Analysis of the Impacts of Bearing on Vibration Characteristics of Rotor

Aiming at a Top Gas Recovery Turbine Unit (TRT) with double support rotor and the extending disk end, theoretical and experimental analysis about influence of cylindrical bearing and four-lobe bearing on vibration of TRT rotor system are conducted in this paper. The results indicate that vibration of the rotor supported by cylindrical bearing is more stable than that supported by four-lobe bearing at the driving end (DE) and the nondriving end (NDE). The amplitude of rotor is supported by both of these types of bearing increases as the speed increases at the NDE, while the amplitude of the DE remains unchanged. Comparing with the result of theoretical analysis, the practical test results are more consistent with the theoretical response analysis conducted by applying unbalanced mass at the extending disk end. This paper presents an analysis method of the critical characteristics of a double support rotor system with the extending disk end and provides reference value for dealing with vibration fault of double support rotor system with the extending disk end

Biomechanical Pulping of Corn Stalk Rind with a White Rot Fungus – Trametes hirsuta – and the Use of Delignified Corn Stalk Pith as a Pulp Additive

Corn stalk rind (CSR) was treated with Trametes hirsuta lg-9 and then refined into pulp. The biotreatment resulted in loss of paper strength and brightness, but energy consumption during refining (ECR) was reduced. Meanwhile, multiple linear regression was carried out, for which ECR served as the dependent variable, and the yield and infrared relative absorbance intensities at 3414 cm-1 and 1653 cm-1 of the biotreated CSR served as independent variables. Results showed that the determining parameters of the biotreated CSR may be used to predict the ECR. In this work, delignified corn stalk pith (CSP) was added to aspen alkaline hydrogen peroxide mechanical pulp (APMP). The CSP enhanced the strength properties of the aspen APMP and inhibited yellowing. The biomechanical pulping of CSR has the potential to produce a low-cost green pulp, and the delignified CSP can serve as a pulp additive

Diet-Induced High Serum Levels of Trimethylamine-N-oxide Enhance the Cellular Inflammatory Response without Exacerbating Acute Intracerebral Hemorrhage Injury in Mice

Trimethylamine-N-oxide (TMAO), an intestinal flora metabolite of choline, may aggravate atherosclerosis by inducing a chronic inflammatory response and thereby promoting the occurrence of cerebrovascular diseases. Knowledge about the influence of TMAO-related inflammatory response on the pathological process of acute stroke is limited. This study was designed to explore the effects of TMAO on neuroinflammation, brain injury severity, and long-term neurologic function in mice with acute intracerebral hemorrhage (ICH). We fed mice with either a regular chow diet or a chow diet supplemented with 1.2% choline pre- and post-ICH. In this study, we measured serum levels of TMAO with ultrahigh-performance liquid chromatography-tandem mass spectrometry at 24 h and 72 h post-ICH. The expression level of P38-mitogen-protein kinase (P38-MAPK), myeloid differentiation factor 88 (MyD88), high-mobility group box1 protein (HMGB1), and interleukin-1β (IL-1β) around hematoma was examined by western blotting at 24 h. Microglial and astrocyte activation and neutrophil infiltration were examined at 72 h. The lesion was examined on days 3 and 28. Neurologic deficits were examined for 28 days. A long-term choline diet significantly increased serum levels of TMAO compared with a regular diet at 24 h and 72 h after sham operation or ICH. Choline diet-induced high serum levels of TMAO did not enhance the expression of P38-MAPK, MyD88, HMGB1, or IL-1β at 24 h. However, it did increase the number of activated microglia and astrocytes around the hematoma at 72 h. Contrary to our expectations, it did not aggravate acute or long-term histologic damage or neurologic deficits after ICH. In summary, choline diet-induced high serum levels of TMAO increased the cellular inflammatory response probably by activating microglia and astrocytes. However, it did not aggravate brain injury or worsen long-term neurologic deficits. Although TMAO might be a potential risk factor for cerebrovascular diseases, this exploratory study did not support that TMAO is a promising target for ICH therapy.Peer Reviewe