18 research outputs found
Activation of cardiac progenitor cells reverses the failing heart senescent phenotype and prolongs lifespan.
Get PDFNO METABOLITES ACCUMULATE IN ERYTHROCYTES IN PROPORTION TO CARBON DIOXIDE AND BICARBONATE CONCENTRATION.
No full textNitric oxide metabolites accumulation in erythrocytes is proportional to the bicarbonate concentration.
No full textAltered expression of a limited number of genes contributes to cardiac decompensation during chronic ventricular tachypacing in dogs.
No full textChanges in cardiac metabolism produced by acute inhibition of NO synthase can be reversed by a long-acting NO donor
No full textCytokines are not a requisite part of the pathophysiology leading to cardiac decompensation.
No full textAmlodipine releases nitric oxide (NO) from coronary microvessels after pacing induced dilated cardiomyopathy.
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Kinin-mediated coronary nitric oxide production contributes to the therapeutic action of angiotensin-converting enzyme and neutral endopeptidase inhibitors and amlodipine in the treatment in heart failure.
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