Electrocardiographic vectorcardiographic alterations and laboratory findings in experimentally myocardial infarcted dogs

In this study significant degrees of changes (p<0 05) according to the size of the infarct area were detected in the haematological (erythrocyte sedimentation rate and leukocyte count values increased) biochemical (LDH and CK MB levels increased) and electrocardiographical (increase in heart frequency and single or multiple focuses of ventricular arrhythmias) findings in a total of 21 experimentally left right and biventricular myocardial infarcted dogs Left ventricular infarctions caused patologic Q wave elevation in ST segment and rotation in the T wave particularly in the I aVL V2 V3 and V10 leads and right ventricular myocardial infarctions caused these particularly in the V1 V4 and V5 and partially in the III and aVF leads and left axis deviation occurred even in individuals with small areas of infarction

Estimation of Electrocardiographic values in healthy Karabash dogs

The aim of this study was to determine the normal electrocardiographic and vectorcardiographic values of healthy Karabash dogs, a breed unique to Turkey, in order to provide information for other interested researchers

Electrocardiographically determination of cardiac enlargements in dogs

In this study the electrocardiographic parameters necessary to determine cardiac enlargements and to establish and distinguish such complaints from each other in the early stage in dogs with circulatory problems were assesse

Determination of Corrected QT Interval in Kangal Breed Dogs

QT interval is one of the important reasons of severe and life threatening ventricular arrhythmias in humans and animals Many formulas have been developed for correcting the QT interval however the best formula did not define yet by the researchers. For this reason, forty-nine clinically healthy Kangal dogs without cardiac problems other than sinus arrhythmia were included to the study. In this study corrected QT interval was determined by Bazet (QTcB = QT/root RR), Fridericia (QTcF = QT/(3)root RR), Famingham (QT+0.154(1-RR)), Van der Water (QTcVdW = QT-0.087((60/HR)-1)), Hodges (QT+0.00175(HR-60)), Matsunaga (QTcM = log600 QT/logRR) formulas. As a result, Bazett's formula showed the best consistency because the slope curve of the regression line was closest to zero (-0.00743) in our study. (C) 2018 PVJ. All rights reserve

M-mode echocardiographic parameters and indices in the normal german shepherd dog

M-mode echocardiographic measurements were made from 50 healthy German Shepherd dogs (30 males and 20 females). The dogs were awake and unsedated, in right lateral recumbent position. The following parameters were measured on the echocardiographic images: interventricular septal thickness at end-diastole (IVSd), interventricular septal thickness at end-systole (IVSs), left ventricular internal dimension at end-diastole (LVIDd), left ventricular internal dimension at end-systole (LVIDs), left ventricular posterior wall thickness at end-diastole (LVPWd), left ventricular posterior wall thickness at end-systole (LVPWs), left atrial dimension (LAD), aortic root dimension (AOD), left atrial to aortic root ratio (LAD/AOD), right ventricular internal dimension at end-diastole (RVID), amplitude of mitral valve excursion (DE amplitude), velocity of mitral valve opening (D-E slope), and velocity of mitral valve closure (E-F slope). Fractional shortening (FS) was also calculated. The effect of gender and age on each echocardiographic parameter was analyzed and the relationship between body weight (BW) and each parameter was also investigated. There was a significant relationship between gender and LVPW in systole and diastole and FS. Significant association was also found between BW and IVS, LVID, and LVPW in systole and diastole, FS, LAD, AOD, RVID, DE amplitude, and D-E slope of the mitral valve

Dilated cardiomyopathy in a spider monkey (Ateles paniscus)

Background A spider monkey with severe dyspnea was referred to our clinic

Dilated cardiomyopathy in a spider monkey (Ateles paniscus)

Background A spider monkey with severe dyspnea was referred to our clinic

Serum protein electrophoresis in dogs with intestinal parasites

The serum of 66 dogs with intestinal parasites (showing gastrointestinal problems caused by taeniosis, coccidiosis, ancylostomosis, trichuriosis and ascarididosis) was examined by electrophoresis. There were 6 dogs with coccidiosis, 6 dogs with ancylostomosis, 6 dogs with trichuriosis. 24 dogs with taeniosis and 24 dogs with ascarididosis. After agar gel protein electorphoresis of the serum samples, alpha 1 globulin levels were significantly lower in the coccidiosis group than in the other groups (p<0.05). While alpha 2 globulin levels increased in the ancylostomosis group (p<0.05), these levels decreased significantly in the dogs with ascarididosis (p<0.05). There was no change in beta and gamma globulin levels in any of the groups. While the protein level increased in the dogs with taeniosis (p<0.05), these levels were lower in the coccididosis group than in the other groups (p<0.05)

Determination of intracardiac blood pressures and cardiac functions in dogs

Intracardiac blood pressure was determined in twenty-seven healthy adult mongrel dogs weighting 14-32 kg. Cardiac catheterization was used to measure intracardiac blood pressure. A Pingtail catheter was placed in the aorta and the left ventricle via the A. carotis communis, and a Swanz-Ganz catheter was placed in the right ventricle via the V. jugularis. End-systolic blood pressure was found to be 113+/-3.1 mm/Hg in the left ventricle 53+/-2.9 mm/Hg in the right ventricle, 3+/-2.5 mm/Hg in the left atrium, 3+/-1.3 mm/Hg in the right atrium and 127+/-3 mm/Hg in the aorta. In addition, times of contraction, relaxation, filling, preejection and ejection of the heart were calculated using blood pressure traces. While there was a positive correlation between end sistolic blood pressure in the aorta and end sistolic blood pressure in the left ventricle (r=0.071, p<0.001), there was a negative corelation between RV relaxion time and Ao end sistolic blood pressure (r=-0.064, p<0.05)