Comment on " a unified scheme for flavored mesons and baryons"

We would comment on the results of the paper "a unified scheme for flavored mesons and baryons" (P.C.Vinodkumar, J.N.Panandya, V.M.Bannur, and S.B.Khadkikar Eur. Phys. J. A4(1999)83), and point out some inconsistencies and mistakes in the work for solving the Dirac equation. In terms of an example for a single particle we investigate the reliability of the perturbative method for computing the Coulomb energy and discuss the contribution to the wavefunction at origin from the Coulomb potential. We conclude that the accuracy of their numerical results needs to be reconsidered.Comment: Latex file, 11page

Application of meso-2,3-Dimercaptosuccinic Acid Self-assembled Gold Electrode for Voltammetric Determination of Copper

Fabrication and electrochemical characteristics of the meso-2,3-dimercaptosuccinic acid (DMSA) self-assembled monolayer modified gold electrode were described. The modified electrode exhibited increased sensitivity and selectivity for CuII compared to the bare gold electrode by stripping voltammetry and the peak current was proportional to the concentration of CuII in the range of 8.0 10–7 1.2 10–4 mol/L with the detection limit of 1.1 10–7 mol/L. The influence of coexistent substances was investigated and the modified electrode showed good selectivity for copper determination. The DMSA/Au electrode was applied for CuII determination in a tap water sample with satisfactory results, with the recovery in the range from 99.7 to 101.1 %

Testing the Bell Inequality at Experiments of High Energy Physics

Besides using the laser beam, it is very tempting to directly testify the Bell inequality at high energy experiments where the spin correlation is exactly what the original Bell inequality investigates. In this work, we follow the proposal raised in literature and use the successive decays $J/\psi\to\gamma\eta_c\to \Lambda\bar\Lambda\to p\pi^-\bar p\pi^+$ to testify the Bell inequality. Our goal is twofold, namely, we first make a Monte-Carlo simulation of the processes based on the quantum field theory (QFT). Since the underlying theory is QFT, it implies that we pre-admit the validity of quantum picture. Even though the QFT is true, we need to find how big the database should be, so that we can clearly show deviations of the correlation from the Bell inequality determined by the local hidden variable theory. There have been some critiques on the proposed method, so in the second part, we suggest some improvements which may help to remedy the ambiguities indicated by the critiques. It may be realized at an updated facility of high energy physics, such as BES III.Comment: 16 pages, 5 figure

Ethyl 2-methyl-6-(propan-2-yl­amino)-4-sulfanyl­idene-3H,11H-pyrimido[1,6-c]quinazoline-1-carboxyl­ate

The title compound, C18H22N4O2S, contains a substituted pyrimidine ring fused to both a benzene ring and a substituted thioxopyrimidine ring. The pyrimidine and thioxopyrimidine rings adopt distorted chair conformations. In the crystal, adjacent mol­ecules are linked by pairs of N—H⋯S and N—H⋯O hydrogen bonds to generate centrosymmetric R 2 2(8) and R 2 2(16) loops, respectively. This combination leads to [100] chains of mol­ecules

Formation of a Salsolinol-like Compound, the Neurotoxin, 1-acetyl-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline, in a Cellular Model of Hyperglycemia and a Rat Model of Diabetes

There are statistical data indicating that diabetes is a risk factor for Parkinson\u27s disease (PD). Methylglyoxal (MG), a biologically reactive byproduct of glucose metabolism, the levels of which have been shown to be increase in diabetes, reacts with dopamine to form 1-acetyl-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline (ADTIQ); this formation may provide further insight into the connection between PD and diabetes. In this study, we investigated the role of ADTIQ in these two diseases to determine in an aim to enhance our understanding of the link between PD and diabetes. To this end, a cell model of hyperglycemia and a rat model of diabetes were established. In the cell model of hyperglycemia, compared with the control group, the elevated glucose levels promoted free hydroxyl radical formation (