Linear polarization of the photoluminescence of quantum wells

The degree and orientation of the magnetic-field induced linear polarization of the photoluminescence from a wide range of heterostructures containing (Cd,Mn)Te quantum wells between (Cd,Mn,Mg)Te barriers has been studied as a function of detection photon energy, applied magnetic field strength and orientation in the quantum well plane. A theoretical description of this effect in terms of an in-plane deformation acting on the valence band states is presented and is verified by comparison with the experimental data. We attempted to identify clues to the microscopic origin of the valence band spin anisotropy and to the mechanisms which actually determine the linear polarization of the PL in the quantum wells subject to the in-plane magnetic field. The conclusions of the present paper apply in full measure to non-magnetic QWs as well as ensembles of disk-like QDs with shape and/or strain anisotropy.Comment: 21 pages, 10 figure

Open Access, a breakthrough for science that every neuroscientist should know about

Open Access is the online access to scientific journal literature, free of charge and free of most copyright and licensing restrictions. Open Access was named one of the top science news stories of 2003 by Nature, Science, The Scientist, and The Wall Street Journal. Open Access is supported by major science funding bodies, such as Howard Hughes Medical Institute (USA), the Wellcome trust (UK), the Max Planck Society, the DFG (Germany), the CNRS and INSERM (France). Rapidly rising conventional journal prices have been denounced by leading research universities (Harvard, Stanford, Cornell, Duke, more), found dysfunctional/unsustainable by independent financial analysts (PNB Paribas, Citigroup, Credit Suisse), and are now under investigation by Science and Technology Committee of the United Kingdom House of Commons. The advantages of an Open Access journal are compelling: articles are accessible to everyone with an internet connection; authors reach larger audiences and have greater impact; no permission is needed for copying, printing, distributing, storing or other educational uses; copyright remains with authors. Open Access does not violate copyright, but uses the consent of the copyright holder. Open Access journals are peer-reviewed and use the same high standards (ex. WAME) as conventional journals. Open Access journals are indexed in major databases (ex. National Institutes of Health, National Library of Medicine PubMed; ISI Science Citation Index) and major search engines (Google, Yahoo). Open Access journals may deposit copies of their articles in central archives (ex. PubMed Central), enhancing their preservation/availability. There are different funding models for Open Access journals in different circumstances; their costs are usually paid by the same sources that fund research. Of a thousand Open Access Journals listed in the Directory of Open Access Journals (DOAJ) many have no publication charge. Some journals are entirely Open Access (ex. PLoS Biology), and some conventional journals are experimenting with Open Access (ex. PNAS). Scientists who publish in conventional journals can provide Open Access to the same articles by depositing them in Open Access archives. Open Access accelerates research, shares knowledge, improves the usefulness of science journals articles. Wider adoption of Open Access depends on educating the scientific community about its benefits

The 1/N Expansion in Noncommutative Quantum Mechanics

We study the 1/N expansion in noncommutative quantum mechanics for the anharmonic and Coulombian potentials. The expansion for the anharmonic oscillator presented good convergence properties, but for the Coulombian potential, we found a divergent large N expansion when using the usual noncommutative generalization of the potential. We proposed a modified version of the noncommutative Coulombian potential which provides a well-behaved 1/N expansion.Comment: v2: resided version, to appear in PRD, 18 pages, 4 figure

Suris tetrons: possible spectroscopic evidence for four-particle optical excitations of the 2D electron gas

The excitations of a two-dimensional electron gas in quantum wells with intermediate carrier density (~10^{11} cm^{-2}), i.e., between the exciton-trion- and the Fermi-Sea range, are so far poorly understood. We report on an approach to bridge this gap by a magneto-photoluminescence study of modulation-doped (Cd,Mn)Te quantum well structures. Employing their enhanced spin splitting, we analyzed the characteristic magnetic-field behavior of the individual photoluminescence features. Based on these results and earlier findings by other authors, we present a new approach for understanding the optical transitions at intermediate densities in terms of four-particle excitations, the Suris tetrons, which were up to now only predicted theoretically. All characteristic photoluminescence features are attributed to emission from these quasi-particles when attaining different final states.Comment: 12 pages, 3 figure

Exciton spin decay modified by strong electron-hole exchange interaction

We study exciton spin decay in the regime of strong electron-hole exchange interaction. In this regime the electron spin precession is restricted within a sector formed by the external magnetic field and the effective exchange fields triggered by random spin flips of the hole. Using Hanle effect measurements, we demonstrate that this mechanism dominates our experiments in CdTe/(Cd,Mg)Te quantum wells. The calculations provide a consistent description of the experimental results, which is supported by independent measurements of the parameters entering the model.Comment: 5 pages, 3 figure

Survival of adult neurons lacking cholesterol synthesis in vivo

BACKGROUND: Cholesterol, an essential component of all mammalian plasma membranes, is highly enriched in the brain. Both during development and in the adult, brain cholesterol is derived from local cholesterol synthesis and not taken up from the circulation. However, the contribution of neurons and glial cells to total brain cholesterol metabolism is unknown. RESULTS: Using conditional gene inactivation in the mouse, we disrupted the squalene synthase gene (fdft1), which is critical for cholesterol synthesis, in cerebellar granule cells and some precerebellar nuclei. Mutant mice showed no histological signs of neuronal degeneration, displayed ultrastructurally normal synapses, and exhibited normal motor coordination. This revealed that these adult neurons do not require cell-autonomous cholesterol synthesis for survival or function. CONCLUSION: We conclude that at least some adult neurons no longer require endogenous cholesterol synthesis and can fully meet their cholesterol needs by uptake from their surrounding. Glia are a likely source of cholesterol in the central nervous system

Guanosine effect on cholesterol efflux and apolipoprotein E expression in astrocytes

The main source of cholesterol in the central nervous system (CNS) is represented by glial cells, mainly astrocytes, which also synthesise and secrete apolipoproteins, in particular apolipoprotein E (ApoE), the major apolipoprotein in the brain, thus generating cholesterol-rich high density lipoproteins (HDLs). This cholesterol trafficking, even though still poorly known, is considered to play a key role in different aspects of neuronal plasticity and in the stabilisation of synaptic transmission. Moreover, cell cholesterol depletion has recently been linked to a reduction in amyloid beta formation. Here we demonstrate that guanosine, which we previously reported to exert several neuroprotective effects, was able to increase cholesterol efflux from astrocytes and C6 rat glioma cells in the absence of exogenously added acceptors. In this effect the phosphoinositide 3 kinase/extracellular signal-regulated kinase 1/2 (PI3K/ERK1/2) pathway seems to play a pivotal role. Guanosine was also able to increase the expression of ApoE in astrocytes, whereas it did not modify the levels of ATP-binding cassette protein A1 (ABCA1), considered the main cholesterol transporter in the CNS. Given the emerging role of cholesterol balance in neuronal repair, these effects provide evidence for a role of guanosine as a potential pharmacological tool in the modulation of cholesterol homeostasis in the brain