The Next 1616 Higher Spin Currents and Three-Point Functions in the Large N=4{\cal N}=4 Holography

By using the known operator product expansions (OPEs) between the lowest $16$ higher spin currents of spins $(1, \frac{3}{2}, \frac{3}{2}, \frac{3}{2}, \frac{3}{2}, 2,2,2,2,2,2, \frac{5}{2}, \frac{5}{2}, \frac{5}{2}, \frac{5}{2}, 3)$ in an extension of the large ${\cal N}=4$ linear superconformal algebra, one determines the OPEs between the lowest $16$ higher spin currents in an extension of the large ${\cal N}=4$ nonlinear superconformal algebra for generic $N$ and $k$. The Wolf space coset contains the group $G =SU(N+2)$ and the affine Kac-Moody spin $1$ current has the level $k$. The next $16$ higher spin currents of spins $(2,\frac{5}{2}, \frac{5}{2}, \frac{5}{2}, \frac{5}{2}, 3,3,3,3,3,3, \frac{7}{2}, \frac{7}{2}, \frac{7}{2}, \frac{7}{2},4)$ arise in the above OPEs. The most general lowest higher spin $2$ current in this multiplet can be determined in terms of affine Kac-Moody spin $\frac{1}{2}, 1$ currents. By careful analysis of the zero mode (higher spin) eigenvalue equations, the three-point functions of bosonic higher spin $2, 3, 4$ currents with two scalars are obtained for finite $N$ and $k$. Furthermore, we also analyze the three-point functions of bosonic higher spin $2, 3, 4$ currents in the extension of the large ${\cal N}=4$ linear superconformal algebra. It turns out that the three-point functions of higher spin $2,3$ currents in the two cases are equal to each other at finite $N$ and $k$. Under the large $(N,k)$ 't Hooft limit, the two descriptions for the three-point functions of higher spin $4$ current coincide with each other. The higher spin extension of $SO(4)$ Knizhnik Bershadsky algebra is described.Comment: 94 pages; The 43 and 44 pages added, the mathematica notebook files included and to appear epj

Organizing Pneumonia by Paragonimiasis and Coexistent Aspergilloma Manifested as a Pulmonary Irregular Nodule

Organizing pneumonia by paragonimiasis and coexistent aspergilloma as a pulmonary nodule is a rare case of lung disease. Its radiographic or CT feature has not been described before in the radiologic literature. We present organizing pneumonia by paragonimiasis and coexistent aspergilloma manifested as a pulmonary irregular nodule on CT

GaAs droplet quantum dots with nanometer-thin capping layer for plasmonic applications

We report on the growth and optical characterisation of droplet GaAs quantum dots with extremely-thin (11 nm) capping layers. To achieve such result, an internal thermal heating step is introduced during the growth and its role in the morphological properties of the quantum dots obtained is investigated via scanning electron and atomic force microscopy. Photoluminescence measurements at cryogenic temperatures show optically stable, sharp and bright emission from single quantum dots, at near-infrared wavelengths. Given the quality of their optical properties and the proximity to the surface, such emitters are ideal candidates for the investigation of near field effects, like the coupling to plasmonic modes, in order to strongly control the directionality of the emission and/or the spontaneous emission rate, crucial parameters for quantum photonic applications.Comment: 1 pages, 3 figure

Korean Red Ginseng inhibits apoptosis in neuroblastoma cells via estrogen receptor β-mediated phosphatidylinositol-3 kinase/Akt signaling

AbstractBackgroundGinseng has been shown to exert antistress effects both in vitro and in vivo. However, the effects of ginseng on stress in brain cells are not well understood. This study investigated how Korean Red Ginseng (KRG) controls hydrogen peroxide-induced apoptosis via regulation of phosphatidylinositol-3 kinase (PI3K)/Akt and estrogen receptor (ER)-β signaling.MethodsHuman neuroblastoma SK-N-SH cells were pretreated with KRG and subsequently exposed to H2O2. The ability of KRG to inhibit oxidative stress-induced apoptosis was assessed in MTT cytotoxicity assays. Apoptotic protein expression was examined by Western blot analysis. The roles of ER-β, PI3K, and p-Akt signaling in KRG regulation of apoptosis were studied using small interfering RNAs and/or target antagonists.ResultsPretreating SK-N-SH cells with KRG decreased expression of the proapoptotic proteins p-p53 and caspase-3, but increased expression of the antiapoptotic protein BCL2. KRG pretreatment was also associated with increased ER-β, PI3K, and p-Akt expression. Conversely, ER-β inhibition with small interfering RNA or inhibitor treatment increased p-p53 and caspase-3 levels, but decreased BCL2, PI3K, and p-Akt expression. Moreover, inhibition of PI3K/Akt signaling diminished p-p53 and caspase-3 levels, but increased BCL2 expression.ConclusionCollectively, the data indicate that KRG represses oxidative stress-induced apoptosis by enhancing PI3K/Akt signaling via upregulation of ER-β expression