Performance of Anthurium (Anthurium anderanum Lindl) cultivars under hill zone of Karnataka

An investigation was carried out at experimental block, College of Horticulture, Mudigere. Tropical recorded maximum LAI 2.83, and had maximum plant height, number of leaves, leaf area and Leaf area Index. Cultivar Crinkle Red recorded maximum number of flowers per plant per year (13.14), which was on par with Tropical (11.77), Cheers (10.60) and Fire (10.25). Cultivar Midori recorded maximum vase life (35.00 days) followed by Tropical (33.33 days) and it was on par with Fire (32.22 days). Cultivar Midori recorded maximum vase life (35.00 days), followed by Tropical (33.33 days) and it was on par with Fire (32.22 days) and highest B:C ratio is recorded in cultivar Tropical (1.83) and it was least in Fantacia (1.13)

Alterations of renal phenotype and gene expression profiles due to protein overload in NOD-related mouse strains

BACKGROUND: Despite multiple causes, Chronic Kidney Disease is commonly associated with proteinuria. A previous study on Non Obese Diabetic mice (NOD), which spontaneously develop type 1 diabetes, described histological and gene expression changes incurred by diabetes in the kidney. Because proteinuria is coincident to diabetes, the effects of proteinuria are difficult to distinguish from those of other factors such as hyperglycemia. Proteinuria can nevertheless be induced in mice by peritoneal injection of Bovine Serum Albumin (BSA). To gain more information on the specific effects of proteinuria, this study addresses renal changes in diabetes resistant NOD-related mouse strains (NON and NOD.B10) that were made to develop proteinuria by BSA overload. METHODS: Proteinuria was induced by protein overload on NON and NOD.B10 mouse strains and histology and microarray technology were used to follow the kidney response. The effects of proteinuria were assessed and subsequently compared to changes that were observed in a prior study on NOD diabetic nephropathy. RESULTS: Overload treatment significantly modified the renal phenotype and out of 5760 clones screened, 21 and 7 kidney transcripts were respectively altered in the NON and NOD.B10. Upregulated transcripts encoded signal transduction genes, as well as markers for inflammation (Calmodulin kinase beta). Down-regulated transcripts included FKBP52 which was also down-regulated in diabetic NOD kidney. Comparison of transcripts altered by proteinuria to those altered by diabetes identified mannosidase 2 alpha 1 as being more specifically induced by proteinuria. CONCLUSION: By simulating a component of diabetes, and looking at the global response on mice resistant to the disease, by virtue of a small genetic difference, we were able to identify key factors in disease progression. This suggests the power of this approach in unraveling multifactorial disease processes

Clinico-Etiological Profile of Cardiac Tamponade in a Tertiary Care Centre

Background: Pericardial tamponade, a life-threatening condition caused by the accumulation of fluid in the pericardial sac, can be acute or chronic. Mortality and morbidity can be minimized on prompt diagnosis and treatment with percutaneous drainage