THE FORAGE UTILIZATION OF WINTER PEA-CEREAL MIXTURE IN AGRICULTURE LOW-INPUT SYSTEM

Abstract HAKL, J., BRANT, V., MÁŠKOVÁ, K., NECKÁŘ, K., PIVEC, J.: The forage utilization of winter pea-cereal mixture in agriculture low-input system. Acta univ. agric. et silvic. Mendel. Brun., 2011, LIX, No. 5, pp. 47-52 The aim of this paper is to evaluate the forage utilization of winter catch crop in relation to yield and mixture composition. In 2006-2009, the plot experiment with winter pea in mixtures with rye and triticale was conducted under completely randomized design with four replicates. The productivity of mixture was above 10 t.ha −1 whilst the signifi cantly lowest value was observed for pea monoculture. The rye represented the most productive component in mixture but it achieved lower forage quality in comparison with triticale. The ratio of pea varied from 7 to 38 % in dependence on year and companion cereal. The triticale mixture provided higher ratio in comparison with rye and signifi cant diff erences between rye varieties were also detected. The quality of mixture was depended mainly on ratio of crops in the mixture. The ratio of pea signifi cantly increased crude protein content in mixture according to linear regression where crude protein = 9.56 + 0.11* weight percentage pea ratio (P < 0.000, R 2 = 0.89). Quality of pea forage was also infl uenced by companion crop where pea in mixture with rye achieved signifi cantly lower quality. The amount of weeds was highest in the pea monoculture and lowest in mixture with rye

N-acetylcysteine treatment prevents the up-regulation of MnSOD in chronically hypoxic rat hearts

Summary Chronic intermittent hypoxia (CIH) is associated with increased production of reactive oxygen species that contributes to the adaptive mechanism underlying the improved myocardial ischemic tolerance. The aim was to find out whether the antioxidative enzyme manganese superoxide dismutase (MnSOD) can play a role in CIH-induced cardioprotection. Adult male Wistar rats were exposed to intermittent hypobaric hypoxi

Ostadal B: MCC-134, a blocker of mitochondrial and opener of sarcolemmal ATP-sensitive K+ channels, abrogates cardioprotective effects of chronic hypoxia

Summary We examined the effect of MCC-134, a novel inhibitor of mitochondrial ATP-sensitive K + (mitoK ATP ) channels and activator of sarcolemmal ATP-sensitive K + (sarcK ATP ) channels, on cardioprotection conferred by adaptation to chronic hypoxia. Adult male Wistar rats were exposed to intermittent hypobaric hypoxia (7000 m, 8 h/day, 5-6 weeks) and susceptibility of their hearts to ventricular arrhythmias and myocardial infarction was evaluated in anesthetized openchest animals subjected to 20-min coronary artery occlusion and 3-h reperfusion on the day after the last hypoxic exposure. MCC-134 was administered intravenously 10 min before ischemia and 5 min before reperfusion in a total dose of 0.3 mg/kg or 3 mg/kg divided into two equal boluses. The infarct size (tetrazolium staining) was reduced from 59.2±4.4 % of the area at risk in normoxic controls to 43.2±3.3 % in the chronically hypoxic group. Chronic hypoxia decreased the reperfusion arrhythmia score from 2.4±0.5 in normoxic animals to 0.7±0.5. Both doses of MCC-134 completely abolished the antiarrhythmic protection (score 2.4±0.7 and 2.5±0.5, respectively) but only the high dose blocked the infarct size-limiting effect of chronic hypoxia (54.2±3.7 %). MCC-134 had no effect in the normoxic group. These results support the view that the opening of mitoK ATP channels but not sarcK ATP channels plays a crucial role in the mechanism by which chronic hypoxia improves cardiac tolerance to ischemia/reperfusion injury