322 research outputs found
Coulomb scattering lifetime of a two-dimensional electron gas
Motivated by a recent tunneling experiment in a double quantum-well system,
which reports an anomalously enhanced electronic scattering rate in a clean
two-dimensional electron gas, we calculate the inelastic quasiparticle lifetime
due to electron-electron interaction in a single loop dynamically screened
Coulomb interaction within the random-phase-approximation. We obtain excellent
quantitative agreement with the inelastic scattering rates in the tunneling
experiment without any adjustable parameter, finding that the reported large
( a factor of six) disagreement between theory and experiment arises from
quantitative errors in the existing theoretical work and from the off-shell
energy dependence of the electron self-energy.Comment: 11 pages, RevTex, figures included. Also available at
http://www-cmg.physics.umd.edu/~lzheng
Tunneling ``zero-bias'' anomaly in the quasi-ballistic regime
For the first time, we study the tunneling density of states (DOS) of the
interacting electron gas beyond the diffusive limit. A strong correction to the
DOS persists even at electron energies exceeding the inverse transport
relaxation time, which could not be expected from the well-known
Altshuler-Aronov-Lee (AAL) theory. This correction originates from the
interference between the electron waves scattered by an impurity and by the
Friedel oscillation this impurity creates. Account for such processes also
revises the AAL formula for the DOS in the diffusive limit.Comment: 4 pages, 2 .eps figures, submitted to Phys. Rev. Let
Anomalous Superconducting Properties and Field Induced Magnetism in CeCoIn5
In the heavy fermion superconductor CeCoIn5 (Tc=2.3K) the critical field is
large, anisotropic and displays hysteresis. The magnitude of the critical-field
anisotropy in the a-c plane can be as large as 70 kOe and depends on
orientation. Critical field measurements in the (110) plane suggest 2D
superconductivity, whereas conventional effective mass anisotropy is observed
in the (100) plane. Two distinct field-induced magnetic phases are observed: Ha
appears deep in the superconducting phase, while Hb intersects Hc2 at T=1.4 K
and extends well above Tc. These observations suggest the possible realization
of a direct transition from ferromagnetism to Fulde-Ferrel-Larkin-Ovchinnikov
superconductivity in CeCoIn5.Comment: 4 pages, 3 figure
Are markers of inflammation more strongly associated with risk for fatal than for nonfatal vascular events?
<p><b>Background:</b> Circulating inflammatory markers may more strongly relate to risk of fatal versus nonfatal cardiovascular disease (CVD) events, but robust prospective evidence is lacking. We tested whether interleukin (IL)-6, C-reactive protein (CRP), and fibrinogen more strongly associate with fatal compared to nonfatal myocardial infarction (MI) and stroke.</p>
<p><b>Methods and Findings:</b> In the Prospective Study of Pravastatin in the Elderly at Risk (PROSPER), baseline inflammatory markers in up to 5,680 men and women aged 70-82 y were related to risk for endpoints; nonfatal CVD (i.e., nonfatal MI and nonfatal stroke [n = 672]), fatal CVD (n = 190), death from other CV causes (n = 38), and non-CVD mortality (n = 300), over 3.2-y follow-up. Elevations in baseline IL-6 levels were significantly (p = 0.0009; competing risks model analysis) more strongly associated with fatal CVD (hazard ratio [HR] for 1 log unit increase in IL-6 1.75, 95% confidence interval [CI] 1.44-2.12) than with risk of nonfatal CVD (1.17, 95% CI 1.04-1.31), in analyses adjusted for treatment allocation. The findings were consistent in a fully adjusted model. These broad trends were similar for CRP and, to a lesser extent, for fibrinogen. The results were also similar in placebo and statin recipients (i.e., no interaction). The C-statistic for fatal CVD using traditional risk factors was significantly (+0.017; p<0.0001) improved by inclusion of IL-6 but not so for nonfatal CVD events (p = 0.20).</p>
<p><b>Conclusions:</b> In PROSPER, inflammatory markers, in particular IL-6 and CRP, are more strongly associated with risk of fatal vascular events than nonfatal vascular events. These novel observations may have important implications for better understanding aetiology of CVD mortality, and have potential clinical relevance.</p>
Mott Transition in Degenerate Hubbard Models: Application to Doped Fullerenes
The Mott-Hubbard transition is studied for a Hubbard model with orbital
degeneracy N, using a diffusion Monte-Carlo method. Based on general arguments,
we conjecture that the Mott-Hubbard transition takes place for U/W \propto
\sqrt{N}, where U is the Coulomb interaction and W is the band width. This is
supported by exact diagonalization and Monte-Carlo calculations. Realistic
parameters for the doped fullerenes lead to the conclusion that stoichiometric
A_3 C_60 (A=K, Rb) are near the Mott-Hubbard transition, in a correlated
metallic state.Comment: 4 pages, revtex, 1 eps figure included, to be published in Phys.Rev.B
Rapid Com
Carbon clusters near the crossover to fullerene stability
The thermodynamic stability of structural isomers of ,
, and , including
fullerenes, is studied using density functional and quantum Monte Carlo
methods. The energetic ordering of the different isomers depends sensitively on
the treatment of electron correlation. Fixed-node diffusion quantum Monte Carlo
calculations predict that a isomer is the smallest stable
graphitic fragment and that the smallest stable fullerenes are the
and clusters with and
symmetry, respectively. These results support proposals that a
solid could be synthesized by cluster deposition.Comment: 4 pages, includes 4 figures. For additional graphics, online paper
and related information see http://www.tcm.phy.cam.ac.uk/~prck
Practicing Imperfect Forgiveness
Forgiveness is typically regarded as a good thing - even a virtue - but acts of forgiveness can vary widely in value, depending on their context and motivation. Faced with this variation, philosophers have tended to reinforce everyday concepts of forgiveness with strict sets of conditions, creating ideals or paradigms of forgiveness. These are meant to distinguish good or praiseworthy instances of forgiveness from problematic instances and, in particular, to protect the self-respect of would-be forgivers. But paradigmatic forgiveness is problematic for a number of reasons, including its inattention to forgiveness as a gendered trait. We can account for the values and the risks associated with forgiving far better if we treat it as a moral practice and not an ideal
Groundnut Yield Response to Single Superphosphate, Calcitic Lime and Gypsum on Acid Granitic Sandy Soil
Protease-anti-protease compartmentalization in SARS-CoV-2 ARDS: Therapeutic implications
Background Interleukin-6 (IL-6) is elevated in SARS-CoV-2 infection. IL-6 regulates acute-phase proteins, such as alpha-1 antitrypsin (AAT), a key lung anti-protease. We investigated the protease-anti-protease balance in the circulation and pulmonary compartments in SARS-CoV-2 acute respiratory distress syndrome (ARDS) compared to non-SARS-CoV-2 ARDS (nsARDS) and the effects of tocilizumab (IL-6 receptor antagonist) on anti-protease defence in SARS-CoV-2 infection. Methods Levels and activity of AAT and neutrophil elastase (NE) were measured in plasma, airway tissue and tracheal secretions (TA) of people with SARS-CoV-2 ARDS or nsARDS. AAT and IL-6 levels were evaluated in people with moderate SARS-CoV-2 infection who received standard of care +/- tocilizumab. Findings AAT plasma levels doubled in SARS-CoV-2 ARDS. In lung parenchyma AAT levels were increased, as was the percentage of neutrophils involved in NET formation. A protease-anti-protease imbalance was detected in TA with active NE and no active AAT. The airway anti-protease, secretory leukoprotease inhibitor was decreased in SARS-CoV-2-infected lungs and cleaved in TA. In nsARDS, plasma AAT levels were elevated but TA samples had less AAT cleavage, with no detectable active NE in most samples. Induction of AAT in ARDS occurred mainly through IL-6. Tocilizumab down-regulated AAT during SARS-CoV-2 infection. Interpretation There is a protease-anti-protease imbalance in the airways of SARS-CoV-2-ARDS patients. This imbalance is a target for anti-protease therapy. Funding NIH Serological Sciences Network, National Heart, Lung, and Blood Institute and National Institute of Diabetes and Digestive and Kidney Diseases
Production, composition and fatty acid profile of milk and butter texture of dairy cows fed ground or pelleted concentrate with sunflower and/or lignosulfonate
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