Demonstration of the double Q^2-rescaling model

In this paper we have demonstrated the double Q^2-rescaling model (DQ^2RM) of parton distribution functions of nucleon bounded in nucleus. With different x-region of l-A deep inelastic scattering process we take different approach: in high x-region (0.1\le x\le 0.7) we use the distorted QCD vacuum model which resulted from topologically multi -connected domain vacuum structure of nucleus; in low x-region (10^{-4}\le x\le10^{-3}) we adopt the Glauber (Mueller) multi- scattering formula for gluon coherently rescattering in nucleus. From these two approach we justified the rescaling parton distribution functions in bound nucleon are in agreement well with those we got from DQ^2RM, thus the validity for this phenomenologically model are demonstrated.Comment: 19 page, RevTex, 5 figures in postscrip

J/ψ+jetJ/\psi + jet diffractive production in the direct photon process at HERA

We present a study of $J/\psi + jet$ diffractive production in the direct photon process at HERA based on the factorization theorem for lepton-induced hard diffractive scattering and the factorization formalism of the nonrelativistic QCD (NRQCD) for quarkonia production. Using the diffractive gluon distribution function extracted from HERA data on diffractive deep inelastic scattering and diffractive dijet photon production, we show that this process can be studied at HERA with present integrated luminosity, and can give valuable insights in the color-octet mechanism for heavy quarkonia production.Comment: Revtex, 21 pages, 7 EPS figure

In Vivo

Diester diterpenoid alkaloids (DDAs), such as aconitine (AC), mesaconitine (MA), and hypaconitine (HA), are both pharmacologically active compounds and toxic ingredients in a traditional Chinese herb, the Aconitum species. Many DDA metabolism studies have been performed to explore mechanisms for reducing toxicity in these compounds and in Aconitum species extracts for safe clinical administration. In this review, we summarize recent progress on the metabolism of toxic AC, MA, and HA and corresponding monoester diterpenoid alkaloids (MDAs) in the gastrointestinal tract and liver in different animal species and humans in vivo and/or in vitro, where these alkaloids are primarily metabolized by cytochrome P450 enzymes, carboxylesterases, and intestinal bacteria, which produces phase I metabolites, ester hydrolysed products, and lipoalkaloids. Furthermore, we classify metabolites detected in the blood and urine, where the aforementioned metabolites are absorbed and excreted. Less toxic MDAs and nontoxic alcohol amines are the primary DDA metabolites detected in the blood. Most other DDAs metabolites produced in the intestine and liver detected in the urine have not been reported in the blood. We propose an explanation for this nonconformity. Finally, taking AC, for instance, we generalize a process of toxicity reduction in the body after oral AC administration for the first time