661 research outputs found

    Representation Theory Approach to the Polynomial Solutions of q - Difference Equations : U_q(sl(3)) and Beyond,

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    A new approach to the theory of polynomial solutions of q - difference equations is proposed. The approach is based on the representation theory of simple Lie algebras and their q - deformations and is presented here for U_q(sl(n)). First a q - difference realization of U_q(sl(n)) in terms of n(n-1)/2 commuting variables and depending on n-1 complex representation parameters r_i, is constructed. From this realization lowest weight modules (LWM) are obtained which are studied in detail for the case n=3 (the well known n=2 case is also recovered). All reducible LWM are found and the polynomial bases of their invariant irreducible subrepresentations are explicitly given. This also gives a classification of the quasi-exactly solvable operators in the present setting. The invariant subspaces are obtained as solutions of certain invariant q - difference equations, i.e., these are kernels of invariant q - difference operators, which are also explicitly given. Such operators were not used until now in the theory of polynomial solutions. Finally the states in all subrepresentations are depicted graphically via the so called Newton diagrams.Comment: uuencoded Z-compressed .tar file containing two ps files

    Anti de Sitter Holography via Sekiguchi Decomposition

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    In the present paper we start consideration of anti de Sitter holography in the general case of the (q+1)-dimensional anti de Sitter bulk with boundary q-dimensional Minkowski space-time. We present the group-theoretic foundations that are necessary in our approach. Comparing what is done for q=3 the new element in the present paper is the presentation of the bulk space as the homogeneous space G/H = SO(q,2)/SO(q,1), which homogeneous space was studied by Sekiguchi.Comment: 10 pages, to appear in the Proceedings of the XI International Workshop "Lie Theory and Its Applications in Physics", (Varna, Bulgaria, June 2015

    A Mixed Phase of SUSY Gauge Theories from a-Maximization

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    We study N=1 supersymmetric SU(N) gauge theories with an antisymmetric tensor and F flavors using the recent proposal of a-maximization by Intriligator and Wecht. This theory had previously been studied using the method of "deconfinement", but such an analysis was not conclusive since anomalous dimensions in the non-perturbative regime could not be calculated. Using a-maximization we show that for a large range of F the theory is at an interacting superconformal fixed point. However, we also find evidence that for a range of F the theory in the IR splits into a free "magnetic" gauge sector and an interacting superconformal sector.Comment: 18 pages, 3 figure

    Duality for Exotic Bialgebras

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    In the classification of Hietarinta, three triangular 4×44\times 4 RR-matrices lead, via the FRT formalism, to matrix bialgebras which are not deformations of the trivial one. In this paper, we find the bialgebras which are in duality with these three exotic matrix bialgebras. We note that the LTL-T duality of FRT is not sufficient for the construction of the bialgebras in duality. We find also the quantum planes corresponding to these bialgebras both by the Wess-Zumino R-matrix method and by Manin's method.Comment: 25 pages, LaTeX2e, using packages: cite, amsfonts, amsmath, subeq

    On twist-two operators in N=4 SYM

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    We propose a mechanism for calculating anomalous dimensions of higher-spin twist-two operators in N=4 SYM. We consider the ratio of the two-point functions of the operators and of their superconformal descendants or, alternatively, of the three-point functions of the operators and of the descendants with two protected half-BPS operators. These ratios are proportional to the anomalous dimension and can be evaluated at n-1 loop in order to determine the anomalous dimension at n loops. We illustrate the method by reproducing the well-known one-loop result by doing only tree-level calculations. We work out the complete form of the first-generation descendants of the twist-two operators and the scalar sector of the second-generation descendants.Comment: references added; typos correcte

    Operator Representations on Quantum Spaces

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    In this article we present explicit formulae for q-differentiation on quantum spaces which could be of particular importance in physics, i.e., q-deformed Minkowski space and q-deformed Euclidean space in three or four dimensions. The calculations are based on the covariant differential calculus of these quantum spaces. Furthermore, our formulae can be regarded as a generalization of Jackson's q-derivative to three and four dimensions.Comment: 34 pages, Latex, major modifications to improve clarity, corrected typo

    Probing the accuracy of explicit solvent constant pH molecular dynamics simulations for peptides

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    Protonation states of titratable amino acids play a key role in many biomolecular processes. Knowledge of protonatable residue charges at a given pH is essential for a correct understanding of protein catalysis, inter- and intramolecular interactions, substrate binding, and protein dynamics for instance. However, acquiring experimental values for individual amino acid protonation states of complex systems is not straightforward; therefore, several in silico approaches have been developed to tackle this issue. In this work, we assess the accuracy of our previously developed constant pH MD approach by comparing our theoretically obtained pKa values for titratable residues with experimental values from an equivalent NMR study. We selected a set of four pentapeptides, of adequately small size to ensure comprehensive sampling, but concurrently, due to their charge composition, posing a challenge for protonation state calculation. The comparison of the pKa values shows good agreement of the experimental and the theoretical approach with a largest difference of 0.25 pKa units. Further, the corresponding titration curves are in fair agreement, although the shift of the Hill coefficient from a value of 1 was not always reproduced in simulations. The phase space overlap in Cartesian space between trajectories generated in constant pH and standard MD simulations is fair and suggests that our constant pH MD approach reasonably well preserves the dynamics of the system, allowing dynamic protonation MD simulations without introducing structural artifacts

    Duality for the Jordanian Matrix Quantum Group GLg,h(2)GL_{g,h}(2)

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    We find the Hopf algebra Ug,hU_{g,h} dual to the Jordanian matrix quantum group GLg,h(2)GL_{g,h}(2). As an algebra it depends only on the sum of the two parameters and is split in two subalgebras: Ug,hU'_{g,h} (with three generators) and U(Z)U(Z) (with one generator). The subalgebra U(Z)U(Z) is a central Hopf subalgebra of Ug,hU_{g,h}. The subalgebra Ug,hU'_{g,h} is not a Hopf subalgebra and its coalgebra structure depends on both parameters. We discuss also two one-parameter special cases: g=hg =h and g=hg=-h. The subalgebra Uh,hU'_{h,h} is a Hopf algebra and coincides with the algebra introduced by Ohn as the dual of SLh(2)SL_h(2). The subalgebra Uh,hU'_{-h,h} is isomorphic to U(sl(2))U(sl(2)) as an algebra but has a nontrivial coalgebra structure and again is not a Hopf subalgebra of Uh,hU_{-h,h}.Comment: plain TeX with harvmac, 16 pages, added Appendix implementing the ACC nonlinear ma

    Cellular bases for human atrial fibrillation

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    Atrial fibrillation (AF) causes substantial morbidity and mortality. It may be triggered and sustained by either reentrant or nonreentrant electrical activity. Human atrial cellular refractory period is shortened in chronic AF, likely aiding reentry. The ionic and molecular mechanisms are not fully understood and may include increased inward rectifier K<sup>+</sup> current and altered Ca<sup>2+</sup> handling. Heart failure, a major cause of AF, may involve arrhythmogenic atrial electrical remodeling, but the pattern is unclear in humans. Beta-blocker therapy prolongs atrial cell refractory period; a potentially antiarrhythmic influence, but the ionic and molecular mechanisms are unclear. The search for drugs to suppress AF without causing ventricular arrhythmias has been aided by basic studies of cellular mechanisms of AF. It remains to be seen whether such drugs will improve patient treatment
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