Deletion of myeloid-PTP1B decreases MHC Class I expression and peptide presentation through an IL-10 dependent mechanism in response to LPS challenge

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Serum levels of RBP4 and adipose tissue levels of PTP1B are increased in obese men resident in northeast Scotland without associated changes in ER stress response genes

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Protein Tyrosine Phosphatase 1B (PTP1B) in the immune system

Journal not available online when checked 02/04/19. DOI: 10.14800/ics.965Peer reviewedPublisher PD

The Role of Inflammation in Diabetic Retinopathy

Funding Information: This paper was financially supported by Ministry of Health and Fondazione Roma. Publisher Copyright: © 2020 Verduci Editore s.r.l. All rights reserved. Copyright: Copyright 2020 Elsevier B.V., All rights reserved.Peer reviewedPublisher PD

Effects of dietary restriction on metabolic and cognitive health

M. S. M. is a recipient of the Elphinstone Scholarship of the University of Aberdeen as well as Institute of Medical Sciences postdoctoral studentship. Work in B. P. and M. D. laboratories is funded by Alzheimer's Research UK, British Heart Foundation and Diabetes UK. Tenovus Scotland and BBSRC DTP studentship funded the published work on methionine restriction in M. D. laboratory.Peer reviewedPostprin

How stra(i)nge are your controls? : A comparative analysis of metabolic phenotypes in commonly used C57BL/6 substrains

Open Access via the PLOS Agreement Funding: The authors received no specific funding for this work.Peer reviewedPublisher PD

Impact of Diabetes on COVID-19 mortality and hospital outcomes, a global perspective : An umbrella systematic review and meta-analysis

OA via Wiley agreementPeer reviewedPublisher PD

Female adipose tissue-specific Bscl2 knockout mice develop only moderate metabolic dysfunction when housed at thermoneutrality and fed a high-fat diet

The authors would like to thank the staff at the University of Aberdeen’s Medical Research Facility. We are very grateful for the gift of the Adiponectin-Cre mice from Dr. Evan Rosen (Beth Israel Deaconess Medical Centre and Harvard Medical School, Boston, USA). Work was supported by the Medical Research Council (GDM/JJR; MR/L002620/1, MC/PC/15077), the Biotechnology and Biological Sciences Research Council (JJR; BB/K017772/1), the British Heart Foundation (MD; PG/14/43/30889) and The Agency for Science, Technology and Research, Singapore (A*STAR) (WH). The datasets generated during and/or analysed during the current study are available from the corresponding author on reasonable request.Peer reviewedPublisher PD

Knock-in of Mutated hTAU Causes Insulin Resistance, Inflammation and Proteostasis Disturbance in a Mouse Model of Frontotemporal Dementia

Open Access via Springer Compact Agreement.Peer reviewedPublisher PD