On gaugino condensation in the effective theory

We analyze the gaugino condensation in the effective theory for N=1 SU(N) Supersymmetric QCD with $N_{f}$ flavors. It is known that taking the vacuum expectation value of the matter field to be infinite, we can show that gaugino condensation can occur. At such a limit we should consider only pure supersymmetric Yang-Mills theory. But when we include an interaction term of order $O(\frac{1}{v})$, the situation can change. We analyze the effect of this interaction term and examine the gaugino condensation in the low energy Yang-Mills theory by using the scheme of Nambu-Jona-Lasinio.Comment: 5 pages, Plain Late

A natural solution to the μ\mu-problem in dynamical supergravity model

The Higgs mixing term coefficient $\mu$ is calculated in the supersymmetric theory which possesses a non-anomalous $U(1)_{R}$ symmetry in the limit of global supersymmetry. In this model, supersymmetry is assumed to be broken by gaugino condensation in the hidden sector when the supergravity effects are turned on. The soft breaking terms in the visible sector and the $\mu$ term of order the weak scale are produced in a simple manner.Comment: 6 pages, Latex, accepted for publication in Phys.Rev.

Partially disordered state and spin-lattice coupling in an S=3/2 triangular lattice antiferromagnet Ag2CrO2

Ag2CrO2 is an S=3/2 frustrated triangular lattice antiferromagnet without orbital degree of freedom. With decreasing temperature, a 4-sublatice spin state develops. However, a long-range partially disordered state with 5 sublattices abruptly appears at TN=24 K, accompanied by a structural distortion, and persists at least down to 2 K. The spin-lattice coupling stabilizes the anomalous state, which is expected to appear only in limited ranges of further-neighbor interactions and temperature. It was found that the spin-lattice coupling is a common feature in triangular lattice antiferromagnets with multiple-sublattice spin states, since the triangular lattice is elastic.Comment: 5 pages, 5 figure

Collapse to Black Holes in Brans-Dicke Theory: I. Horizon Boundary Conditions for Dynamical Spacetimes

We present a new numerical code that evolves a spherically symmetric configuration of collisionless matter in the Brans-Dicke theory of gravitation. In this theory the spacetime is dynamical even in spherical symmetry, where it can contain gravitational radiation. Our code is capable of accurately tracking collapse to a black hole in a dynamical spacetime arbitrarily far into the future, without encountering either coordinate pathologies or spacetime singularities. This is accomplished by truncating the spacetime at a spherical surface inside the apparent horizon, and subsequently solving the evolution and constraint equations only in the exterior region. We use our code to address a number of long-standing theoretical questions about collapse to black holes in Brans-Dicke theory.Comment: 46 pages including figures, uuencoded gz-compressed postscript, Submitted to Phys Rev

Collapse to Black Holes in Brans-Dicke Theory: II. Comparison with General Relativity

We discuss a number of long-standing theoretical questions about collapse to black holes in the Brans-Dicke theory of gravitation. Using a new numerical code, we show that Oppenheimer-Snyder collapse in this theory produces black holes that are identical to those of general relativity in final equilibrium, but are quite different from those of general relativity during dynamical evolution. We find that there are epochs during which the apparent horizon of such a black hole passes {\it outside\/} the event horizon, and that the surface area of the event horizon {\it decreases\/} with time. This behavior is possible because theorems which prove otherwise assume $R_{ab}l^al^b \ge 0$ for all null vectors $l^a$. We show that dynamical spacetimes in Brans-Dicke theory can violate this inequality, even in vacuum, for any value of $\omega$.Comment: 24 pages including figures, uuencoded gz-compressed postscript, Submitted to Phys Rev

Femtosecond nonlinear ultrasonics in gold probed with ultrashort surface plasmons

Fundamental interactions induced by lattice vibrations on ultrafast time scales become increasingly important for modern nanoscience and technology. Experimental access to the physical properties of acoustic phonons in the THz frequency range and over the entire Brillouin zone is crucial for understanding electric and thermal transport in solids and their compounds. Here, we report on the generation and nonlinear propagation of giant (1 percent) acoustic strain pulses in hybrid gold/cobalt bilayer structures probed with ultrafast surface plasmon interferometry. This new technique allows for unambiguous characterization of arbitrary ultrafast acoustic transients. The giant acoustic pulses experience substantial nonlinear reshaping already after a propagation distance of 100 nm in a crystalline gold layer. Excellent agreement with the Korteveg-de Vries model points to future quantitative nonlinear femtosecond THz-ultrasonics at the nano-scale in metals at room temperature

Slow relaxations and history dependence of the transport properties of layered superconductors

We study numerically the time evolution of the transport properties of layered superconductors after different preparations. We show that, in accordance with recent experiments in BSCCO performed in the second peak region of the phase diagram (Portier et al, 2001), the relaxation strongly depends on the initial conditions and is extremely slow. We investigate the dependence on the pinning center density and the perturbation applied. We compare the measurements to recent findings in tapped granular matter and we interpret our results with a rather simple picture.Comment: 4 pages, 4 fig

Exercise, cognition and Alzheimer’s disease: More is not necessarily better

Regional hypoperfusion, associated with a reduction in cerebral metabolism, is a hallmark of Alzheimer’s disease (AD) and contributes to cognitive decline. Cerebral perfusion and hence cognition can be enhanced by exercise. The present review describes first how the effects of exercise on cerebral perfusion in AD are mediated by nitric oxide (NO) and tissue-type plasminogen activator, the release of which is regulated by NO. A conclusion of clinical relevance is that exercise may not be beneficial for the cognitive functioning of all people with dementia if cardiovascular risk factors are present. The extent to which cardiovascular risk factors play a role in the selection of older people with dementia in clinical studies will be addressed in the second part of the review in which the effects of exercise on cognition are presented. Only eight relevant studies were found in the literature, emphasizing the paucity of studies in this field. Positive effects of exercise on cognition were reported in seven studies, including two that excluded and two that included patients with cardiovascular risk factors. These findings suggest that cardiovascular risk factors do not necessarily undo the beneficial effects of exercise on cognition in cognitively impaired people. Further research is called for, in view of the limitations of the clinical studies reviewed here.

Inhibition of Y1 receptor signaling improves islet transplant outcome

Failure to secrete sufficient quantities of insulin is a pathological feature of type-1 and type-2 diabetes, and also reduces the success of islet cell transplantation. Here we demonstrate that Y1 receptor signaling inhibits insulin release in β-cells, and show that this can be pharmacologically exploited to boost insulin secretion. Transplanting islets with Y1 receptor deficiency accelerates the normalization of hyperglycemia in chemically induced diabetic recipient mice, which can also be achieved by short-term pharmacological blockade of Y1 receptors in transplanted mouse and human islets. Furthermore, treatment of non-obese diabetic mice with a Y1 receptor antagonist delays the onset of diabetes. Mechanistically, Y1 receptor signaling inhibits the production of cAMP in islets, which via CREB mediated pathways results in the down-regulation of several key enzymes in glycolysis and ATP production. Thus, manipulating Y1 receptor signaling in β-cells offers a unique therapeutic opportunity for correcting insulin deficiency as it occurs in the pathological state of type-1 diabetes as well as during islet transplantation.Islet transplantation is considered one of the potential treatments for T1DM but limited islet survival and their impaired function pose limitations to this approach. Here Loh et al. show that the Y1 receptor is expressed in β- cells and inhibition of its signalling, both genetic and pharmacological, improves mouse and human islet function.info:eu-repo/semantics/publishe