11 research outputs found
Confining QCD Strings, Casimir Scaling, and a Euclidean Approach to High-Energy Scattering
We compute the chromo-field distributions of static color-dipoles in the
fundamental and adjoint representation of SU(Nc) in the loop-loop correlation
model and find Casimir scaling in agreement with recent lattice results. Our
model combines perturbative gluon exchange with the non-perturbative stochastic
vacuum model which leads to confinement of the color-charges in the dipole via
a string of color-fields. We compute the energy stored in the confining string
and use low-energy theorems to show consistency with the static quark-antiquark
potential. We generalize Meggiolaro's analytic continuation from parton-parton
to gauge-invariant dipole-dipole scattering and obtain a Euclidean approach to
high-energy scattering that allows us in principle to calculate S-matrix
elements directly in lattice simulations of QCD. We apply this approach and
compute the S-matrix element for high-energy dipole-dipole scattering with the
presented Euclidean loop-loop correlation model. The result confirms the
analytic continuation of the gluon field strength correlator used in all
earlier applications of the stochastic vacuum model to high-energy scattering.Comment: 65 pages, 13 figures, extended and revised version to be published in
Phys. Rev. D (results unchanged, 2 new figures, 1 new table, additional
discussions in Sec.2.3 and Sec.5, new appendix on the non-Abelian Stokes
theorem, old Appendix A -> Sec.3, several references added
Prolonged head down bed rest-induced inactivity impairs tonic autonomic regulation while sparing oscillatory cardiovascular rhythms in healthy humans
Background Physical inactivity represents a major risk for cardiovascular disorders, such as hypertension, myocardial infarction or sudden death; however, underlying mechanisms are not clearly elucidated. Clinical and epidemiological investigations suggest, beyond molecular changes, the possibility of an induced impairment in autonomic cardiovascular regulation. However, this hypothesis has not been tested directly. Methods Accordingly, we planned a study with noninvasive, minimally intrusive, techniques on healthy volunteers. Participants were maintained for 90 days strictly in bed, 24 h a day, in head-down (-6 degrees) position (HDBR). Physical activity was thus virtually abolished for the entire period of HDBR. We examined efferent muscle sympathetic nerve activity, as a measure of vascular sympathetic control, baroreceptor reflex sensitivity, heart rate variability (assessing cardiovagal regulation), RR and systolic arterial pressure and low-frequency and high-frequency normalized components (as a window on central oscillatory regulation). Measures were obtained at rest and during simple maneuvers (moderate handgrip, lower body negative pressure and active standing) to assess potential changes in autonomic cardiovascular responsiveness to standard stimuli and the related oscillatory profiles. Results HDBR transiently reduced muscle sympathetic nerve activity, RR, heart rate variability and baroreceptor reflex sensitivity late during HDBR or early during the recovery phase. Conversely, oscillatory profiles of RR and systolic arterial pressure variability were maintained throughout. Responsiveness to test stimuli was also largely maintained. Conclusion Prolonged inactivity as induced by HDBR in healthy volunteers reduces both cardiovagal and vascular sympathetic regulation, while largely maintaining peripheral responsiveness to standardized stimuli and sparing the functional structure of central oscillatory cardiovascular regulation. J Hypertens 27:551-561 (C) 2009 Wolters Kluwer Health/Lippincott Williams & Wilkins