19,501 research outputs found
Valence Quark Distribution in A=3 Nuclei
We calculate the quark distribution function for 3He/3H in a relativistic
quark model of nuclear structure which adequately reproduces the nucleon
approximation, nuclear binding energies, and nuclear sizes for small nuclei.
The results show a clear distortion from the quark distribution function for
individual nucleons (EMC effect) arising dominantly from a combination of
recoil and quark tunneling effects. Antisymmetrization (Pauli) effects are
found to be small due to limited spatial overlaps. We compare our predictions
with a published parameterization of the nuclear valence quark distributions
and find significant agreement.Comment: 18pp., revtex4, 4 fig
Semi-regular masas of transfinite length
In 1965 Tauer produced a countably infinite family of semi-regular masas in
the hyperfinite factor, no pair of which are conjugate by an
automorphism. This was achieved by iterating the process of passing to the
algebra generated by the normalisers and, for each , finding
masas for which this procedure terminates at the -th stage. Such masas are
said to have length . In this paper we consider a transfinite version of
this idea, giving rise to a notion of ordinal valued length. We show that all
countable ordinals arise as lengths of semi-regular masas in the hyperfinite
factor. Furthermore, building on work of Jones and Popa, we
obtain all possible combinations of regular inclusions of irreducible
subfactors in the normalising tower.Comment: 14 page
Generation and remote detection of THz sound using semiconductor superlattices
The authors introduce a novel approach to study the propagation of high
frequency acoustic phonons in which the generation and detection involves two
spatially separated superlattices apart. Propagating modes
of frequencies up to escape from the superlattice where they
are generated and reach the second superlattice where they are detected. The
measured frequency spectrum reveals finite size effects, which can be accounted
for by a continuum elastic model.Comment: Submitted to Applied Physics Letter
The Competitive Performance of Life Insurance Firms in the Retirement Asset Market
This paper summarizes the findings of the joint Wharton Financial Institutions Center and KPMG study of the retirement assets market and the role of life insurance companies within it. The study began with the following goals: Investigate how people save for retirement and whether this is adequate. Determine the primary products and institutions of the retirement asset market and observe how these have changed through time. Key findings: For most, asset accumulation is less than adequate for a comfortable retirement. The average worker exhibits little of the needed financial understanding to adequately plan for retirement. Upon retirement, households do not spend down their assets optimally. The retirement asset market is rapidly expanding. Products in retirement portfolios have shifted with time. The market share of mutual funds has exploded, mostly at the expense of depository institutions. Life insurance companies maintain a large, but slipping share.
Positive regulation of meiotic DNA double-strand break formation by activation of the DNA damage checkpoint kinase Mec1(ATR)
During meiosis, formation and repair of programmed DNA double-strand breaks (DSBs) create genetic exchange between homologous chromosomes-a process that is critical for reductional meiotic chromosome segregation and the production of genetically diverse sexually reproducing populations. Meiotic DSB formation is a complex process, requiring numerous proteins, of which Spo11 is the evolutionarily conserved catalytic subunit. Precisely how Spo11 and its accessory proteins function or are regulated is unclear. Here, we use Saccharomyces cerevisiae to reveal that meiotic DSB formation is modulated by the Mec1(ATR) branch of the DNA damage signalling cascade, promoting DSB formation when Spo11-mediated catalysis is compromised. Activation of the positive feedback pathway correlates with the formation of single-stranded DNA (ssDNA) recombination intermediates and activation of the downstream kinase, Mek1. We show that the requirement for checkpoint activation can be rescued by prolonging meiotic prophase by deleting the NDT80 transcription factor, and that even transient prophase arrest caused by Ndt80 depletion is sufficient to restore meiotic spore viability in checkpoint mutants. Our observations are unexpected given recent reports that the complementary kinase pathway Tel1(ATM) acts to inhibit DSB formation. We propose that such antagonistic regulation of DSB formation by Mec1 and Tel1 creates a regulatory mechanism, where the absolute frequency of DSBs is maintained at a level optimal for genetic exchange and efficient chromosome segregation
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