Revolution Unfinished: Comparing Collective Memory in the Kyiv Post and RT

From November 2013 – February 2014, Ukraine’s Independence Square (or Maidan) became the site of revolution. The Maidan Revolution culminated in the deaths of over 100 protesters and law enforcement, and the removal of former President Viktor Yanukovych. Subsequently, several studies have observed how Maidan is being remembered (see Kozachenko, 2020; Nuzov, 2016; Shevel, 2016). I rely on the perspectives of Maurice Halbwachs (2011/1925) on collective memory, Robin Wagner-Pacifici (1996; 2010; 2017) on events, and various perspectives on media framing in journalism. This paper builds upon existing literature by exploring the formation of collective memory in 52 newspaper articles from the Kyiv Post and RT. From my findings I argue both news outlets accept Maidan as part of their taken for granted memory. Both outlets primarily frame Maidan using national memory narratives. Like Kozachenko (2020), I observed the presence of Ukrainophile and Sovietophile historical frames. Though both news outlets frame Maidan as a failed revolution, I argue Maidan is characterized differently by the Kyiv Post and RT. Whereas, the Kyiv Post frames Maidan as a tragic unfinished revolution, RT constructs a framing of Maidan as a coup which allows them to compare it to current events. Presented in absentia on April 27, 2020 at "Student Research Day" at MacEwan University in Edmonton, Alberta. (Conference cancelled) Faculty Mentor: Jeff Stepnisky  Department: Sociology&nbsp

Is a veterinary student's performance on multiple-mini interviews affected by personality preferences?

ObjectivesThe objectives of this study were to evaluate the association between a student's Myers-Briggs Type Indicator (MBTI) preference pairs and resulting types and his or her multiple-mini interview (MMI) scores upon admission, and to determine the proportions of types among veterinary classes over five years.MethodsA cross-sectional study was conducted for data collected from 706 students admitted into the University of California Davis School of Veterinary Medicine (UCDSVM) program beginning in the fall of 2013 and ending in the fall of 2018. Data consisted of a candidate's MBTI preference pairs and types which were collected during the first week of enrollment and multiple-mini interview scores from his or her admission data.ResultsA total of 706 students from 5 classes completed the MBTI survey. Multivariate analysis showed no significant association between the MBTI preference pairs of extroversion and introversion (F(1, 697) = 3.30, p=0.0959), sensing and intuition (F(1, 697) = 0.40, p=0.4395), thinking and feeling (F(1, 697) = 3.59, p=0.0591), or judging and perceiving (F(1, 697) = 0.38, p = 0.5657) and MMI score. Analysis showed no trends (χ2 (60, N=706) =76.51, p=0.074) in the student's MBTI types over the 5-year period.ConclusionsThe MMI score of a candidate admitted into the UCDSVM is unlikely to be affected by personality preferences. Therefore, it is unlikely that multiple-mini interview scores included in the admission process will affect the personality diversity of candidates admitted into a veterinary class

Lesão na camada glomerular do bulbo olfatório interfere na olfação e nos comportamentos tipo-depressivos em modelo animal de parkinsonismo

Orientador: Prof. Dr. Marcelo de Meira Santos LimaDissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Fisiologia. Defesa : Curitiba, 26/05/2017Inclui referênciasResumo: Os distúrbios olfatórios e a depressão são sintomas não-motores comumente relatados por indivíduos com a Doença de Parkinson (DP), sendo manifestados anos antes dos sintomas motores. Os mecanismos subjacentes a essas manifestações clínicas não estão completamente elucidados, no entanto, o desequilíbrio na neurotransmissão dopaminérgica parece ter um papel importante nesse contexto. Em indivíduos e em modelos animais da DP, é observado um aumento dos interneurônios dopaminérgicos da camada glomerular de bulbo olfatório (BO-gl), o que poderia contribuir para o prejuízo olfatório. Além disso, o desequilíbrio neuronal no BO está relacionado a sintomas depressivos, como demonstrado pela bulbectomia olfatória química. Dessa forma, nós hipotetizamos que a redução no número e/ou densidade dos neurônios dopaminérgicos presentes no BO poderia promover uma melhora olfatória e, em contrapartida, acentuaria o comportamento tipo-depressivo no modelo de Parkinsonismo induzido por 6-hidroxidopamina (6- OHDA). Assim, foi feita a administração intranigral de 6-OHDA e, posteriormente, a injeção da mesma droga na BO-gl em ratos, sendo esses avaliados em dois diferentes tempos (7 e 14 dias após a infusão intranigral de 6-OHDA). Observamos que, após 7 dias, o grupo lesionado apenas na SNpc apresentou comprometimento olfatório, assim como o grupo com a lesão apenas no BO. No entanto, a combinação das lesões na SNpc e BO foi capaz de reverter esse padrão, fazendo que os animais discriminassem ambos os compartimentos. Em relação ao comportamento tipodepressivo, observamos que a lesão na SNpc promoveu comportamento tipodepressivo, sendo esse acentuado após uma lesão dupla. A influência da SNpc e do BO no comportamento tipo-depressivo é corroborada por correlações entre parâmetros depressivos e neurônios TH-ir da SNpc em ambos tempos avaliados e com neurônios TH-ir do BO na avaliação de 14 dias. Esses resultados indicam um papel crucial do sistema dopaminérgico no BO e na SNpc tanto na modulação da olfação quanto no comportamento tipo-depressivo em um modelo de Parkinsonismo induzido por 6-OHDA. Palavras-chave: Doença de Parkinson, depressão, distúrbios olfatórios, sistema dopaminérgico.Abstract: Olfactory impairments and depressive behavior are commonly reported by individuals with Parkinson's disease (PD) being manifested before motor symptoms. The mechanisms underlying these clinical manifestations are not fully elucidated, however, the imbalance in dopaminergic neurotransmission seems to play an important role in the context. In individuals and animal models of PD, an increase in the dopaminergic interneurons of the glomerular layer in olfactory bulb (OB-gl) is observed, which could contribute to the olfactory impairment. In addition, neuronal imbalance in OB is related to depressive symptoms, as demonstrated by chemical olfactory bulbectomy. In view of that, we hypothesized that the reduction in the number and / or density of dopaminergic neurons present in OB could promote an olfactory improvement and, in contrast, would accentuate the depressive-like behavior in the 6-hydroxydopamine (6-OHDA) induced Parkinsonism model. Thereby, intranigral administration of 6-OHDA and subsequent injection of the same drug into OB-gl in rats were performed, being evaluated at two different time-points (7 and 14 days after intranigral infusion of 6-OHDA). We observed that, after 7 days, the group only injured in the SNpc presented olfactory impairment, as the group with the lesion only in the BO. However, the combination of the lesions in SNpc and BO was able to reverse this pattern, causing the animals to discriminate both compartments. In relation to depressive-like behaviors, we observed that the SNpc injury promoted depressive-like behavior, being accentuated after a double injury. The SNpc and OB influence on depressive-like behavior is corroborated by correlations between depressive parameters and SNpc TH-ir neurons at both evaluated times and with OB-gl TH-ir neurons in later time-point. These results indicate a crucial role of dopaminergic system in OB and SNpc in olfaction and humor modulation in a model of Parkinsonism induced by 6-OHDA. Keywords: Parkinson's disease, depression, olfactory disorders, dopaminergic system

"School Strike 4 Climate": Social Media and the International Youth Protest on Climate Change

Beginning in 2018, youth across the globe participated in protest activities aimed at encouraging government action on climate change. This activism was initiated and led by Swedish teenager, Greta Thunberg. Like other contemporary movements, the School Strike 4 Climate used social media. For this article, we use Twitter trace data to examine the global dynamics of the student strike on March 15, 2019. We offer a nuanced analysis of 993 tweets, employing a combination of qualitative and quantitative analysis. Like other movements, the primary function of these tweets was to share information, but we highlight a unique type of information shared in these tweets - documentation of local events across the globe. We also examine opinions shared about youth, the tactic (protest/strike), and climate change, as well as the assignment of blame on government and other institutions for their inaction and compliance in the climate crisis. This global climate strike reflects a trend in international protest events, which are connected through social media and other digital media tools. More broadly, it allows us to rethink how social media platforms are transforming political engagement by offering actors - especially the younger generation - agency through the ability to voice their concerns to a global audience

Correction to: Assessment of burnout in veterinary medical students using the Maslach burnout inventory-educational survey: a survey during two semesters.

Following publication of the original article [1], we've been notified by an author that they have published their manuscript without seeking permission for the survey that was included in one of their tables (Table 1)

Influência da neuroinflamação sobre a olfação em modelo animal de Parkinson

Orientador: Prof. Dr. Marcelo de Meira Santos LimaCoorientadora: Dra. Lais Soares RodriguesTese (doutorado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Fisiologia. Defesa : Curitiba, 30/05/2023Inclui referênciasResumo: A doença de Parkinson (DP) é a segunda doença neurodegenerativa mais prevalente na população mundial, sendo caracterizada pela degeneração dos neurônios dopaminérgicos da substância negra pars compacta (SNpc). Sabe-se que a neuroinflamação está diretamente relacionada ao surgimento e progressão da DP, sendo associada à diminuição da neurogênese adulta em seus locais de ocorrência, zona subgranular do hipocampo e zona subventricular (ZSV), uma vez que a administração de anti-inflamatórios foi capaz de promover melhora do quadro. Durante os estágios iniciais da DP estão presentes distúrbios não-motores como alterações olfatórias, de sono (principalmente de sono REM) e sinais e sintomas depressivos como apatia e anedonia. Detectamos, em estudo prévio, que a diminuição dos neurônios da camada glomerular do bulbo olfatório (BO) por lesão local de 6-OHDA foi capaz de reverter o prejuízo olfatório em modelo animal de parkinsonismo ao apresentar redução dos neurônios periglomerulares. Frente a este dado, o objetivo do presente estudo foi investigar a neuroinflamação e seus possíveis efeitos sobre olfação em modelo de lesão dupla (intranigral e intrabulbar) com a neurotoxina 6-hidroxidopamina (6-OHDA) através de testes comportamentais de reconhecimento olfatório e reconhecimento social, análise neuroinflamatória através da quantificação de proteína ácida fibrilar glial (GFAP) por Western Blot de BO e SNpc. O resultado do teste de reconhecimento olfatório não corroborou dados prévios do laboratório obtidos nos mesmos grupos. Desse modo, a exploração ativa do ambiente estimulada pela preferência ao odor demonstrou-se prejudicada em todos os casos de lesão aguda. Porém em 14 dias observou-se o comportamento de tendência limítrofe para a diferença de exploração no grupo com a dupla lesão. Em relação ao teste de reconhecimento social não houve diferenças significativas entre os grupos lesionados e o controle nas duas timelines, sugerindo que o prejuízo olfatório no modelo se deve a alterações de detecção do olfato e não a memória olfatória. Já para análise de GFAP no BO não observamos diferenças significativas entre os grupos ou linhas do tempo para alfa-GFAP enquanto para beta foi observado um aumento no grupo com a lesão somente na SNpc comparado ao com a dupla lesão. Na SNpc foi observado um aumento significativo no grupo somente com a lesão no bulbo em relação a todos os outros grupos, 7 dias após a cirurgia. Assim, observamos o interessante papel da neuroinflamação sobre o prejuízo olfatório de animais modelo de parkinsonismo.Abstract: Parkinson's disease (PD) is the second most prevalent neurodegenerative disease in the world population, being characterized by the degeneration of dopaminergic neurons of the substantia nigra pars compacta (SNpc). It is known that neuroinflammation is directly related to the onset and progression of PD, being associated with a decrease in adult neurogenesis in its sites of occurrence, subgranular zone of the hippocampus and subventricular zone (SVZ), since the administration of anti-inflammatory drugs was able to promote improvement of the picture. During the initial stages of PD, non-motor disturbances are present, such as olfactory and sleep disorders (mainly REM sleep) and depressive signs and symptoms such as apathy and anhedonia. In a previous study, we detected that the reduction of neurons in the glomerular layer of the olfactory bulb (BO) by local 6-OHDA injury was capable of reversing the olfactory impairment in an animal model of parkinsonism by presenting a reduction in periglomerular neurons. In view of this data, the aim of the present study was to investigate neuroinflammation and its possible effects on olfaction in a model of double lesion (intranigral and intrabulbar) with the neurotoxin 6-hydroxydopamine (6-OHDA) through behavioral tests of olfactory recognition and olfactory recognition. social, neuroinflammatory analysis through the quantification of glial fibrillary acidic protein (GFAP) by Western Blot of BO and SNpc. The result of the olfactory recognition test did not corroborate previous laboratory data obtained in the same groups. Thus, the active exploration of the environment stimulated by preference for odor was impaired in all cases of acute injury. Regarding the social recognition test, there were no significant differences between the injured and control groups in the two timelines, suggesting that the olfactory impairment in the model is due to changes in smell detection and not olfactory memory. As for the analysis of GFAP in BO, we did not observe significant differences between groups or timelines for alpha-GFAP, while for beta an increase was observed in the group with the lesion only in the SNpc compared to the group with the double lesion. In SNpc, a significant increase was observed in the group with only the bulb lesion in relation to all other groups, 7 days after surgery. Thus, we observed the interesting role of neuroinflammation on the olfactory impairment of parkinsonism model animals