Hybrid Approach for Energy-Aware Synchronization in Sensor Networks

This book chapter discusses a time synchronization scheme for wireless sensor networks that aims to save sensor battery power while maintaining network connectivity for as long as possible

Anchor nodes placement for effective passive localization

This paper discusses anchor nodes placement for effective passive localization. The authors show that, for effective passive localization, the optimal placement of the anchor nodes is at the center of the network in such a way that no three anchor nodes share linearity

Nonuniform Grid-Based Coordinated Routing in Wireless Sensor Networks

A nonuniform grid-based coordinated routing design in wireless sensor networks is presented. The conditions leading to network partition and analysis of energy consumption that prolongs the network lifetime are studied. We focus on implementing routing in densely populated sensor networks. By maintaining constant values for parameters such as path loss exponent, receiver sensitivity and transmit power, and varying between uniform and non-uniform grids, we observe energy consumption patterns for each of the grid structures and infer from the network lifetime the better suited grids for uniformly and randomly deployed sensor nodes

Non-uniform grid-based routing in sensor networks

This paper discusses non-uniform grid-based routing in sensor networks

User-Based Channel Assignment Algorithm in a Load-Balanced IEEE 802.11 WLAN

This article discusses a user-based channel assignment algorithm in a load-balanced IEEE 802.11 WLAN

Channel Assignment and Load Distribution in a Power-Managed Wlan

This paper discusses a proposed algorithm

User-Based Channel Assignment Algorithm in a Load-Balanced IEEE 802.11 WLAN

This article discusses a user-based channel assignment algorithm in a load-balanced IEEE 802.11 WLAN

Progressive loss of CD3 expression after HTLV-I infection results from chromatin remodeling affecting all the CD3 genes and persists despite early viral genes silencing

BACKGROUND: HTLV-I infected CD4+ T-cells lines usually progress towards a CD3- or CD3low phenotype. In this paper, we studied expression, kinetics, chromatin remodeling of the CD3 gene at different time-points post HTLV-I infection. RESULTS: The onset of this phenomenon coincided with a decrease of CD3gamma followed by the subsequent progressive reduction in CD3delta, then CD3epsilon and CD3zeta mRNA. Transient transfection experiments showed that the CD3gamma promoter was still active in CD3- HTLV-I infected cells demonstrating that adequate amounts of the required transcription factors were available. We next looked at whether epigenetic mechanisms could be responsible for this progressive decrease in CD3 expression using DNase I hypersensitivity (DHS) experiments examining the CD3gamma and CD3delta promoters and the CD3delta enhancer. In uninfected and cells immediately post-infection all three DHS sites were open, then the CD3gamma promoter became non accessible, and this was followed by a sequential closure of all the DHS sites corresponding to all three transcriptional control regions. Furthermore, a continuous decrease of in vivo bound transcription initiation factors to the CD3gamma promoter was observed after silencing of the viral genome. Coincidently, cells with a lower expression of CD3 grew more rapidly. CONCLUSION: We conclude that HTLV-I infection initiates a process leading to a complete loss of CD3 membrane expression by an epigenetic mechanism which continues along time, despite an early silencing of the viral genome. Whether CD3 progressive loss is an epiphenomenon or a causal event in the process of eventual malignant transformation remains to be investigated.Journal ArticleResearch Support, Non-U.S. Gov'tinfo:eu-repo/semantics/publishe

Enhanced Channel Assignment and Load Distribution in IEEE 802.11 WLANs

This paper discusses enhanced channel assignment and load distribution in IEEE 802.11 WLANs