Multiple (inverse) binomial sums of arbitrary weight and depth and the all-order epsilon-expansion of generalized hypergeometric functions with one half-integer value of parameter

We continue the study of the construction of analytical coefficients of the epsilon-expansion of hypergeometric functions and their connection with Feynman diagrams. In this paper, we show the following results: Theorem A: The multiple (inverse) binomial sums of arbitrary weight and depth (see Eq. (1.1)) are expressible in terms of Remiddi-Vermaseren functions. Theorem B: The epsilon expansion of a hypergeometric function with one half-integer value of parameter (see Eq. (1.2)) is expressible in terms of the harmonic polylogarithms of Remiddi and Vermaseren with coefficients that are ratios of polynomials. Some extra materials are available via the www at this http://theor.jinr.ru/~kalmykov/hypergeom/hyper.htmlComment: 24 pages, latex with amsmath and JHEP3.cls; v2: some typos corrected and a few references added; v3: few references added

Ultrastructural Localisation of Antigen in Plasma Cells

Following a second injection of horse-radish peroxidase (HPO) or &lt;i&gt;E. coli&lt;/i&gt; alkaline phosphatase into the hind foot pads of immune mice, the medullae of the popliteal lymph nodes were examined for plasma cells containing antigen. Small numbers of plasma cells containing HPO were observed 8, 18 and 42 h after injection, but not at 3 and 5 days. The antigen was located in the cisternae of the rough endoplasmic reticulum of large mature-looking plasma cells. It is suggested that HPO antigen is taken up by plasma cells which are already producing specific antibodies. No alkaline phosphatase antigen was detected in plasma cells, the possible reasons for this are discussed.</jats:p

Intestinal Goblet Cell Differentiation in &lt;i&gt;Nippostrongylus&lt;/i&gt;-Infected Rats after Transfer of Fractionated Thoracic Duct Lymphocytes

Adoptive immunization of &lt;i&gt;Nippostrongylus&lt;/i&gt;-infected rats augmented goblet cell differentiation in the intestinal epithelium. After fractionation of immune thoracic duct lymphocytes (TDL), cells lacking surface immunoglobulin (sIg––) were the most potent stimulators of goblet cell differentiation. Moreover, TDL drained from rats harbouring a primary infection were more effective than TDL from hyperimmune rats.</jats:p

Role of Mucosal Mast Cells in Intestinal Graft-versus-Host Reaction in the Mouse

Hyperplasia of mucosal mast cells (MMC) is found in many enteropathies which are caused by T lymphocytes, but their exact role is unknown. In this study we have investigated whether MMC play a part in the immunologically mediated enteropathy which occurs in mice with graft-versus-host reaction (GvHR). There were simultaneous increases in the numbers of jejunal MMC and in the concentrations of mouse intestinal mast cell proteinase both in serum and in the intestine in two separate models of GvHR. Although these changes developed in parallel with the evolving GvHR, there was no correlation between the degree of MMC activation and the severity of the intestinal pathology. In addition, mast cell deficient W/W&lt;sup&gt;v&lt;/sup&gt; mice developed systemic and intestinal GvHR as severe as their normal congenic littermates, despite a markedly deficient MMC response. We conclude that the role of MMC in enteropathy may be to regulate or repair T lymphocyte mediated immunopathology.</jats:p