Anomaly polynomial of E-string theories

We determine the anomaly polynomial of the E-string theory and its higher-rank generalizations, that is, the 6d $\mathcal{N} =(1, 0)$ superconformal theories on the worldvolume of one or multiple M5-branes embedded within the end-of-the-world brane with $E_8$ symmetry.Comment: v2: 16 pages; typos correcte

In vitro studies on target cells of oncogenic adenoviruses in hamster brain. II. In vitro transformation of brain cells of hamsters at various ages by human adenovirus type 12

In vitro transformations of brain cells of hamsters of various ages were examined after the administration of human adenovirus type 12 (Ad 12) to determine the type and origin of the target cell. Hamster brain cells at all examined ages were transformed by Ad12. Although the virus was not isolated, virus specific tumor antigen was demonstrated in the transformed cells. The histological features of tumors that developed by transplantation of transformed cells closely resembled Ad12-induced brain tumors. The transformed cell focus tended to appear near the embryonic brain cell (EB cell) or glioblastic cell (GB cell). The transformed cells were morphologically similar to the EB or GB cell. Some subcultured transformed cells showed a rosette-like pattern, and the surrounding space arrangement was similar to that of the ventricular wall. The incidence of brain cell transformations decreased with increased hamster age. This decreased incidence with age corresponded to the decreased numbers of EB or GB cells present in progressively older hamsters. From these results, it is concluded that the target cells of AD12 in hamster brain cell cultures are probably the EB or GB cells.</p

In vitro studies on target cells of oncogenic adenoviruses in hamster brain. III. In vitro transformation of brain cells of hamsters at various ages by bovine adenovirus type 3

In vitro transformation of brain cells of hamsters at various ages was examined after the addition of bovine adenovirus type 3 to determine the type and origin of the target cells. Cellular transformations occurred only in cultures of fetus and newborn animals and at low incidences. Nine cell lines were obtained. Virus specific tumor antigens were demonstrated in the transformed cells. The present investigation suggested that bovine adenovirus type 3 might transform mesenchymal cells (ME cell) and that these cells are probably of meningeal or vascular origin. The histological picture of tumors following transplantation of the transformed cells resembled human primary sarcoma of the meninges and brain.</p

Small instanton transitions for M5 fractions

M5-branes on an ADE singularity are described by certain six-dimensional "conformal matter" superconformal field theories. Their Higgs moduli spaces contain information about various dynamical processes for the M5s; however, they are not directly accessible due to the lack of a Lagrangian formulation. Using anomaly matching, we compute their dimensions. The result implies that M5 fractions can recombine in several different ways, where the M5s are leaving behind frozen versions of the singularity. The anomaly polynomial gives hints about the nature of the freezing. We also check the Higgs dimension formula by comparing it with various existing conjectures for the CFTs one obtains by torus compactifications down to four and three dimensions. Aided by our results, we also extend those conjectures to compactifications of theories not previously considered. These involve class S theories with twisted punctures in four dimensions, and affine-Dynkin-shaped quivers in three dimensions.Comment: 39 pages, 1 figure; v2 published in JHE

Brain tumors induced in rats by human adenovirus type 12

Oncogenesis of human adenovirus type 12 in the brain of rats was examined. Newborn rats of Sprague-Dawley and Donryu&#12288;strains were injected intracranially with human adenovirus type 12.&#12288;The incidence of intracranial tumors was 91% (30/33) in SpragueDawley&#12288;and 56% (14/25) in Donryu rats. Except for one tumor nodule&#12288;located in the parietal cortex of a Sprague.Dawley rat, all tumors&#12288;developed in the paraventricular areas or in the meninges. Tumors were quite similar histologically to those induced in hamsters and&#12288;mice resembling the undifferentiated human brain tumors such as medulloblastoma,&#12288;ependymoblastoma and embryonic gliomas. From&#12288;the histological features and primary sites of tumor development, it is&#12288;suggested that the tumors in the brain of rats induced by adenovirus type 12 originate from the embryonic cells in the&#12288;paraventricular area&#12288;and also from the undifferentiated supporting cells of the peripheral&#12288;nerves in the&#12288;leptomeninges.</p