Penalty discount rate: I

Monetary policy - United States ; Discount

Penalty discount rate: II

Monetary policy - United States ; Discount ; Bank reserves

Practice guidelines for prenatal and perinatal immunohematology, revisited

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/73778/1/j.1537-2995.2001.41111445.x.pd

On proper terminology

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/72876/1/j.1537-2995.1992.32192116443.x.pd

On the high probability that a perceived lack of value of obtaining a p value will be detrimental to patient care

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/92406/1/j.1537-2995.1997.37897424414.x.pd

Appropriate Serological Testing in Pregnancy

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/72557/1/j.1423-0410.1992.tb01243.x.pd

Persistence of cefotetan on red blood cells

Cefotetan can cause severe immune hemolytic anemia that may persist long after the drug is discontinued. To study the binding of cefotetan to RBCs, patients who received cefotetan were followed and tested for the presence of antibody to cefotetan. STUDY DESIGN AND METHODS:  Patients receiving cefotetan were identified from pharmacy and nursing records. Blood samples obtained for routine hematology tests were analyzed. Cefotetan binding to patients’ RBCs was tested using a previously characterized high-titer anticefotetan serum by gel technique. To determine the minimum amount of drug necessary for binding to occur, RBCs were incubated with serial dilutions of cefotetan at pH 7.4. RESULTS:  Sixty patients receiving 1 to 25 g IV (median, 2 g) of cefotetan were followed for 1 to 123 days (median, 18 days). All were initially positive, for cefotetan on RBCs. Positivity persisted for up to 98 days after the last dose of drug. Fifteen patients became negative during follow-up. The first negative sample occurred at Day 30 to 123. Using the midpoint between the last positive and first negative to estimate of the duration of positivity, we estimate that cefotetan remains RBC-bound for 16.5 to 92 days (median, 67.5 days). During the follow-up period, five patients developed anticefotetan detectable in the serum. Twenty patients receiving other cephalosporin antibiotics showed no specific reactivity of their RBCs with anticefotetan. In vitro studies showed a minimum necessary drug concentration of 1 µmol/L at physiologic pH, which was not significantly altered by RBC pretreatment with ficin, sialydase, or DTT. CONCLUSIONS:  Cefotetan is tightly bound to RBCs after intravenous administration and remains detectable for weeks after the last dose. Antibodies to cefotetan may occur in about 8 percent of patients receiving the drug. The minimum necessary concentration for RBC binding is low compared to an estimated plasma concentration of 240 µmol/L from a single IV dose of 1 g.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/72360/1/j.1537-2995.2004.03360.x.pd

St. John\u27s Church: A History and Appreciation: Produced on the Occasion of the Jubilee 2000

A history of St. John\u27s Catholic Church on York Street in Bangor, Maine, dating from the 1830s to 2000.https://digicom.bpl.lib.me.us/books_pubs/1343/thumbnail.jp

Inactivation of Kell blood group antigens by 2-aminoethylisothiouronium bromide

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/75007/1/j.1365-2141.1982.tb07295.x.pd

Anti-Lu14: A Lutheran Antibody Defining the Product of an Allele at the Lu8 Blood Group Locus 1

A ‘new’ Lutheran-related antibody, named anti-Lu14, reacts with approximately 2.4% of random bloods. Red cells of the rare Lu:-8 phenotype are Lu:14. The data indicate, with a high probability, that the Lu 14 antigen is a product of an allele of Lu 8 and that Lu 14 and Lu 8 comprise a third pair of alleles at the Lutheran locus. Red cells of the original Sw (a+) propositus are Lu:14. By coincidence, he has inherited two low-incidence genes. This observation may explain the discrepancy in different families concerning a possible relationship between Sw a and Lutheran. Pedigree information now suggests that Sw a is not a Lutheran gene.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/75055/1/j.1423-0410.1977.tb00632.x.pd