Generation of two-mode field squeezing through selective dynamics in cavity QED

We propose a scheme for the generation of a two-mode field squeezed state in cavity QED. It is based on two-channel Raman excitations of a beam of three-level atoms with random arrival times by two classical fields and two high-Q resonator modes. It is shown that by suitably choosing the intensities and detunings of fields the dynamical processes can be selective and two-mode squeezing between the cavity modes can be generated at steady state. This proposal does not need the preparation of the initial states of atoms and cavity modes, and is robust against atomic spontaneous decay.Comment: 4 pages,2 figure

Incremental Learning for Multi-organ Segmentation with Partially Labeled Datasets

There exists a large number of datasets for organ segmentation, which are partially annotated, and sequentially constructed. A typical dataset is constructed at a certain time by curating medical images and annotating the organs of interest. In other words, new datasets with annotations of new organ categories are built over time. To unleash the potential behind these partially labeled, sequentially-constructed datasets, we propose to learn a multi-organ segmentation model through incremental learning (IL). In each IL stage, we lose access to the previous annotations, whose knowledge is assumingly captured by the current model, and gain the access to a new dataset with annotations of new organ categories, from which we learn to update the organ segmentation model to include the new organs. We give the first attempt to conjecture that the different distribution is the key reason for 'catastrophic forgetting' that commonly exists in IL methods, and verify that IL has the natural adaptability to medical image scenarios. Extensive experiments on five open-sourced datasets are conducted to prove the effectiveness of our method and the conjecture mentioned above

Functional cooperation of of IL-1β and RGS4 in the brachial plexus avulsion mediated brain reorganization

<p>Abstract</p> <p>Backgrounds</p> <p>There is considerable evidence that central nervous system is continuously modulated by activity, behavior and skill acquisition. This study is to examine the reorganization in cortical and subcortical regions in response to brachial plexus avulsion.</p> <p>Methods</p> <p>Adult C57BL/6 mice were divided into four groups: control, 1, 3 and 6 month of brachial plexus avulsion. IL-1β, IL-6 and RGS4 expression in cortex, brainstem and spinal cord were detected by BiostarM-140 s microarray and real-time PCR. RGS4 subcellular distribution and modulation were further analyzed by primary neuron culture and Western Blot.</p> <p>Results</p> <p>After 1, 3 and 6 months of brachial plexus avulsion, 49 (0 up, 49 down), 29 (17 up, 12 down), 13 (9 up, 4 down) genes in cerebral cortex, 40 (8 up, 32 down), 11 (7 up, 4 down), 137 (63 up, 74 down) in brainstem, 27 (14 up, 13 down), 33 (18 up, 15 down), 60 (29 up, 31 down) in spinal cord were identified. Among the regulated gene, IL-1β and IL-6 were sustainable enhanced in brain stem, while PKACβ and RGS4 were up-regulated throughout cerebral cortex, brainstem and spinal cord in 3 and 6 month of nerve injury. Intriguingly, subcellular distribution of RGS4 in above three regions was dependent on the functional correlation of PKA and IL-1β.</p> <p>Conclusion</p> <p>Data herein indicated that brachial plexus avulsion could efficiently initiate and perpetuate the brain reorganization. Network involved IL-1β and RGS4 signaling might implicate in the re-establish and strengthening of the local circuits at the cortical and subcortical levels.</p

Generation of two-mode entanglement between separated cavities

We propose a scheme for the generation of two-mode entangled states between two spatially separated cavities. It utilizes a two-level atom sequentially coupling to two high-Q cavities with a strong classical driving field. It is shown that by suitably choosing the intensities and detunings of the fields and coherent control of the dynamics, several different entangled states such as entangled coherent states and Bell states can be produced between the modes of the two cavities.Comment: 4pages,1figur

Boosting Multi-modal Model Performance with Adaptive Gradient Modulation

While the field of multi-modal learning keeps growing fast, the deficiency of the standard joint training paradigm has become clear through recent studies. They attribute the sub-optimal performance of the jointly trained model to the modality competition phenomenon. Existing works attempt to improve the jointly trained model by modulating the training process. Despite their effectiveness, those methods can only apply to late fusion models. More importantly, the mechanism of the modality competition remains unexplored. In this paper, we first propose an adaptive gradient modulation method that can boost the performance of multi-modal models with various fusion strategies. Extensive experiments show that our method surpasses all existing modulation methods. Furthermore, to have a quantitative understanding of the modality competition and the mechanism behind the effectiveness of our modulation method, we introduce a novel metric to measure the competition strength. This metric is built on the mono-modal concept, a function that is designed to represent the competition-less state of a modality. Through systematic investigation, our results confirm the intuition that the modulation encourages the model to rely on the more informative modality. In addition, we find that the jointly trained model typically has a preferred modality on which the competition is weaker than other modalities. However, this preferred modality need not dominate others. Our code will be available at https://github.com/lihong2303/AGM_ICCV2023.Comment: Accepted by ICCV202

Cullin4 E3 ubiquitin ligases regulate male gonocyte migration, proliferation and blood-testis barrier homeostasis

Ubiquitination, an essential posttranslational modification, plays fundamental roles during mammalian spermatogenesis. We previously reported the requirement of two Cullin 4 ubiquitin ligase family genes, Cullin 4a