Electrically tunable waveguide laser based on ferroelectric liquid crystal

This article may be downloaded for personal use only. Any other use requires prior permission of the author and AIP Publishing. This article appeared in Masahiro Kasano, Masanori Ozaki, and Katsumi Yoshino, Appl. Phys. Lett. 82, 4026 (2003) and may be found at https://doi.org/10.1063/1.1580992

Tunable lasing in doped liquid crystals with one dimensional periodic structure

Optical Science and Technology, SPIE's 48th Annual Meeting, 2003, San Diego, California, United StatesMasanori Ozaki, Masahiro Kasano, Kazuhiro Funamoto, Ryotaro Ozaki, Tatsunosuke Matsui, and Katsumi Yoshino "Tunable lasing in doped liquid crystals with one-dimensional periodic structure", Proc. SPIE 5213, Liquid Crystals VII, (5 January 2004). DOI: https://doi.org/10.1117/12.50918

6-(Methylsulfinyl) Hexyl Isothiocyanate Inhibits IL-6 and CXCL10 Production in TNF-α-Stimulated Human Oral Epithelial Cells

6-(Methylsulfinyl) hexyl isothiocyanate (6-MSITC) is a bioactive substance found in wasabi (Wasabia japonica) and has been reported to have some bioactive effects including anticancer and antioxidant effects. However, there are no reports on its effects on periodontal resident cells, and many points remain unclear. In this study, we aimed to investigate whether 6-MSITC exerts anti-inflammatory effects on human oral epithelial cells, including effects on signal transduction pathway activation. 6-MSITC inhibited interleukin (IL)-6 and C-X-C motif chemokine ligand 10 (CXCL10) production in TNF-α-stimulated TR146 cells, which are a human oral epithelial cell line. Moreover, we found that 6-MSITC could suppress signal transducer and activator of transcription (STAT)3, nuclear factor (NF)-κB, and p70S6 kinase (p70S6K)-S6 ribosomal protein (S6) pathways activation in TNF-α-stimulated TR146 cells. Furthermore, STAT3 and NF-κB inhibitors could suppress IL-6 and CXCL10 production in TNF-α-treated TR146 cells. In summary, 6-MSITC could decrease IL-6 and CXCL10 production in human oral epithelial cell by inhibiting STAT3 and NF-κB activation