4 research outputs found
Spin gap in the Quasi-One-Dimensional S=1/2 Antiferromagnet: Cu2(1,4-diazacycloheptane)2Cl4
Cu_{2}(1,4-diazacycloheptane)_{2}Cl_{4} contains double chains of spin 1/2
Cu^{2+} ions. We report ac susceptibility, specific heat, and inelastic neutron
scattering measurements on this material. The magnetic susceptibility,
, shows a rounded maximum at T = 8 K indicative of a low dimensional
antiferromagnet with no zero field magnetic phase transition. We compare the
data to exact diagonalization results for various one dimensional
spin Hamiltonians and find excellent agreement for a spin ladder with
intra-rung coupling meV and two mutually frustrating
inter-rung interactions: meV and meV. The
specific heat in zero field is exponentially activated with an activation
energy meV. A spin gap is also found through inelastic
neutron scattering on powder samples which identify a band of magnetic
excitations for meV. Using sum-rules we derive an
expression for the dynamic spin correlation function associated with
non-interacting propagating triplets in a spin ladder. The van-Hove
singularities of such a model are not observed in our scattering data
indicating that magnetic excitations in Cu_{2}(1,4-diazacycloheptane)_{2}Cl_{4}
are more complicated. For magnetic fields above T specific
heat data versus temperature show anomalies indicating a phase transition to an
ordered state below T = 1 K.Comment: 9 pages, 8 postscript figures, LaTeX, Submitted to PRB 8/4/97, e-mail
Comments to [email protected]
Morphological and Chemical Mechanisms of Elongated Mineral Particle Toxicities
Much of our understanding regarding the mechanisms for induction of disease following inhalation of respirable elongated mineral particles (REMP) is based on studies involving the biological effects of asbestos fibers. The factors governing the disease potential of an exposure include duration and frequency of exposures; tissue-specific dose over time; impacts on dose persistence from in vivo REMP dissolution, comminution, and clearance; individual susceptibility; and the mineral type and surface characteristics. The mechanisms associated with asbestos particle toxicity involve two facets for each particle's contribution: (1) the physical features of the inhaled REMP, which include width, length, aspect ratio, and effective surface area available for cell contact; and (2) the surface chemical composition and reactivity of the individual fiber/elongated particle. Studies in cell-free systems and with cultured cells suggest an important way in which REMP from asbestos damage cellular molecules or influence cellular processes. This may involve an unfortunate combination of the ability of REMP to chemically generate potentially damaging reactive oxygen species, through surface iron, and the interaction of the unique surfaces with cell membranes to trigger membrane receptor activation. Together these events appear to lead to a cascade of cellular events, including the production of damaging reactive nitrogen species, which may contribute to the disease process. Thus, there is a need to be more cognizant of the potential impact that the total surface area of REMP contributes to the generation of events resulting in pathological changes in biological systems. The information presented has applicability to inhaled dusts, in general, and specifically to respirable elongated mineral particles