1,012 research outputs found

    Quantum data hiding with spontaneous parameter down-conversion

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    Here we analyze the practical implication of the existing quantum data hiding protocol with Bell states produced with optical downconverter. We show that the uncertainty for the producing of the Bell states with spontaneous parameter down-conversion should be taken into account, because it will cause serious trouble to the hider encoding procedure. A set of extended Bell states and a generalized Bell states analyzer are proposed to describe and analyze the possible states of two photons distributing in two paths. Then we present a method to integrate the above uncertainty of Bell states preparation into the dating hiding procedure, when we encode the secret with the set of extended Bell states. These modifications greatly simplify the hider's encoding operations, and thus paves the way for the implementation of quantum data hiding with present-day quantum optics.Comment: 4 pages, 1 figure, adding some analyse for security proof, to be appear in Phys. Rev.

    General-Purpose Parallel Simulator for Quantum Computing

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    With current technologies, it seems to be very difficult to implement quantum computers with many qubits. It is therefore of importance to simulate quantum algorithms and circuits on the existing computers. However, for a large-size problem, the simulation often requires more computational power than is available from sequential processing. Therefore, the simulation methods using parallel processing are required. We have developed a general-purpose simulator for quantum computing on the parallel computer (Sun, Enterprise4500). It can deal with up-to 30 qubits. We have performed Shor's factorization and Grover's database search by using the simulator, and we analyzed robustness of the corresponding quantum circuits in the presence of decoherence and operational errors. The corresponding results, statistics and analyses are presented.Comment: 15 pages, 15 figure

    Study on the Activity of PI3K/AKT, Death Receptor and 14-3-3 Mediated Signaling Pathways Regulating Hepatocyte Apoptosis during Rat Liver Regeneration

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    Studies have shown that apoptosis is closely related to the rat liver regeneration. To understand the mechanism of hepatocyte apoptosis during rat liver regeneration at the gene transcription level, the Rat Genome 230 2.0 Array was used to determine the expression changes of genes. Then the genes associated with cell apoptosis were searched by GO and NCBI databases, and cell apoptosis signaling pathways were searched by the database of QIAGEN and KEGG. Their signaling activities were calculated by spectral function E(t). The mechanism of hepatocyte apoptosis during rat liver regeneration was analyzed by Ingenuity Pathway Analysis 9.0 (IPA). The results showed that among the 27 signaling pathways regulating cell apoptosis, the E(t) values of Apoptosis signaling pathway and 14-3-3 mediated signaling pathway were significantly increased in the progression phase (6-72h after PH) of rat liver regeneration, and the E(t) values of hepatocyte apoptosis mediated by mitochondria rout were also significantly increased. The E(t) values of death receptor signaling pathway and PI3K/AKT branch of 14-3-3 mediated signaling pathway were significantly increased in the progression phase and the terminal phase (72-168h after PH) of rat liver regeneration, and the E(t) values of hepatocyte apoptosis mediated by cytomembrane route and nucleus route were also significantly increased. Conclusion: PI3K/AKT, death receptor and mitochondria branch played a key role in promoting cell apoptosis during rat liver regeneration

    Chiral extrapolation of lattice data for B-meson decay constant

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    The B-meson decay constant fB has been calculated from unquenched lattice QCD in the unphysical region. For extrapolating the lattice data to the physical region, we propose a phenomenological functional form based on the effective chiral perturbation theory for heavy mesons, which respects both the heavy quark symmetry and the chiral symmetry, and the non-relativistic constituent quark model which is valid at large pion masses. The inclusion of pion loop corrections leads to nonanalytic contributions to fB when the pion mass is small. The finite-range regularization technique is employed for the resummation of higher order terms of the chiral expansion. We also take into account the finite volume effects in lattice simulations. The dependence on the parameters and other uncertainties in our model are discussed.Comment: 11 pages, 3 Postscript figures, accepted for publication in EPJ

    Detailed quantitative comparison of half-bridge modular multilevel converter modelling methods

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    This paper presents a detailed comparison of different modelling methods of the half-bridge modular multilevel converter (HB-MMC), namely, switching function, Thevenin equivalent and averaged, considering both MMC implementations (large and reduced number of cells). The theoretical basis that underpins each modelling method are discussed. Offline PSCAD simulations are used to validate user-defined switching function and averaged MMC models against the Thevenin equivalent model provided in PSCAD library for accuracy, considering steady-state and dc fault conditions. Furthermore, the RTDS based real-time simulation results of the user-defined HB-MMC switching function model are validated against the above mentioned offline models, considering steady-state and dc short circuit fault operations. Simulation speed and efficiency of different offline HB-MMC models being studied in this paper are compared. From comprehensive corroboration of different HB-MMC models presented in this paper, it has been found that the averaged, switching function and Thevenin equivalent models produce practically identical results during steady-state and dc faults. In detailed offline and real-time simulation studies where fundamental and harmonic dynamics are of interest, switching function model is found to be faster and computational efficient compared to the Thevenin equivalent model

    Chiral Extrapolation of Lattice Data for Heavy Baryons

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    The masses of heavy baryons containing a b quark have been calculated numerically in lattice QCD with pion masses which are much larger than its physical value. In the present work we extrapolate these lattice data to the physical mass of the pion by applying the effective chiral Lagrangian for heavy baryons, which is invariant under chiral symmetry when the light quark masses go to zero and heavy quark symmetry when the heavy quark masses go to infinity. A phenomenological functional form with three parameters, which has the correct behavior in the chiral limit and appropriate behavior when the pion mass is large, is proposed to extrapolate the lattice data. It is found that the extrapolation deviates noticably from the naive linear extrapolation when the pion mass is smaller than about 500MeV. The mass differences between Sigma_b and Sigma_b^* and between Sigma_b^{(*)} and Lambda_b are also presented. Uncertainties arising from both lattice data and our model parameters are discussed in detail. We also give a comparision of the results in our model with those obtained in the naive linear extrapolations.Comment: 29 pages, 9 figure

    Controlled order rearrangement encryption for quantum key distribution

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    A novel technique is devised to perform orthogonal state quantum key distribution. In this scheme, entangled parts of a quantum information carrier are sent from Alice to Bob through two quantum channels. However before the transmission, the orders of the quantum information carrier in one channel is reordered so that Eve can not steal useful information. At the receiver's end, the order of the quantum information carrier is restored. The order rearrangement operation in both parties is controlled by a prior shared control key which is used repeatedly in a quantum key distribution session.Comment: 5 pages and 2 figure

    Collapse of ρxx\rho_{xx} ringlike structures in 2DEGs under tilted magnetic fields

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    In the quantum Hall regime, the longitudinal resistivity ρxx\rho_{xx} plotted as a density--magnetic-field (n2DBn_{2D}-B) diagram displays ringlike structures due to the crossings of two sets of spin split Landau levels from different subbands [e.g., Zhang \textit{et al.}, Phys. Rev. Lett. \textbf{95}, 216801 (2005)]. For tilted magnetic fields, some of these ringlike structures "shrink" as the tilt angle is increased and fully collapse at θc6\theta_c \approx 6^\circ. Here we theoretically investigate the topology of these structures via a non-interacting model for the 2DEG. We account for the inter Landau-level coupling induced by the tilted magnetic field via perturbation theory. This coupling results in anti-crossings of Landau levels with parallel spins. With the new energy spectrum, we calculate the corresponding n2DBn_{2D}-B diagram of the density of states (DOS) near the Fermi level. We argue that the DOS displays the same topology as ρxx\rho_{xx} in the n2DBn_{2D}-B diagram. For the ring with filling factor ν=4\nu=4, we find that the anti-crossings make it shrink for increasing tilt angles and collapse at a large enough angle. Using effective parameters to fit the θ=0\theta = 0^\circ data, we find a collapsing angle θc3.6\theta_c \approx 3.6^\circ. Despite this factor-of-two discrepancy with the experimental data, our model captures the essential mechanism underlying the ring collapse.Comment: 3 pages, 2 figures; Proceedings of the PASPS V Conference Held in August 2008 in Foz do Igua\c{c}u, Brazi

    Study on the Activity of PI3K/AKT, Death Receptor and 14-3-3 Mediated Signaling Pathways Regulating Hepatocyte Apoptosis during Rat Liver Regeneration

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    Studies have shown that apoptosis is closely related to the rat liver regeneration. To understand the mechanism of hepatocyte apoptosis during rat liver regeneration at the gene transcription level, the Rat Genome 230 2.0 Array was used to determine the expression changes of genes. Then the genes associated with cell apoptosis were searched by GO and NCBI databases, and cell apoptosis signaling pathways were searched by the database of QIAGEN and KEGG. Their signaling activities were calculated by spectral function E(t). The mechanism of hepatocyte apoptosis during rat liver regeneration was analyzed by Ingenuity Pathway Analysis 9.0 (IPA). The results showed that among the 27 signaling pathways regulating cell apoptosis, the E(t) values of Apoptosis signaling pathway and 14-3-3 mediated signaling pathway were significantly increased in the progression phase (6-72h after PH) of rat liver regeneration, and the E(t) values of hepatocyte apoptosis mediated by mitochondria rout were also significantly increased. The E(t) values of death receptor signaling pathway and PI3K/AKT branch of 14-3-3 mediated signaling pathway were significantly increased in the progression phase and the terminal phase (72-168h after PH) of rat liver regeneration, and the E(t) values of hepatocyte apoptosis mediated by cytomembrane route and nucleus route were also significantly increased. Conclusion: PI3K/AKT, death receptor and mitochondria branch played a key role in promoting cell apoptosis during rat liver regeneration

    Energy transfer in a fast-slow Hamiltonian system

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    We consider a finite region of a lattice of weakly interacting geodesic flows on manifolds of negative curvature and we show that, when rescaling the interactions and the time appropriately, the energies of the flows evolve according to a non linear diffusion equation. This is a first step toward the derivation of macroscopic equations from a Hamiltonian microscopic dynamics in the case of weakly coupled systems
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