Effect of Charge Fluctuations on the Persistent Current through a Quantum Dot

We study coherent charge transfer between an Aharonov-Bohm ring and a side-attached quantum dot. The charge fluctuation between the two sub-structures is shown to give rise to algebraic suppression of the persistent current circulating the ring as the size of the ring becomes relatively large. The charge fluctuation at resonance provides transition between the diamagnetic and the paramagnetic states. Universal scaling, crossover behavior of the persistent current from a continuous to a discrete energy limit in the ring is also discussed.Comment: 5 pages, 4 figure

Study of 0-π\pi phase transition in hybrid superconductor-InSb nanowire quantum dot devices

Hybrid superconductor-semiconducting nanowire devices provide an ideal platform to investigating novel intragap bound states, such as the Andreev bound states (ABSs), Yu-Shiba-Rusinov (YSR) states, and the Majorana bound states. The competition between Kondo correlations and superconductivity in Josephson quantum dot (QD) devices results in two different ground states and the occurrence of a 0-$\pi$ quantum phase transition. Here we report on transport measurements on hybrid superconductor-InSb nanowire QD devices with different device geometries. We demonstrate a realization of continuous gate-tunable ABSs with both 0-type levels and $\pi$-type levels. This allow us to manipulate the transition between 0 and $\pi$ junction and explore charge transport and spectrum in the vicinity of the quantum phase transition regime. Furthermore, we find a coexistence of 0-type ABS and $\pi$-type ABS in the same charge state. By measuring temperature and magnetic field evolution of the ABSs, the different natures of the two sets of ABSs are verified, being consistent with the scenario of phase transition between the singlet and doublet ground state. Our study provides insights into Andreev transport properties of hybrid superconductor-QD devices and sheds light on the crossover behavior of the subgap spectrum in the vicinity of 0-$\pi$ transition

Spin Fluctuation Induced Dephasing in a Mesoscopic Ring

We investigate the persistent current in a hybrid Aharonov-Bohm ring - quantum dot system coupled to a reservoir which provides spin fluctuations. It is shown that the spin exchange interaction between the quantum dot and the reservoir induces dephasing in the absence of direct charge transfer. We demonstrate an anomalous nature of this spin-fluctuation induced dephasing which tends to enhance the persistent current. We explain our result in terms of the separation of the spin from the charge degree of freedom. The nature of the spin fluctuation induced dephasing is analyzed in detail.Comment: 4 pages, 4 figure

State space collapse and diffusion approximation for a network operating under a fair bandwidth sharing policy

We consider a connection-level model of Internet congestion control, introduced by Massouli\'{e} and Roberts [Telecommunication Systems 15 (2000) 185--201], that represents the randomly varying number of flows present in a network. Here, bandwidth is shared fairly among elastic document transfers according to a weighted $\alpha$-fair bandwidth sharing policy introduced by Mo and Walrand [IEEE/ACM Transactions on Networking 8 (2000) 556--567] [$\alpha\in (0,\infty)$]. Assuming Poisson arrivals and exponentially distributed document sizes, we focus on the heavy traffic regime in which the average load placed on each resource is approximately equal to its capacity. A fluid model (or functional law of large numbers approximation) for this stochastic model was derived and analyzed in a prior work [Ann. Appl. Probab. 14 (2004) 1055--1083] by two of the authors. Here, we use the long-time behavior of the solutions of the fluid model established in that paper to derive a property called multiplicative state space collapse, which, loosely speaking, shows that in diffusion scale, the flow count process for the stochastic model can be approximately recovered as a continuous lifting of the workload process.Comment: Published in at http://dx.doi.org/10.1214/08-AAP591 the Annals of Applied Probability (http://www.imstat.org/aap/) by the Institute of Mathematical Statistics (http://www.imstat.org

Diffusion-Limited Aggregation Processes with 3-Particle Elementary Reactions

A diffusion-limited aggregation process, in which clusters coalesce by means of 3-particle reaction, A+A+A->A, is investigated. In one dimension we give a heuristic argument that predicts logarithmic corrections to the mean-field asymptotic behavior for the concentration of clusters of mass $m$ at time $t$, $c(m,t)~m^{-1/2}(log(t)/t)^{3/4}$, for $1 << m << \sqrt{t/log(t)}$. The total concentration of clusters, $c(t)$, decays as $c(t)~\sqrt{log(t)/t}$ at $t --> \infty$. We also investigate the problem with a localized steady source of monomers and find that the steady-state concentration $c(r)$ scales as $r^{-1}(log(r))^{1/2}$, $r^{-1}$, and $r^{-1}(log(r))^{-1/2}$, respectively, for the spatial dimension $d$ equal to 1, 2, and 3. The total number of clusters, $N(t)$, grows with time as $(log(t))^{3/2}$, $t^{1/2}$, and $t(log(t))^{-1/2}$ for $d$ = 1, 2, and 3. Furthermore, in three dimensions we obtain an asymptotic solution for the steady state cluster-mass distribution: $c(m,r) \sim r^{-1}(log(r))^{-1}\Phi(z)$, with the scaling function $\Phi(z)=z^{-1/2}\exp(-z)$ and the scaling variable $z ~ m/\sqrt{log(r)}$.Comment: 12 pages, plain Te

Modelling spatially regulated B-catenin dynamics & invasion in intestinal crypts

Experimental data (e.g., genetic lineage and cell population studies) on intestinal crypts reveal that regulatory features of crypt behavior, such as control via morphogen gradients, are remarkably well conserved among numerous organisms (e.g., from mouse and rat to human) and throughout the different regions of the small and large intestines. In this article, we construct a partial differential equation model of a single colonic crypt that describes the spatial distribution of Wnt pathway proteins along the crypt axis. The novelty of our continuum model is that it is based upon assumptions that can be directly related to processes at the cellular and subcellular scales. We use the model to predict how the distributions of Wnt pathway proteins are affected by mutations. The model is then extended to investigate how mutant cell populations can invade neighboring crypts. The model simulations suggest that cell crowding caused by increased proliferation and decreased cell loss may be sufficient for a mutant cell population to colonize a neighboring healthy crypt