Unlocking Reading Motivation

Teachers have been trying to solve the puzzle of what motivates students to become engaged with literacy tasks in the classroom for decades. Researchers and teachers have long established that motivation to read impacts reading ability, but how do we foster a classroom environment that encourages students to want to read (Wigfield, Allan, & Guthrie, 2000)? Donalyn Miller said she realized every lesson she taught, every assignment she gave, “must lead students away from [her] and toward their autonomy as literate people (Miller, 2010).” The elevation of self-efficacy in this way has been shown to increase a student’s reading engagement (McKool, 2007). In addition to fostering a student’s autonomy, another key to reading motivation that has been identified, but not thoroughly explored, is how a student’s social network impacts their motivation (Brozo & Flynt, 2008). Presenters will share literacy tasks that foster self-efficacy and collaboration, and the case for how they impact a student’s motivation for literacy engagement will be discussed (Chinn, Anderson & Waggoner, 2001). References Brozo, W., & Flynt, E. (2008). Content literacy: Motivating students to read in the content classroom: Six evidence-based principles.The Reading Teacher, 62(2), 172-174. Chinn, C., Anderson, R., & Waggoner, M. (2001). Patterns of discourse in two kinds of literature discussion. Reading Research Quarterly, 36(4), 378-411. McKool, S.S. (2007). Factors that influence the decision to read: An investigation of fifth grade students’ out-of-school reading habits. Reading Improvement, 44(3), 111-131 Miller, D. (2010). The Book Whisperer: Awakening The Inner Reader in Every Child. San Francisco, California: John Wiley & Sons. Wigfield, A. and J.T. Guthrie (2000). Engagement and motivation in reading. Handbook of Reading Research III, 403-422

Hubungan Religiusitas dengan Penerimaan Diri pada Masyarakat Miskin

Kesejahteraan, Kenyamanan hidup dan kesetaraan hidup dalam segi moril dan iman sangatlah di harapkan oleh setiap orang. Tetapi tidak dapat dirasakan untuk seseorang yang mengalami kemiskinan dengan pendapat rendah yang sebagian besar kehidupannya di rasakan dengan kerja keras lebih tanpa hasil yang memadai untuk kesejahteraannya. Religiusitas yang berada pada hati dan diri tiap individu merupakan salah satu faktor yang dapat mempengaruhi seorang individu untuk memperkuat imannya. Apabila religiusitas juga di bantu dengan faktor internal lainnya seperti penerimaan diri, dimana masyarakat miskin menerima keadaan bahwa dirinya miskin. Maka, seseorang yang mengalami kemiskinan dengan pendapatan rendah yaitu dimana warga tersebut diharuskan mencari nafkah untuk dirinya dan keluarga dapat berpikir positif lebih dari pada seseorang yang tidak di dukung dengan religiusitas dan penerimaan diri. Tujuan penelitian ini adalah untuk mengetahui seberapa besar hubungan religiusitas dengan penerimaan diri pada masyarakat miskin. Partisipan masyarakat miskin berdomisili di Depok, RT 002 RW 019 Kecamatan Pancoran Mas, Kelurahan Depok, Kampong Lio Depok. Teknik pengambilan sample penelitian ini menggunakan teknik purposive sampling. Jumlah total partisapan 30, yang berkarakteristik laki-laki dan perempuan dengan umur yang berusia 20-80 tahun. Penggunaan alat ukur dalam penelitian ini yaitu skala religiusitas dan skala penerimaan diri. Penganalisisan data dalam penelitian ini menggunakan uji korelasi product moment pearson. Uji hipotesis memperoleh signifikansi dengan p= 0,721 (p >0,05), dan nilai r= -0,068, dengan ini berarti bahwa hipotesis ditolak. Hal ini menunjukan bahwa tidak ada hubungan antara religiusitas dengan penerimaan diri pada warga miskin

Risk Factor Analysis of Bird Beak Occurrence after Thoracic Endovascular Aortic Repair

ObjectivesThe aim was to analyze the role played by anatomy and stent graft in the incidence of incomplete apposition to aortic arch.MethodsBetween 2007 and 2014 data including available and suitable computed tomographic angiography (CTA) imaging of patients who had undergone thoracic endovascular aortic repair were reviewed. The study included 80 patients (65 men, 54 ± 21 years) treated for traumatic aortic rupture (n = 27), thoracic aortic aneurysm (n = 15), type B aortic dissection (n = 24), penetrating aortic ulcer (n = 5), intramural hematoma (n = 2), aorto-oesophageal fistula (n = 2), and aortic mural thrombus (n = 5). Pre- and post-operative CTA images were analyzed to characterize bird beak in terms of length and angle, and to calculate aortic angulation within a 30 mm range at the proximal deployment zone.ResultsBird beak configuration was detected in 46 patients (57%): mean stent protrusion length was 16 mm (range: 8–29 mm) and mean bird beak angle was 20° (range: 7–40°). The bird beak effect was significantly more frequent after traumatic aortic rupture treatment (p = .05) and in landing zone 2 (p = .01). No influence of either stent graft type or generation, or degree of oversizing was observed (p = .29, p = .28, p = .81 respectively). However, the mean aortic angle of patients with bird beak was higher in the Pro-form group than that in the Zenith TX2 group (62° vs. 48°, p = .13). Multivariate analysis identified the aortic angle of the deployment zone as the unique independent risk factor of malapposition (HR = 1.05, 95% CI 1–1.10, p = .005). The cutoff value of 51° was found to be predictive of bird beak occurrence with a sensitivity of 58% and a specificity of 85%.ConclusionsAssessment of proximal landing zone morphology to avoid deployment zones generating an aortic angle of over 50° can be recommended to improve aortic curvature apposition with the current available devices

Age-Related Increases in the Shoulder Strength of High School Wrestlers

This is the publisher's version, also found at http://ehis.ebscohost.com/ehost/detail?vid=4&sid=34ab1967-2aea-457b-b261-e90e7b05e38c%40sessionmgr11&hid=2&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=s3h&AN=20752108The purpose of this investigation was to examine age-related differences in absolute and relative isokinetic shoulder strength of high school wrestlers. A total of 122 high school wrestlers (Mage = 16.31 ± 1.18 yrs) volunteered to be measured for arm flexion and extension strength at the shoulder joint using a Cybex II dynamometer at 30, 180, and 300°-s"'. The sample was divided into four age groups: 13.75-15.00 (n=22), 15.08-16.00 (n=27). 16.08-17.00 (n=34), and 17.08-18.83 years (n=39). The results ofthis study indicated significant increases in absolute and relative arm flexion and extension strength across age when covaried for BW and FFW. In addition, comparisons with previously published data indicated differences between muscle groups in the pattern of strength gains that were dependent upon the speed of muscular contraction and may have been influenced by fiber type distribution characteristics

Isokinetic Peak Torque in Young Wrestlers

This is the publisher's version, also found at http://ehis.ebscohost.com/ehost/detail?vid=3&sid=34ab1967-2aea-457b-b261-e90e7b05e38c%40sessionmgr11&hid=2&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=s3h&AN=20752369The purpose of the present study was to examine age-related changes in isokinetic leg flexion and extension peak torque (PT), PT/body weight (PT/ BW), and F*T/fat-free weight (PT/FFW) in young wrestlers. Male wrestlers (A^ = 108; age M ± SD = 11.3 + 1.5 years) volunteered to be measured for peak torque at 30, 180, and 300° • s'. In addition, underwater weighing was performed to determine body composition characteristics. The sample was divided into six age groups (8.1-8.9, n = 10; 9.0-9.9, n= 11; 10.0-10.9, n = 25; 11.0-11.9, n = 22; 12.0-12.9, n = 28; 13.0-13.9, n= 12), and repeated measures ANOVAs with Tukey post hoc comparisons showed increases across age for PT, PT/BW, and PT/FFW. The results of this study indicated that there were age-related increases in peak torque that could not be accounted for by changes in BW or FFW. It is possible that either an increase in muscle mass per unit of FFW, neural maturation, or both, contributes to the increase in strength across age in young male athletes

Biofilm formation and cellulose expression by <i>Bordetella avium</i> 197N, the causative agent of bordetellosis in birds and an opportunistic respiratory pathogen in humans

Although bacterial cellulose synthase (bcs) operons are widespread within the Proteobacteria phylum, subunits required for the partial-acetylation of the polymer appear to be restricted to a few γ-group soil, plant-associated and phytopathogenic pseudomonads, including Pseudomonas fluorescens SBW25 and several Pseudomonas syringae pathovars. However, a bcs operon with acetylation subunits has also been annotated in the unrelated β-group respiratory pathogen, Bordetella avium 197N. Our comparison of subunit protein sequences and GC content analyses confirms the close similarity between the B. avium 197N and pseudomonad operons and suggests that, in both cases, the cellulose synthase and acetylation subunits were acquired as a single unit. Using static liquid microcosms, we can confirm that B. avium 197N expresses low levels of cellulose in air–liquid interface biofilms and that biofilm strength and attachment levels could be increased by elevating c-di-GMP levels like the pseudomonads, but cellulose was not required for biofilm formation itself. The finding that B. avium 197N is capable of producing cellulose from a highly-conserved, but relatively uncommon bcs operon raises the question of what functional role this modified polymer plays during the infection of the upper respiratory tract or survival between hosts, and what environmental signals control its production

Development and Application of Ultra-Performance Liquid Chromatography-TOF MS for Precision Large Scale Urinary Metabolic Phenotyping

To better understand the molecular mechanisms underpinning physiological variation in human populations, metabolic phenotyping approaches are increasingly being applied to studies involving hundreds and thousands of biofluid samples. Hyphenated ultra-performance liquid chromatography-mass spectrometry (UPLC-MS) has become a fundamental tool for this purpose. However, the seemingly inevitable need to analyze large studies in multiple analytical batches for UPLC-MS analysis poses a challenge to data quality which has been recognized in the field. Herein, we describe in detail a fit-for-purpose UPLC-MS platform, method set, and sample analysis workflow, capable of sustained analysis on an industrial scale and allowing batch-free operation for large studies. Using complementary reversed-phase chromatography (RPC) and hydrophilic interaction liquid chromatography (HILIC) together with high resolution orthogonal acceleration time-of-flight mass spectrometry (oaTOF-MS), exceptional measurement precision is exemplified with independent epidemiological sample sets of approximately 650 and 1000 participant samples. Evaluation of molecular reference targets in repeated injections of pooled quality control (QC) samples distributed throughout each experiment demonstrates a mean retention time relative standard deviation (RSD) of <0.3% across all assays in both studies and a mean peak area RSD of <15% in the raw data. To more globally assess the quality of the profiling data, untargeted feature extraction was performed followed by data filtration according to feature intensity response to QC sample dilution. Analysis of the remaining features within the repeated QC sample measurements demonstrated median peak area RSD values of <20% for the RPC assays and <25% for the HILIC assays. These values represent the quality of the raw data, as no normalization or feature-specific intensity correction was applied. While the data in each experiment was acquired in a single continuous batch, instances of minor time-dependent intensity drift were observed, highlighting the utility of data correction techniques despite reducing the dependency on them for generating high quality data. These results demonstrate that the platform and methodology presented herein is fit-for-use in large scale metabolic phenotyping studies, challenging the assertion that such screening is inherently limited by batch effects. Details of the pipeline used to generate high quality raw data and mitigate the need for batch correction are provided

Melanoma cells break down LPA to establish local gradients that drive chemotactic dispersal.

The high mortality of melanoma is caused by rapid spread of cancer cells, which occurs unusually early in tumour evolution. Unlike most solid tumours, thickness rather than cytological markers or differentiation is the best guide to metastatic potential. Multiple stimuli that drive melanoma cell migration have been described, but it is not clear which are responsible for invasion, nor if chemotactic gradients exist in real tumours. In a chamber-based assay for melanoma dispersal, we find that cells migrate efficiently away from one another, even in initially homogeneous medium. This dispersal is driven by positive chemotaxis rather than chemorepulsion or contact inhibition. The principal chemoattractant, unexpectedly active across all tumour stages, is the lipid agonist lysophosphatidic acid (LPA) acting through the LPA receptor LPAR1. LPA induces chemotaxis of remarkable accuracy, and is both necessary and sufficient for chemotaxis and invasion in 2-D and 3-D assays. Growth factors, often described as tumour attractants, cause negligible chemotaxis themselves, but potentiate chemotaxis to LPA. Cells rapidly break down LPA present at substantial levels in culture medium and normal skin to generate outward-facing gradients. We measure LPA gradients across the margins of melanomas in vivo, confirming the physiological importance of our results. We conclude that LPA chemotaxis provides a strong drive for melanoma cells to invade outwards. Cells create their own gradients by acting as a sink, breaking down locally present LPA, and thus forming a gradient that is low in the tumour and high in the surrounding areas. The key step is not acquisition of sensitivity to the chemoattractant, but rather the tumour growing to break down enough LPA to form a gradient. Thus the stimulus that drives cell dispersal is not the presence of LPA itself, but the self-generated, outward-directed gradient