Wavelength limits on isobaricity of perturbations in a thermally unstable radiatively cooling medium

Nonlinear evolution of one-dimensional planar perturbations in an optically thin radiatively cooling medium in the long-wavelength limit is studied numerically. The accepted cooling function generates in thermal equilibrium a bistable equation of state $P(\rho)$. The unperturbed state is taken close to the upper (low-density) unstable state with infinite compressibility ($dP/d\rho= 0$). The evolution is shown to proceed in three different stages. At first stage, pressure and density set in the equilibrium equation of state, and velocity profile steepens gradually as in case of pressure-free flows. At second stage, those regions of the flow where anomalous pressure (i.e. with negative compressibility) holds, create velocity profile more sharp than in pressure-free case, which in turn results in formation of a very narrow (short-wavelength) region where gas separates the equilibrium equation of state and pressure equilibrium sets in rapidly. On this stage, variation in pressure between narrow dense region and extended environment does not exceed more than 0.01 of the unperturbed value. On third stage, gas in the short-wavelength region reaches the second (high-density) stable state, and pressure balance establishes through the flow with pressure equal to the one in the unperturbed state. In external (long-wavelength) regions, gas forms slow isobaric inflow toward the short-wavelength layer. The duration of these stages decreases when the ratio of the acoustic time to the radiative cooling time increases. Limits in which nonlinear evolution of thermally unstable long-wavelength perturbations develops in isobaric regime are obtained.Comment: 21 pages with 7 figures, Revtex, accepted in Physics of Plasma

The effect of hypodynamia on mineral and protein metabolism in calcified tissues of the maxillodental system (experimental radioisotope study)

Mineral and protein metabolism was studied in experiments on 60 white rats, using P-32 and Ca-45 uptake in the mineral fractions, 2C-14-glycine in the protein fractions, and P-32 in both fractions of calcified tissues as indices over a 100 day period of experimental hypodynamia. Combined alterations in mineral and protein metabolism occurred in the calcified tissues of the experimental animals. The most pronounced changes were found in P-32 and 2C-14-glycine metabolism. In the incisors and femoral bones, these alterations occurred in two phases: P-32 and 2C-14-glycine uptake first increased, then decreased. Changes in Ca-45 metabolism were less pronounced, particularly in the initial period of the experiment. A marked reduction in P-32, Ca-45, and 2C-14-glycine uptake was found in various fractions of the calcified tissues on the 100th day of experimental hypodynamia