Role of the Coulomb and the vector-isovector ρ\rho potentials in the isospin asymmetry of nuclear pseudospin

We investigate the role of the Coulomb and the vector-isovector $\rho$ potentials in the asymmetry of the neutron and proton pseudospin splittings in nuclei. To this end, we solve the Dirac equation for the nucleons using central vector and scalar potentials with Woods-Saxon shape and $Z$ and $N-Z$ dependent Coulomb and $\rho$ potentials added to the vector potential. We study the effect of these potentials on the energy splittings of proton and neutron pseudospin partners along a Sn isotopic chain. We use an energy decomposition proposed in a previous work to assess the effect of a pseudospin-orbit potential on those splittings. We conclude that the effect of the Coulomb potential is quite small and the $\rho$ potential gives the main contribution to the observed isospin asymmetry of the pseudospin splittings. This isospin asymmetry results from a cancellation of the various energy terms and cannot be attributed only to the pseudospin-orbit term, confirming the dynamical character of this symmetry pointed out in previous works.Comment: 9 pages, 11 figures, uses revtex4; title was changed and several small corrections were made throughout the tex

Hyperphagia and hyperleptinemia induced by low-protein, high-carbohydrate diet is reversed at a later stage of development in rats

Abstract This study investigated whether increased food intake after 15 days of low-protein, high-carbohydrate (LPHC) and its normalization in the later period of development change the content of key proteins related to leptin or adiponectin signaling in the hypothalamus. Male rats were divided into five groups: Control groups received a control diet (17% protein, 63% carbohydrate) for 15 (C15) or 45 (C45) days; LPHC groups received an LPHC diet (6% protein, 74% carbohydrate) for 15 (LPHC15) or 45 (LPHC45) days; and Reverse group (R): received LPHC diet for 15 days followed by control diet for another 30 days. The LPHC15 group showed increased adiposity index, leptin level, and adiponectin level, as well as decreased the leptin receptor (ObRb) and pro-opiomelanocortin (POMC) content in the hypothalamus compared with the C15 group. LPHC diet for 45 days or diet reversion (R group) rescued these alterations, except the adiponectin level in LPHC45 rats, which was higher. In summary, LPHC diet reduced hypothalamic leptin action by diminishing ObRb and POMC levels, leading to hyperphagia and adiposity body. Medium-term administration of LPHC diet or reverting to control diet restored the levels of these proteins, thereby improving body lipid mass rearrangement in adulthood