BARBELL ACCELERATION ANALYSIS ON VARIOUS INTENSITIES OF WEIGHTLIFTING

The purpose of this study was to examine how various intensity levels influence the peak barbell acceleration in weightlifting. USA weightlifting resident team members (n=9, men:5 & women:4) participated in this study. They performed two repetitions at intensities of 80, 85, and 90% of 1 repetition maximum (total six repetitions). The peak barbell acceleration was measured at the 2nd pull phase of the snatch/clean. A one-way repeated measure ANOVA was used to analyze the effects of the intensity levels (p = .05). The results showed that intensity has a significant effect on the peak barbell acceleration (F(2,16) = 11.49, p < .001). The peak barbell acceleration decreased as the intensity level increased (80%: 19.63±3.04, 85%: 16.78±3.56, 90%: 13.65±3.50). Comparison between elite and beginners or other power-oriented athletes can be considered in future studies

Fluoxetine during Development Reverses the Effects of Prenatal Stress on Depressive-Like Behavior and Hippocampal Neurogenesis in Adolescence

Depression during pregnancy and the postpartum period is a growing health problem, which affects up to 20% of women. Currently, selective serotonin reuptake inhibitor (SSRIs) medications are commonly used for treatment of maternal depression. Unfortunately, there is very little research on the long-term effect of maternal depression and perinatal SSRI exposure on offspring development. Therefore, the aim of this study was to determine the role of exposure to fluoxetine during development on affective-like behaviors and hippocampal neurogenesis in adolescent offspring in a rodent model of maternal depression. To do this, gestationally stressed and non-stressed Sprague-Dawley rat dams were treated with either fluoxetine (5 mg/kg/day) or vehicle beginning on postnatal day 1 (P1). Adolescent male and female offspring were divided into 4 groups: 1) prenatal stress+fluoxetine exposure, 2) prenatal stress+vehicle, 3) fluoxetine exposure alone, and 4) vehicle alone. Adolescent offspring were assessed for anxiety-like behavior using the Open Field Test and depressive-like behavior using the Forced Swim Test. Brains were analyzed for endogenous markers of hippocampal neurogenesis via immunohistochemistry. Results demonstrate that maternal fluoxetine exposure reverses the reduction in immobility evident in prenatally stressed adolescent offspring. In addition, maternal fluoxetine exposure reverses the decrease in hippocampal cell proliferation and neurogenesis in maternally stressed adolescent offspring. This research provides important evidence on the long-term effect of fluoxetine exposure during development in a model of maternal adversity