Waves in a plane graphene – dielectric waveguide structure

The features of the guided TE modes propagation have been investigated on the basis of computer simulations in a planar structure consisting of a set of alternating layers of dielectric and graphene. Within the framework of the effective medium approximation, the dispersion relations have been received for symmetric and antisymmetric waveguide modes, determined by the frequency range of their existence. The wave field distribution by structure, frequency dependences of the constants of propagation and transverse components of the wave vectors, as well as group and phase velocities of waveguide modes have been obtained, the effect of the graphene part in a structure on the waveguide mode behavior has been shown

A Surface Plasmon–Polariton in a Symmetric Dielectric Waveguide with Active Graphene Plates

A theoretical study of the plasmon modes’ characteristics is carried out in a structure consisting of two active graphene layers separated by a dielectric barrier layer. A general dispersion relation is obtained, the numerical analysis of which reveals the possibility of controlling the parameters of amplified surface modes in the region of graphene negative conductivity. In particular, their dispersion is controlled by changing the chemical potential of the graphene layers. For antisymmetric plasmons, their dependence on the barrier layer parameters was revealed. An increase in the chemical potential makes it possible to expand the region of existence of the amplified plasmons, which is accompanied not only by an increase in the amplification coefficient but also by a shift to the region of higher frequencies of the amplified modes. For the first time, modal bistability was also demonstrated in a limited frequency range for antisymmetric plasmons, due to the appearance of additional modes, in which the phase velocity decreases sharply near the cutoff, and the group velocities of the modes entering the bistability turn out to be opposite in sign. The frequency dependences of the real and imaginary parts of the plasmon propagation constant are analyzed, the distributions of wave fields in the structure are plotted, and the frequency dependence of the depth of the plasmon–polariton is given

Study of Systems Error Compensation Methods Based on Molecular-Electronic Transducers of Motion Parameters

The main objective of the paper is to study the system errors of azimuth determination in the dynamic scheme of north finding on the base of the molecular-electronic sensitive angular motion sensor. Introduced theoretical and experimental study of some error compensation methods. Investigated the most significant system inaccuracies of azimuth determination depended on MET sensor g-sensitivity factor and the occurrence of rotation uneven in the system and as a result of tiny angular accelerations which appeared. Methods and algorithms of error reduce are experimentally verified

Поверхностные плазмон-поляритоны в двуслойной графеновой структуре с диэлектрическим барьерным слоем при различных режимах управления

The dispersion dependences of a symmetric dielectric waveguide with graphene plates were compared in two regimes of graphene potential control from two sides of the structure. A general dispersion relation was obtained, the numerical analysis of which reveals the possibility of controlling the parameters of amplified surface modes. A numerical analysis of the dispersion relation solutions in the case of symmetry and asymmetry of the control potential on graphene layers has been carried out. A solution has been found that grows in amplitude with time in the region of negative conductivity of graphene in the presence of an inverse population of the levelsСравниваются дисперсионные зависимости симметричного диэлектрического волновода с графеновыми обкладками в двух режимах управления потенциалом графена с двух сторон структуры. Получено общее дисперсионное соотношение, численный анализ которого выявил возможность управления параметрами усиливаемых поверхностных мод. Проведен численный анализ решений дисперсионного соотношения в случае симметрии и асимметрии управляющего потенциала на графеновых слоях. В области отрицательной проводимости графена при наличии инверсной населенности уровней обнаружено решение, растущее по амплитуде во времен

ALK-positive histiocytosis: a new clinicopathologic spectrum highlighting neurologic involvement and responses to ALK inhibition

ALK-positive histiocytosis is a rare subtype of histiocytic neoplasm first described in 2008 in 3 infants with multisystemic disease involving the liver and hematopoietic system. This entity has subsequently been documented in case reports and series to occupy a wider clinicopathologic spectrum with recurrent KIF5B-ALK fusions. The full clinicopathologic and molecular spectra of ALK-positive histiocytosis remain, however, poorly characterized. Here, we describe the largest study of ALK-positive histiocytosis to date, with detailed clinicopathologic data of 39 cases, including 37 cases with confirmed ALK rearrangements. The clinical spectrum comprised distinct clinical phenotypic groups: infants with multisystemic disease with liver and hematopoietic involvement, as originally described (Group 1A: 6/39), other patients with multisystemic disease (Group 1B: 10/39), and patients with single-system disease (Group 2: 23/39). Nineteen patients of the entire cohort (49%) had neurologic involvement (7 and 12 from Groups 1B and 2, respectively). Histology included classic xanthogranuloma features in almost one-third of cases, whereas the majority displayed a more densely cellular, monomorphic appearance without lipidized histiocytes but sometimes more spindled or epithelioid morphology. Neoplastic histiocytes were positive for macrophage markers and often conferred strong expression of phosphorylated extracellular signal-regulated kinase, confirming MAPK pathway activation. KIF5B-ALK fusions were detected in 27 patients, whereas CLTC-ALK, TPM3-ALK, TFG-ALK, EML4-ALK, and DCTN1-ALK fusions were identified in single cases. Robust and durable responses were observed in 11/11 patients treated with ALK inhibition, 10 with neurologic involvement. This study presents the existing clinicopathologic and molecular landscape of ALK-positive histiocytosis and provides guidance for the clinical management of this emerging histiocytic entity

ALK-positive histiocytosis: a new clinicopathologic spectrum highlighting neurologic involvement and responses to ALK inhibition

ALK-positive histiocytosis is a rare subtype of histiocytic neoplasm first described in 2008 in 3 infants with multisystemic disease involving the liver and hematopoietic system. This entity has subsequently been documented in case reports and series to occupy a wider clinicopathologic spectrum with recurrent KIF5B-ALK fusions. The full clinicopathologic and molecular spectra of ALK-positive histiocytosis remain, however, poorly characterized. Here, we describe the largest study of ALK-positive histiocytosis to date, with detailed clinicopathologic data of 39 cases, including 37 cases with confirmed ALK rearrangements. The clinical spectrum comprised distinct clinical phenotypic groups: infants with multisystemic disease with liver and hematopoietic involvement, as originally described (Group 1A: 6/39), other patients with multisystemic disease (Group 1B: 10/39), and patients with single-system disease (Group 2: 23/39). Nineteen patients of the entire cohort (49%) had neurologic involvement (7 and 12 from Groups 1B and 2, respectively). Histology included classic xanthogranuloma features in almost one-third of cases, whereas the majority displayed a more densely cellular, monomorphic appearance without lipidized histiocytes but sometimes more spindled or epithelioid morphology. Neoplastic histiocytes were positive for macrophage markers and often conferred strong expression of phosphorylated extracellular signal-regulated kinase, confirming MAPK pathway activation. KIF5B-ALK fusions were detected in 27 patients, whereas CLTC-ALK, TPM3-ALK, TFG-ALK, EML4-ALK, and DCTN1-ALK fusions were identified in single cases. Robust and durable responses were observed in 11/11 patients treated with ALK inhibition, 10 with neurologic involvement. This study presents the existing clinicopathologic and molecular landscape of ALK-positive histiocytosis and provides guidance for the clinical management of this emerging histiocytic entity