Fast Vacuum Decay into Quark Pairs in Strong Color Electric and Magnetic Fields

We study quark-pair creations in strong color electromagnetic fields. We point out that, for massless quarks, the vacuum persistency probability per unit space-time volume is zero, i.e., the quark-pair creation rate w is infinite, in general homogeneous color electromagnetic fields, while it is finite when the color magnetic field is absent. We find that the contribution from the lowest Landau level (LLL) dominates this phenomenon. With an effective theory of the LLL projection, we also discuss dynamics of the vacuum decay, taking into account the back reaction of pair creations.Comment: 4 pages, 1 figure, contribution to the proceedings of International conference on the structure of baryons (BARYONS'10), RCNP, Osaka, Japan, Dec. 7-11, 2010; fig.2 delete

Two-band superconductivity featuring different anisotropies in the ternary iron silicide Lu2_{2}Fe3_{3}Si5_{5}

We report detailed studies of the upper critical field and low-temperature specific heat in the two-gap superconductor Lu$_{2}$Fe$_{3}$Si$_{5}$. The anisotropy of the upper critical field suggests that the active band is quasi-one-dimensional. Low-temperature specific heat in magnetic fields reveals that the virtual $H_{c2}$ in the passive band is almost isotropic. These results strongly indicate that the two bands have two different anisotropies, similar to the typical two-gap superconductor MgB$_{2}$, and their interplay may be essential to the two-gap superconductivity in Lu$_{2}$Fe$_{3}$Si$_{5}$.Comment: 5 pages, 5 figure

Metallic partial melting processes on the acapulcoite-lodranite parent body.

第3回極域科学シンポジウム/第35回南極隕石シンポジウム 11月29日（木） 国立国語研究所 2階講

Spatio-temporal variation of riverine N and P concentration in the Lake Hachiro watershed

Poster Sessio

PCNA–MutSα-mediated binding of MutLα to replicative DNA with mismatched bases to induce apoptosis in human cells

Modified bases, such as O(6)-methylguanines, are produced in cells exposed to alkylating agents and cause apoptosis. In human cells treated with N-methyl-N-nitrosourea, we detected a protein complex composed of MutSα, MutLα and PCNA on damaged DNA by immunoprecipitation method using chromatin extracts, in which protein–protein interactions were stabilized by chemical crosslinking. Time course experiments revealed that MutSα, consisting of MSH2 and MSH6 proteins, and PCNA bind to DNA to form an initial complex, and MutLα, composed of MLH1 and PMS2, binds to the complex when the DNA is damaged. This sequential mode of binding was further confirmed by the findings that the association of PCNA–MutSα complex on chromatin was observed even in the cells that lack MLH1, whereas in the absence of MSH2 no association of MutLα with the chromatin was achieved. Moreover, reduction in the PCNA content by small-interfering RNA or inhibition of DNA replication by aphidicolin, an inhibitor of DNA polymerase, significantly reduced the levels of the PCNA–MutSα–MutLα complex and also suppressed an increase in the caspase-3 activity, a hallmark for the induction of apoptosis. These observations imply that the induction of apoptosis is coupled with the progression of DNA replication through the action of PCNA