7 research outputs found

    Demographic and lifestyle characteristics by quartiles of fruit and vegetable consumption of 20,069 Dutch participants<sup>1</sup>.

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    1<p>Data are presented as mean (SD) or percentages.</p>2<p>100 gram of fruit and vegetables equals 1 medium-sized piece of fruit or 1 cup cut-up raw fruit, fruit juice, cooked vegetables, or 2 cups raw leafy vegetables.</p>3<p>Low educational level is defined as primary school and lower, intermediate general education.</p>4<p>High alcohol consumption is defined as >1 glass per day in women and >2 glasses per day in men.</p>5<p>Physically active on 5 d/wk and ≥0.5 hr/d with an intensity of ≥4 metabolic equivalents. In sub sample of participants enrolled from 1994 onwards (<i>n</i> = 15,433).</p>6<p>Family history of AMI is defined as occurrence of AMI before 55y of the father or before 65y of the mother.</p>7<p>Fish consumption is defined as the highest quartile of fish intake (median: 17 g/d, i.e. ∼1 portion of fish/week).</p

    Hazard ratios and 95% confidence intervals of CHD incidence by quartiles of fruit. and vegetable intake of 20,069 Dutch participants<sup>1</sup>.

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    1<p>Hazard ratios (95% CIs) were obtained from Cox proportional hazards models. Model 1 was adjusted for age and gender (<i>n</i> = 20,069). Model 2 was the same as model 1 with additional adjustments for energy intake, alcohol intake, smoking status, educational level, dietary supplement use, use of hormone replacement therapy, family history of MI before 60, BMI (<i>n</i> = 19,819). Model 3 was adjusted as model 2 with additional adjustments for intake of fish, whole grain foods and processed meat (<i>n</i> = 19,819).</p>2<p>Reference group.</p>3<p>Additionally adjusted for processed fruit and vegetable intake.</p>4<p>Additionally adjusted for raw fruit and vegetable intake.</p

    Relation between baseline kidney function and all-cause mortality with 95%-confidence intervals (dotted lines) among 4561 post-myocardial infarction patients.

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    <p>Hazard ratios for all-cause mortality depending on kidney function were modeled by separate restricted cubic splines for cystatin C-based kidney function in a Cox-regression model. The model was adjusted for n-3 fatty acids treatment, age, sex, diabetes, current smoking, ratio serum cholesterol-HDL, statin-use, use of anti-hypertensive medication, systolic and diastolic blood pressure. An eGFR of 120 ml/min/1.73m<sup>2</sup> was taken as the reference point (hazard ratio 1).</p

    Relation between baseline kidney function and non-cardiovascular-non-cancer mortality with 95%-confidence intervals (dotted lines) among 4561 post-myocardial infarction patients.

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    <p>Hazard ratios for non-cardiovascular-non-cancer causes of mortality depending on kidney function were modeled by separate restricted cubic splines for cystatin C-based kidney function in a Cox-regression model. The model was adjusted for n-3 fatty acids treatment, age, sex, diabetes, current smoking, ratio serum cholesterol-HDL, statin-use, use of anti-hypertensive medication, systolic and diastolic blood pressure. An eGFR of 120 ml/min/1.73m<sup>2</sup> was taken as the reference point (hazard ratio 1).</p

    Relation between baseline kidney function and cardiovascular mortality with 95%-confidence intervals (dotted lines) among 4561 post-myocardial infarction patients.

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    <p>Hazard ratios for cardiovascular mortality depending on kidney function were modeled by separate restricted cubic splines for cystatin C based-kidney function in a Cox-regression model. The model was adjusted for n-3 fatty acids treatment, age, sex, diabetes, current smoking, ratio serum cholesterol-HDL, statin-use, use of anti-hypertensive medication, systolic and diastolic blood pressure. An eGFR of 120 ml/min/1.73m<sup>2</sup> was taken as the reference point (hazard ratio 1).</p
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