6 research outputs found
Decision Fusion Network with Perception Fine-tuning for Defect Classification
Surface defect inspection is an important task in industrial inspection. Deep
learning-based methods have demonstrated promising performance in this domain.
Nevertheless, these methods still suffer from misjudgment when encountering
challenges such as low-contrast defects and complex backgrounds. To overcome
these issues, we present a decision fusion network (DFNet) that incorporates
the semantic decision with the feature decision to strengthen the decision
ability of the network. In particular, we introduce a decision fusion module
(DFM) that extracts a semantic vector from the semantic decision branch and a
feature vector for the feature decision branch and fuses them to make the final
classification decision. In addition, we propose a perception fine-tuning
module (PFM) that fine-tunes the foreground and background during the
segmentation stage. PFM generates the semantic and feature outputs that are
sent to the classification decision stage. Furthermore, we present an
inner-outer separation weight matrix to address the impact of label edge
uncertainty during segmentation supervision. Our experimental results on the
publicly available datasets including KolektorSDD2 (96.1% AP) and
Magnetic-tile-defect-datasets (94.6% mAP) demonstrate the effectiveness of the
proposed method
Global Context Aggregation Network for Lightweight Saliency Detection of Surface Defects
Surface defect inspection is a very challenging task in which surface defects
usually show weak appearances or exist under complex backgrounds. Most
high-accuracy defect detection methods require expensive computation and
storage overhead, making them less practical in some resource-constrained
defect detection applications. Although some lightweight methods have achieved
real-time inference speed with fewer parameters, they show poor detection
accuracy in complex defect scenarios. To this end, we develop a Global Context
Aggregation Network (GCANet) for lightweight saliency detection of surface
defects on the encoder-decoder structure. First, we introduce a novel
transformer encoder on the top layer of the lightweight backbone, which
captures global context information through a novel Depth-wise Self-Attention
(DSA) module. The proposed DSA performs element-wise similarity in channel
dimension while maintaining linear complexity. In addition, we introduce a
novel Channel Reference Attention (CRA) module before each decoder block to
strengthen the representation of multi-level features in the bottom-up path.
The proposed CRA exploits the channel correlation between features at different
layers to adaptively enhance feature representation. The experimental results
on three public defect datasets demonstrate that the proposed network achieves
a better trade-off between accuracy and running efficiency compared with other
17 state-of-the-art methods. Specifically, GCANet achieves competitive accuracy
(91.79% , 93.55% , and 97.35% ) on
SD-saliency-900 while running 272fps on a single gpu
Scanning Tunneling Microscopic Observation of the Atomic Structure of GaAs(001) Surface Grown by Metalorganic Vapor Phase Epitaxy(STM-GaAs)
We present the first atomically resolved scanning-tunneling micrographs of GaAs(001) surfaces prepared by metalorganic vapor-phase epitaxy (MOVPE). Thin films deposited in an MOVPE reactor were transferred to an ultra high vacuum system without air exposure. After heating the samples from 450 to 620℃, high-quality images of the (2x4)/c(2x8), (1x6)/(2x6) and (4x2)/c(8x2) reconstructions were obtained
Bacterial effector restricts liquid-liquid phase separation of ZPR1 to antagonize host UPRER
Summary: How pathogens manipulate host UPRER to mediate immune evasion is largely unknown. Here, we identify the host zinc finger protein ZPR1 as an interacting partner of the enteropathogenic E. coli (EPEC) effector NleE using proximity-enabled protein crosslinking. We show that ZPR1 assembles via liquid-liquid phase separation (LLPS) in vitro and regulates CHOP-mediated UPRER at the transcriptional level. Interestingly, in vitro studies show that the ZPR1 binding ability with K63-ubiquitin chains, which promotes LLPS of ZPR1, is disrupted by NleE. Further analyses indicate that EPEC restricts host UPRER pathways at the transcription level in a NleE-ZPR1 cascade-dependent manner. Together, our study reveals the mechanism by which EPEC interferes with CHOP-UPRER via regulating ZPR1 to help pathogens escape host defense