the demands for the next generation of advanced LIBs. Silicon (Si) anode has ultrahigh theoretical capacity but suffers from significant volume expansion during lithiation and delithiation. In this work, we combined Si nanoparticles with VT glass to prepare Si@VT composite anode for LIBs. The composite was produced through heat-treatment at different temperatures, some of which were hot-pressed under the isostatic pressure of 100 MPa. The Si@VT composite exhibited a synergistic effect that integrated the strengths of both VT glass and Si, resulting in a substantial enhancement of its electrochemical performance. The systematic characterizations of the composite-based anodes revealed the optimal conditions for fabricating the high-performance Si@VT composite: a silicon fraction of 10 wt% and a hot-pressing temperature of 620 K. This composite stood out as the optimal choice, exhibiting a capacity of 353 mA h gâ1 at 1 A gâ1 after 1000 cycles. This capacity surpasses that of VT glass anode by over threefold and that of pure Si anode by twelvefold.</p
Resveratrol attenuates intermittent hypoxia-induced macrophage migration to visceral white adipose tissue and insulin resistance in male mice.
Chronic intermittent hypoxia during sleep (IH), as occurs in sleep apnea, promotes systemic insulin resistance. Resveratrol (Resv) has been reported to ameliorate high-fat diet-induced obesity, inflammation, and insulin resistance. To examine the effect of Resv on IH-induced metabolic dysfunction, male mice were subjected to IH or room air conditions for 8 weeks and treated with either Resv or vehicle (Veh). Fasting plasma levels of glucose, insulin, and leptin were obtained, homeostatic model assessment of insulin resistance index levels were calculated, and insulin sensitivity tests (phosphorylated AKT [also known as protein kinase B]/total AKT) were performed in 2 visceral white adipose tissue (VWAT) depots (epididymal [Epi] and mesenteric [Mes]) along with flow cytometry assessments for VWAT macrophages and phenotypes (M1 and M2). IH-Veh and IH-Resv mice showed initial reductions in food intake with later recovery, with resultant lower body weights after 8 weeks but with IH-Resv showing better increases in body weight vs IH-Veh. IH-Veh and IH-Resv mice exhibited lower fasting glucose levels, but only IH-Veh had increased homeostatic model assessment of insulin resistance index vs all 3 other groups. Leptin levels were preserved in IH-Veh but were significantly lower in IH-Resv. Reduced VWAT phosphorylated-AKT/AKT responses to insulin emerged in both Mes and Epi in IH-Veh but normalized in IH-Resv. Increases total macrophage counts and in M1 to M2 ratios occurred in IH-Veh Mes and Epi compared all other 3 groups. Thus, Resv ameliorates food intake and weight gain during IH exposures and markedly attenuates VWAT inflammation and insulin resistance, thereby providing a potentially useful adjunctive therapy for metabolic morbidity in the context of sleep apnea
Influence of the starting composition on the structural and superconducting properties of MgB2 phase
We report the preparation of MgB (0x0.5) compounds
with the nominal compositions. Single phase MgB was obtained for x=0
sample. For 0x0.5, MgB coexists with "MgB" and the amount
of MgB increases with x. With the increase of x, the lattice parameter
of "MgB" increases and the lattice parameter
decreases, correspondingly T of MgB decreases. The results
were discussed in terms of the presence of Mg vacancies or B interstitials in
the MgB structure. This work is helpful to the understanding of the
MgB films with different T, as well as the Mg site doping effect
for MgB.Comment: 11 pages, 4 figure
Possible tetraquark states in the invariant mass distribution
In this article, we assume that there exist hidden charmed tetraquark states
with the spin-parity , and calculate their masses with the QCD sum
rules. The numerical result indicates that the masses of the vector hidden
charmed tetraquark states are about or
, which are inconsistent with the experimental
data on the invariant mass distribution.
The hidden charmed mesons , or may be scalar hidden charmed
tetraquark states, hadro-charmonium resonances or molecular states.Comment: 12 pages, 4 figure
Heavy quarkonium: progress, puzzles, and opportunities
A golden age for heavy quarkonium physics dawned a decade ago, initiated by
the confluence of exciting advances in quantum chromodynamics (QCD) and an
explosion of related experimental activity. The early years of this period were
chronicled in the Quarkonium Working Group (QWG) CERN Yellow Report (YR) in
2004, which presented a comprehensive review of the status of the field at that
time and provided specific recommendations for further progress. However, the
broad spectrum of subsequent breakthroughs, surprises, and continuing puzzles
could only be partially anticipated. Since the release of the YR, the BESII
program concluded only to give birth to BESIII; the -factories and CLEO-c
flourished; quarkonium production and polarization measurements at HERA and the
Tevatron matured; and heavy-ion collisions at RHIC have opened a window on the
deconfinement regime. All these experiments leave legacies of quality,
precision, and unsolved mysteries for quarkonium physics, and therefore beg for
continuing investigations. The plethora of newly-found quarkonium-like states
unleashed a flood of theoretical investigations into new forms of matter such
as quark-gluon hybrids, mesonic molecules, and tetraquarks. Measurements of the
spectroscopy, decays, production, and in-medium behavior of c\bar{c}, b\bar{b},
and b\bar{c} bound states have been shown to validate some theoretical
approaches to QCD and highlight lack of quantitative success for others. The
intriguing details of quarkonium suppression in heavy-ion collisions that have
emerged from RHIC have elevated the importance of separating hot- and
cold-nuclear-matter effects in quark-gluon plasma studies. This review
systematically addresses all these matters and concludes by prioritizing
directions for ongoing and future efforts.Comment: 182 pages, 112 figures. Editors: N. Brambilla, S. Eidelman, B. K.
Heltsley, R. Vogt. Section Coordinators: G. T. Bodwin, E. Eichten, A. D.
Frawley, A. B. Meyer, R. E. Mitchell, V. Papadimitriou, P. Petreczky, A. A.
Petrov, P. Robbe, A. Vair
Study of J/Psi decays into eta Kstar Kstar-bar
We report the first observation of \mPJpsi \to \mPeta\mPKst\mAPKst decay in
a \mPJpsi sample of 58 million events collected with the BESII detector. The
branching fraction is determined to be . The selected signal event sample is further used to search for the
\mPY resonance through \mPJpsi \to \mPeta \mPY, \mPY\to\mPKst\mAPKst. No
evidence of a signal is seen. An upper limit of \mathrm{Br}(\mPJpsi \to \mPeta
\mPY)\cdot\mathrm{Br}(\mPY\to\mPKst\mAPKst) < 2.52\times 10^{-4} is set at the
90% confidence level.Comment: 11 pages, 4 figure
AcĂșmulo e formas de cobre e zinco no solo apĂłs aplicaçÔes sucessivas de dejeto lĂquido de suĂnos
Promoting inflammatory lymphangiogenesis by vascular endothelial growth factor-C (VEGF-C) aggravated intestinal inflammation in mice with experimental acute colitis
Angiogenesis and lymphangiogenesis are thought to play a role in the pathogenesis of inflammatory bowel diseases (IBD). However, it is not understood if inflammatory lymphangiogenesis is a pathological consequence or a productive attempt to resolve the inflammation. This study investigated the effect of lymphangiogenesis on intestinal inflammation by overexpressing a lymphangiogenesis factor, vascular endothelial growth factor-C (VEGF-C), in a mouse model of acute colitis. Forty eight-week-old female C57BL/6 mice were treated with recombinant adenovirus overexpressing VEGF-C or with recombinant VEGF-C156S protein. Acute colitis was then established by exposing the mice to 5% dextran sodium sulfate (DSS) for 7 days. Mice were evaluated for disease activity index (DAI), colonic inflammatory changes, colon edema, microvessel density, lymphatic vessel density (LVD), and VEGFR-3mRNA expression in colon tissue. When acute colitis was induced in mice overexpressing VEGF-C, there was a significant increase in colonic epithelial damage, inflammatory edema, microvessel density, and neutrophil infiltration compared to control mice. These mice also exhibited increased lymphatic vessel density (73.0±3.9 vs 38.2±1.9, P<0.001) and lymphatic vessel size (1974.6±104.3 vs 1639.0±91.5, P<0.001) compared to control mice. Additionally, the expression of VEGFR-3 mRNA was significantly upregulated in VEGF-C156S mice compared to DSS-treated mice after induction of colitis (42.0±1.4 vs 3.5±0.4, P<0.001). Stimulation of lymphangiogenesis by VEGF-C during acute colitis promoted inflammatory lymphangiogenesis in the colon and aggravated intestinal inflammation. Inflammatory lymphangiogenesis may have pleiotropic effects at different stages of IBD
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