Form Factors for Exclusive Semileptonic BB--Decays

We developed the new parton model approach for exclusive semileptonic decays of $B$-meson to $D,~D^*$ by extending the inclusive parton model, and by combining with the results of the HQET, motivated by Drell-Yan process. Without the nearest pole dominance ans\"atze, we {\bf derived} the dependences of hadronic form factors on $q^2$. We also calculated numerically the slope of the Isgur-Wise function, which is consistent with the experimental results.Comment: 20 pages, RevTex, 2 ps figure files(uuencoded in seperate file

Implication of the weak phase β\beta measured in B→ργB \to \rho \gamma decay

We explore the exclusive $B^0 \to \rho^0 \gamma$ decay to obtain the time-dependent CP asymmetry in $b \to d \gamma$ decay process. We find that the complex RL and RR mass insertion to the squark sector in the MSSM can lead to a large deviation of CP asymmetry from that predicted in the Standard Model.Comment: 4 pages, talk given at the 32nd International Conference on High Energy Physics (ICHEP'04), Beijing, China, 16-22 Aug. 200

Time dependent CP asymmetry in B0→ρ0γB^0 \to \rho^0 \gamma decay to probe the origin of CP violation

Since the CP violation in the $B$ system has been investigated up to now only through processes related to the $B$--$\bar{B}$ mixing, urgently required is new way of study for the CP violation and establishing its origin in the $B$ system independent of the mixing process. In this work, we explore the exclusive $B^0 \to \rho^0 \gamma$ decay to obtain the time-dependent CP asymmetry in $b \to d$ decay process in the standard model and the supersymmetric model. We find that the complex RL and RR mass insertion to the squark sector in the MSSM can lead to a large CP asymmetry in $b \to d \gamma$ decay through the gluino-squark diagrams, which is not predicted in the Standard Model induced by the $B$--$\bar{B}$ mixing.Comment: 10 pages, 4 eps figure

Soft Wilson lines in soft-collinear effective theory

The effects of the soft gluon emission in hard scattering processes at the phase boundary are resummed in the soft-collinear effective theory (SCET). In SCET, the soft gluon emission is decoupled from the energetic collinear part, and is obtained by the vacuum expectation value of the soft Wilson-line operator. The form of the soft Wilson lines is universal in deep inelastic scattering, in the Drell-Yan process, in the jet production from e+e- collisions, and in the gamma* gamma* -> pi0 process, but its analytic structure is slightly different in each process. The anomalous dimensions of the soft Wilson-line operators for these processes are computed along the light-like path at leading order in SCET and to first order in alpha_s, and the renormalization group behavior of the soft Wilson lines is discussed.Comment: 36 pages, 10 figures, 3 table

Autophagy, Cellular Aging and Age-related Human Diseases.

Macroautophagy/autophagy is a conserved degradation system that engulfs intracytoplasmic contents, including aggregated proteins and organelles, which is crucial for cellular homeostasis. During aging, cellular factors suggested as the cause of aging have been reported to be associated with progressively compromised autophagy. Dysfunctional autophagy may contribute to age-related diseases, such as neurodegenerative disease, cancer, and metabolic syndrome, in the elderly. Therefore, restoration of impaired autophagy to normal may help to prevent age-related disease and extend lifespan and longevity. Therefore, this review aims to provide an overview of the mechanisms of autophagy underlying cellular aging and the consequent disease. Understanding the mechanisms of autophagy may provide potential information to aid therapeutic interventions in age-related diseases

New Physics Effects in B→K(∗)ννB \to K^(*) \nu \nu Decays

We present a model-independent analysis of rare B decays, $B \to K^{(*)} \nu \nu$. The effect of possible new physics is written in terms of dimension-6 four-fermi interactions. The lepton number violating scalar- and tensor-type interactions are included, and they induce $B \to K^{(*)} \nu \nu ({\bar \nu} {\bar \nu})$ decays. We show systematically how the branching ratios and missing mass-squared spectrum depend on the coefficients of the four-fermi interactions.Comment: 20 pages with 7 figure

b→sγb \to s \gamma decays in the Left-Right Symmetric Model

We consider $b \to s \gamma$ decays in the Left-Right Symmetric Model. Values of observables sensitive to chiral structure such as the $\Lambda$ polarization in the $\Lambda_b \to \Lambda \gamma$ decays and the mixing-induced CP asymmetries in the $B_{d,s} \to M^0 \gamma$ decays can deviate in the LRSM significantly from the SM values. The combined analysis of $P_\Lambda$ and $A_{CP}$ as well as ${\cal BR}(b \to s \gamma)$ can be used to determine the model parameters.Comment: 16 pages with 7 figures, Version to be published in PR

Singlet Fermionic Dark Matter explains DAMA signal

It has been suggested that, considering channeling effect, the order of a few GeV dark matters which are elastically scattered from detector nuclei might be plausible candidates reconciling the DAMA annual modulation signal with the results of other null experiments. We show that Singlet Fermionic Dark Matter can be such a dark matter candidate, simultaneously providing the correct thermal relic density which is consistent with the WMAP data.Comment: 9 pages, 3 figure

Exome-based Variant Detection in Core Promoters.

Core promoter controls the initiation of transcription. Core promoter sequence change can disrupt transcriptional regulation, lead to impairment of gene expression and ultimately diseases. Therefore, comprehensive characterization of core promoters is essential to understand normal and abnormal gene expression in biomedical studies. Here we report the development of EVDC (Exome-based Variant Detection in Core promoters) method for genome-scale analysis of core-promoter sequence variation. This method is based on the fact that exome sequences contain the sequences not only from coding exons but also from non-coding region including core promoters generated by random fragmentation in exome sequencing process. Using exome data from three cell types of CD4+ T cells, CD19+ B cells and neutrophils of a single individual, we characterized the features of core promoter-mapped exome sequences, and analysed core-promoter variation in this individual genome. We also compared the core promoters between YRI (Yoruba in Ibadan, Nigeria) and the CEU (Utah residents of European decedent) populations using the exome data generated by the 1000 Genome project, and observed much higher variation in YRI population than in CEU population. Our study demonstrates that the EVDC method provides a simple but powerful means for genome-wile de novo characterization of core promoter sequence variation