1,125 research outputs found

    Two-Way Training for Discriminatory Channel Estimation in Wireless MIMO Systems

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    This work examines the use of two-way training to efficiently discriminate the channel estimation performances at a legitimate receiver (LR) and an unauthorized receiver (UR) in a multiple-input multiple-output (MIMO) wireless system. This work improves upon the original discriminatory channel estimation (DCE) scheme proposed by Chang et al where multiple stages of feedback and retraining were used. While most studies on physical layer secrecy are under the information-theoretic framework and focus directly on the data transmission phase, studies on DCE focus on the training phase and aim to provide a practical signal processing technique to discriminate between the channel estimation performances at LR and UR. A key feature of DCE designs is the insertion of artificial noise (AN) in the training signal to degrade the channel estimation performance at UR. To do so, AN must be placed in a carefully chosen subspace based on the transmitter's knowledge of LR's channel in order to minimize its effect on LR. In this paper, we adopt the idea of two-way training that allows both the transmitter and LR to send training signals to facilitate channel estimation at both ends. Both reciprocal and non-reciprocal channels are considered and a two-way DCE scheme is proposed for each scenario. {For mathematical tractability, we assume that all terminals employ the linear minimum mean square error criterion for channel estimation. Based on the mean square error (MSE) of the channel estimates at all terminals,} we formulate and solve an optimization problem where the optimal power allocation between the training signal and AN is found by minimizing the MSE of LR's channel estimate subject to a constraint on the MSE achievable at UR. Numerical results show that the proposed DCE schemes can effectively discriminate between the channel estimation and hence the data detection performances at LR and UR.Comment: 1

    Coordinated Multicasting with Opportunistic User Selection in Multicell Wireless Systems

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    Physical layer multicasting with opportunistic user selection (OUS) is examined for multicell multi-antenna wireless systems. By adopting a two-layer encoding scheme, a rate-adaptive channel code is applied in each fading block to enable successful decoding by a chosen subset of users (which varies over different blocks) and an application layer erasure code is employed across multiple blocks to ensure that every user is able to recover the message after decoding successfully in a sufficient number of blocks. The transmit signal and code-rate in each block determine opportunistically the subset of users that are able to successfully decode and can be chosen to maximize the long-term multicast efficiency. The employment of OUS not only helps avoid rate-limitations caused by the user with the worst channel, but also helps coordinate interference among different cells and multicast groups. In this work, efficient algorithms are proposed for the design of the transmit covariance matrices, the physical layer code-rates, and the target user subsets in each block. In the single group scenario, the system parameters are determined by maximizing the group-rate, defined as the physical layer code-rate times the fraction of users that can successfully decode in each block. In the multi-group scenario, the system parameters are determined by considering a group-rate balancing optimization problem, which is solved by a successive convex approximation (SCA) approach. To further reduce the feedback overhead, we also consider the case where only part of the users feed back their channel vectors in each block and propose a design based on the balancing of the expected group-rates. In addition to SCA, a sample average approximation technique is also introduced to handle the probabilistic terms arising in this problem. The effectiveness of the proposed schemes is demonstrated by computer simulations.Comment: Accepted by IEEE Transactions on Signal Processin

    Clean relaying aided cognitive radio under the coexistence constraint

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    We consider the interference-mitigation based cognitive radio where the primary and secondary users can coexist at the same time and frequency bands, under the constraint that the rate of the primary user (PU) must remain the same with a single-user decoder. To meet such a coexistence constraint, the relaying from the secondary user (SU) can help the PU's transmission under the interference from the SU. However, the relayed signal in the known dirty paper coding (DPC) based scheme is interfered by the SU's signal, and is not "clean". In this paper, under the half-duplex constraints, we propose two new transmission schemes aided by the clean relaying from the SU's transmitter and receiver without interference from the SU. We name them as the clean transmitter relaying (CT) and clean transmitter-receiver relaying (CTR) aided cognitive radio, respectively. The rate and multiplexing gain performances of CT and CTR in fading channels with various availabilities of the channel state information at the transmitters (CSIT) are studied. Our CT generalizes the celebrated DPC based scheme proposed previously. With full CSIT, the multiplexing gain of the CTR is proved to be better (or no less) than that of the previous DPC based schemes. This is because the silent period for decoding the PU's messages for the DPC may not be necessary in the CTR. With only the statistics of CSIT, we further prove that the CTR outperforms the rate performance of the previous scheme in fast Rayleigh fading channels. The numerical examples also show that in a large class of channels, the proposed CT and CTR provide significant rate gains over the previous scheme with small complexity penalties.Comment: 30 page

    Two-way training for discriminatory channel estimation in wireless MIMO systems

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    This work examines the use of two-way training to efficiently discriminate the channel estimation performances at a legitimate receiver (LR) and an unauthorized receiver (UR) in a multiple-input multiple-output (MIMO) wireless system. This work improves upon the original discriminatory channel estimation (DCE) scheme proposed by Chang where multiple stages of feedback and retraining were used. While most studies on physical layer secrecy are under the information-theoretic framework and focus directly on the data transmission phase, studies on DCE focus on the training phase and aim to provide a practical signal processing technique to discriminate between the channel estimation performances (and, thus, the effective received signal qualities) at LR and UR. A key feature of DCE designs is the insertion of artificial noise (AN) in the training signal to degrade the channel estimation performance at UR. To do so, AN must be placed in a carefully chosen subspace, based on the transmitter's knowledge of LR's channel, in order to minimize its effect on LR. In this paper, we adopt the idea of two-way training that allows both the transmitter and LR to send training signals to facilitate channel estimation at both ends. Both reciprocal and nonreciprocal channels are considered and a two-way DCE scheme is proposed for each scenario. For mathematical tractability, we assume that all terminals employ the linear minimum mean square error criterion for channel estimation. Based on the mean square error (MSE) of the channel estimates at all terminals, we formulate and solve an optimization problem where the optimal power allocation between the training signal and AN is found by minimizing the MSE of LR's channel estimate subject to a constraint on the MSE achievable at UR. Numerical results show that the proposed DCE schemes can effectively discriminate between the channel estimation and, hence, the data detection performances at LR and UR.This work was supported in part by the National Science Council, Taiwan, by Grant NSC 100-2628-E-007-025-MY3 and Grant NSC 101-2218-E-011-043, and in part by the Australian Research Council's Discovery Projects Funding Scheme (Project no.DP110102548)

    Tunnelling rates for the nonlinear Wannier-Stark problem

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    We present a method to numerically compute accurate tunnelling rates for a Bose-Einstein condensate which is described by the nonlinear Gross-Pitaevskii equation. Our method is based on a sophisticated real-time integration of the complex-scaled Gross-Pitaevskii equation, and it is capable of finding the stationary eigenvalues for the Wannier-Stark problem. We show that even weak nonlinearities have significant effects in the vicinity of very sensitive resonant tunnelling peaks, which occur in the rates as a function of the Stark field amplitude. The mean-field interaction induces a broadening and a shift of the peaks, and the latter is explained by analytic perturbation theory

    Simulating chemistry efficiently on fault-tolerant quantum computers

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    Quantum computers can in principle simulate quantum physics exponentially faster than their classical counterparts, but some technical hurdles remain. Here we consider methods to make proposed chemical simulation algorithms computationally fast on fault-tolerant quantum computers in the circuit model. Fault tolerance constrains the choice of available gates, so that arbitrary gates required for a simulation algorithm must be constructed from sequences of fundamental operations. We examine techniques for constructing arbitrary gates which perform substantially faster than circuits based on the conventional Solovay-Kitaev algorithm [C.M. Dawson and M.A. Nielsen, \emph{Quantum Inf. Comput.}, \textbf{6}:81, 2006]. For a given approximation error ϵ\epsilon, arbitrary single-qubit gates can be produced fault-tolerantly and using a limited set of gates in time which is O(logϵ)O(\log \epsilon) or O(loglogϵ)O(\log \log \epsilon); with sufficient parallel preparation of ancillas, constant average depth is possible using a method we call programmable ancilla rotations. Moreover, we construct and analyze efficient implementations of first- and second-quantized simulation algorithms using the fault-tolerant arbitrary gates and other techniques, such as implementing various subroutines in constant time. A specific example we analyze is the ground-state energy calculation for Lithium hydride.Comment: 33 pages, 18 figure

    Transverse-Momentum Dependence of the J/psi Nuclear Modification in d+Au Collisions at sqrt(s_NN)=200 GeV

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    We present measured J/psi production rates in d+Au collisions at sqrt(s_NN) = 200 GeV over a broad range of transverse momentum (p_T=0-14 GeV/c) and rapidity (-2.2<y<2.2). We construct the nuclear-modification factor R_dAu for these kinematics and as a function of collision centrality (related to impact parameter for the R_dAu collision). We find that the modification is largest for collisions with small impact parameters, and observe a suppression (R_dAu<1) for p_T<4 GeV/c at positive rapidities. At negative rapidity we observe a suppression for p_T1) for p_T>2 GeV/c. The observed enhancement at negative rapidity has implications for the observed modification in heavy-ion collisions at high p_T.Comment: 384 authors, 24 pages, 19 figures, 13 tables. Submitted to Phys. Rev. C. Plain text data tables for the points plotted in figures for this and previous PHENIX publications are publicly available at http://www.phenix.bnl.gov/phenix/WWW/info/data/ppg123_data.htm

    The Functional DRD3 Ser9Gly Polymorphism (rs6280) Is Pleiotropic, Affecting Reward as Well as Movement

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    Abnormalities of motivation and behavior in the context of reward are a fundamental component of addiction and mood disorders. Here we test the effect of a functional missense mutation in the dopamine 3 receptor (DRD3) gene (ser9gly, rs6280) on reward-associated dopamine (DA) release in the striatum. Twenty-six healthy controls (HCs) and 10 unmedicated subjects with major depressive disorder (MDD) completed two positron emission tomography (PET) scans with [11C]raclopride using the bolus plus constant infusion method. On one occasion subjects completed a sensorimotor task (control condition) and on another occasion subjects completed a gambling task (reward condition). A linear regression analysis controlling for age, sex, diagnosis, and self-reported anhedonia indicated that during receipt of unpredictable monetary reward the glycine allele was associated with a greater reduction in D2/3 receptor binding (i.e., increased reward-related DA release) in the middle (anterior) caudate (p<0.01) and the ventral striatum (p<0.05). The possible functional effect of the ser9gly polymorphism on DA release is consistent with previous work demonstrating that the glycine allele yields D3 autoreceptors that have a higher affinity for DA and display more robust intracellular signaling. Preclinical evidence indicates that chronic stress and aversive stimulation induce activation of the DA system, raising the possibility that the glycine allele, by virtue of its facilitatory effect on striatal DA release, increases susceptibility to hyperdopaminergic responses that have previously been associated with stress, addiction, and psychosis

    The effect of BPIFA1/SPLUNC1 genetic variation on its expression and function in asthmatic airway epithelium

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    Bacterial permeability family member A1 (BPIFA1), also known as short palate, lung, and nasal epithelium clone 1 (SPLUNC1), is a protein involved in the antiinflammatory response. The goal of this study was to determine whether BPIFA1 expression in asthmatic airways is regulated by genetic variations, altering epithelial responses to type 2 cytokines (e.g., IL-13). Nasal epithelial cells from patients with mild to severe asthma were collected from the National Heart, Lung. and Blood Institute Severe Asthma Research Program centers, genotyped for rs750064, and measured for BPIFA1. To determine the function of rs750064, cells were cultured at air-liquid interface and treated with 11-13 with or without recombinant human BPIFA1 (rhBPIFA1). Noncultured nasal cells with the rs750064 CC genotype had significantly less BPIFA1 mRNA expression than the CT and TT genotypes. Cultured CC versus CT and TT cells without stimulation maintained less BPIFA1 expression. With IL-13 treatment, CC genotype cells secreted more eotaxin-3 than CT and TT genotype cells. Also, rhBPIFA1 reduced IL-13-mediated eotaxin-3. BPIFA1 mRNA levels negatively correlated with serum IgE and fractional exhaled nitric oxide. Baseline FEV1% levels were lower in the asthma patients with the CC genotype (n = 1,016). Our data suggest that less BPIFA1 in asthma patients with the CC allele may predispose them to greater eosinophilic inflammation, which could be attenuated by rhBPIFA1 protein therapy.NIH/NHLBI [R01HL125128, U10HL109257, UL1TR00448, U10HL109168]This item from the UA Faculty Publications collection is made available by the University of Arizona with support from the University of Arizona Libraries. If you have questions, please contact us at [email protected]
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