4,593 research outputs found

    Properties of canonical determinants and a test of fugacity expansion for finite density lattice QCD with Wilson fermions

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    We analyze canonical determinants, i.e., grand canonical determinants projected to a fixed net quark number. The canonical determinants are the coefficients in a fugacity expansion of the grand canonical determinant and we evaluate them as the Fourier moments of the grand canonical determinant with respect to imaginary chemical potential, using a dimensional reduction technique. The analysis is done for two mass-degenerate flavors of Wilson fermions at several temperatures below and above the confinement/deconfinement crossover. We discuss various properties of the canonical determinants and analyse the convergence of the fugacity series for different temperatures.Comment: Typo removed, paragraph added in the discussion. Version to appear in Phys. Rev.

    The topology of U-duality (sub-)groups

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    We discuss the topology of the symmetry groups appearing in compactified (super-)gravity, and discuss two applications. First, we demonstrate that for 3 dimensional sigma models on a symmetric space G/H with G non-compact and H the maximal compact subgroup of G, the possibility of oxidation to a higher dimensional theory can immediately be deduced from the topology of H. Second, by comparing the actual symmetry groups appearing in maximal supergravities with the subgroups of SL(32,R) and Spin(32), we argue that these groups cannot serve as a local symmetry group for M-theory in a formulation of de Wit-Nicolai type.Comment: 18 pages, LaTeX, 1 figure, 2 table

    Structural Color 3D Printing By Shrinking Photonic Crystals

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    The rings, spots and stripes found on some butterflies, Pachyrhynchus weevils, and many chameleons are notable examples of natural organisms employing photonic crystals to produce colorful patterns. Despite advances in nanotechnology, we still lack the ability to print arbitrary colors and shapes in all three dimensions at this microscopic length scale. Commercial nanoscale 3D printers based on two-photon polymerization are incapable of patterning photonic crystal structures with the requisite ~300 nm lattice constant to achieve photonic stopbands/ bandgaps in the visible spectrum and generate colors. Here, we introduce a means to produce 3D-printed photonic crystals with a 5x reduction in lattice constants (periodicity as small as 280 nm), achieving sub-100-nm features with a full range of colors. The reliability of this process enables us to engineer the bandstructures of woodpile photonic crystals that match experiments, showing that observed colors can be attributed to either slow light modes or stopbands. With these lattice structures as 3D color volumetric elements (voxels), we printed 3D microscopic scale objects, including the first multi-color microscopic model of the Eiffel Tower measuring only 39-microns tall with a color pixel size of 1.45 microns. The technology to print 3D structures in color at the microscopic scale promises the direct patterning and integration of spectrally selective devices, such as photonic crystal-based color filters, onto free-form optical elements and curved surfaces

    Observation of a Griffiths-like phase in the paramagnetic regime of ErCo_2

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    A systematic x-ray magnetic circular dichroism study of the paramagnetic phase of ErCo2 has recently allowed to identify the inversion of the net magnetization of the Co net moment with respect to the applied field well above the ferrimagnetic ordering temperature, Tc. The study of small angle neutron scattering measurements has also shown the presence of short range order correlations in the same temperature region. This phenomenon, which we have denoted parimagnetism, may be related with the onset of a Griffiths-like phase in paramagnetic ErCo2. We have measured ac susceptibility on ErCo2 as a function of temperature, applied field, and excitation frequency. Several characteristics shared by systems showing a Griffiths phase are present in ErCo2, namely the formation of ferromagnetic clusters in the disordered phase, the loss of analyticity of the magnetic susceptibility and its extreme sensitivity to an applied magnetic field. The paramagnetic susceptibility allows to establish that the magnetic clusters are only formed by Co moments as well as the intrinsic nature of those Co moments

    Triplet‐Induced Lesion Formation at CpT and TpC Sites in DNA

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    UV irradiation induces DNA lesions particularly at dipyrimidine sites. Using time‐resolved UV pump (250 nm) and mid‐IR probe spectroscopy the triplet pathway of cyclobutane pyrimidine dimer (CPD) formation within TpC and CpT sequences was studied. The triplet state is initially localized at the thymine base but decays with 30 ns under formation of a biradical state extending over both bases of the dipyrimidine. Subsequently this state either decays back to the electronic ground state on the 100 ns time scale or forms a cyclobutane pyrimidine dimer lesion (CPD). Stationary IR spectroscopy and triplet sensitization via 2′‐methoxyacetophenone (2‐M) in the UVA range shows that the lesions are formed with an efficiency of approximately 1.5 %. Deamination converts the cytosine moiety of the CPD lesions on the time scale of 10 hours into uracil which gives CPD(UpT) and CPD(TpU) lesions in which the coding potential of the initial cytosine base is vanished

    Hard Thermal Loops, Static Response and the Composite Effective Action

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    First, we investigate the static non-Abelian Kubo equation. We prove that it does not possess finite energy solutions; thereby we establish that gauge theories do not support hard thermal solitons. A similar argument shows that "static" instantons are absent. In addition, we note that the static equations reproduce the expected screening of the non-Abelian electric field by a gauge invariant Debye mass m=gT sqrt((N+N_F/2)/3). Second, we derive the non-Abelian Kubo equation from the composite effective action. This is achieved by showing that the requirement of stationarity of the composite effective action is equivalent, within a kinematical approximation scheme, to the condition of gauge invariance for the generating functional of hard thermal loops.Comment: 17 pages, MIT preprint CTP#2261. An Appendix [including one (appended) PS figure] presenting a numerical analysis of the static solutions has been included. A note relating our approach to alternative ones has been added. We have also added references and comments in Section II

    Ethylene-mediated nitric oxide depletion pre-adapts plants to hypoxia stress

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    Timely perception of adverse environmental changes is critical for survival. Dynamic changes in gases are important cues for plants to sense environmental perturbations, such as submergence. In Arabidopsis thaliana, changes in oxygen and nitric oxide (NO) control the stability of ERFVII transcription factors. ERFVII proteolysis is regulated by the N-degron pathway and mediates adaptation to flooding-induced hypoxia. However, how plants detect and transduce early submergence signals remains elusive. Here we show that plants can rapidly detect submergence through passive ethylene entrapment and use this signal to pre-adapt to impending hypoxia. Ethylene can enhance ERFVII stability prior to hypoxia by increasing the NO-scavenger PHYTOGLOBIN1. This ethylene-mediated NO depletion and consequent ERFVII accumulation pre-adapts plants to survive subsequent hypoxia. Our results reveal the biological link between three gaseous signals for the regulation of flooding survival and identifies key regulatory targets for early stress perception that could be pivotal for developing flood-tolerant crops

    Anisotropic Magnetoresistance in Ga1x_{1-x}Mnx_xAs

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    We have measured the magnetoresistance in a series of Ga1x_{1-x}Mnx_xAs samples with 0.033x\le x \le 0.053 for three mutually orthogonal orientations of the applied magnetic field. The spontaneous resistivity anisotropy (SRA) in these materials is negative (i.e. the sample resistance is higher when its magnetization is perpendicular to the measuring current than when the two are parallel) and has a magnitude on the order of 5% at temperatures near 10K and below. This stands in contrast to the results for most conventional magnetic materials where the SRA is considerably smaller in magnitude for those few cases in which a negative sign is observed. The magnitude of the SRA drops from its maximum at low temperatures to zero at TC_C in a manner that is consistent with mean field theory. These results should provide a significant test for emerging theories of transport in this new class of materials.Comment: 4 pages with 4 figures. Submitted to Physical Review

    The protein kinase C inhibitor, Ro-31-7459, is a potent activator of ERK and JNK MAP kinases in HUVECs and yet inhibits cyclic AMP-stimulated <i>SOCS-3</i> gene induction through inactivation of the transcription factor c-Jun

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    Induction of the suppressor of cytokine signalling 3 (SOCS-3) gene is vital to the normal control of inflammatory signalling. In order to understand these processes we investigated the role of the proto-oncogene component of the AP-1 transcription factor complex, c-Jun, in the regulation of SOCS-3 gene induction. We found that cyclic AMP stimulation of HUVECs promoted phosphorylation and activation of JNK MAP kinase and its substrate c-Jun. The JNK responsive element of the human SOCS-3 promoter mapped to a putative AP-1 site within 1000 bp of the transcription start site. The PKC inhibitors, GF-109203X, Gö-6983 and Ro-317549, were all found to inhibit AP-1 transcriptional activity, transcriptional activation of this minimal SOCS-3 promoter and SOCS-3 gene induction in HUVECs. Interestingly, Ro-317549 treatment was also found to promote PKC-dependent activation of ERK and JNK MAP kinases and promote JNK-dependent hyper-phosphorylation of c-Jun, whereas GF-109203X and Gö-6983 had little effect. Despite this, all three PKC inhibitors were found to be effective inhibitors of c-Jun DNA-binding activity. The JNK-dependent hyper-phosphorylation of c-Jun in response to Ro-317549 treatment of HUVECs does therefore not interfere with its ability to inhibit c-Jun activity and acts as an effective inhibitor of c-Jun-dependent SOCS-3 gene induction
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